ライフサイエンスコーパス: anesthetic drug

1 ges in consciousness from the effects of the anesthetic drugs.

2 nonneuronal mechanism for sedative action of anesthetic drugs.

3 nel's inner pore overlap with those of local anesthetic drugs.

4 ty's effects on the clinical pharmacology of anesthetic drugs.

5 an systems decreases the margin of safety of anesthetic drugs.

6 eration in rat pups exposed to commonly used anesthetic drugs.

7 ne cardiac-specific external paths for local anesthetic drugs.

8 Each altered arousal state results from the anesthetic drugs acting at multiple targets in the centr

9 results of preclinical studies suggest that anesthetic drugs administered to neonatal animals cause

10 f the immunological effects of commonly used anesthetic drugs and highlight their potential impact on

11 o alterations of physiology and in choice of anesthetic drugs and techniques.

12 Anesthetic drugs are known to interact with GABAA recept

13 Anesthetic drugs are thought to mainly target neurons in

14 e than a decade of mounting animal data that anesthetic drugs can cause apoptosis during a critical p

15 Propofol, like most general anesthetic drugs, can induce both behavioral and electro

16 The neurophysiological mechanisms by which anesthetic drugs cause loss of consciousness are poorly

17 How anesthetic drugs create the state of general anesthesia

18 BIS monitor results in less use of hypnotic anesthetic drugs, decreased time to extubation, decrease

19 This retards anesthetic drug development, confounds interpretation of

20 Devices which monitor some aspect of anesthetic drug effects have evolved in the past few yea

21 ofol (2,6-di-isopropylphenol), a widely used anesthetic drug, exerts its effect primarily by modulati

22 f induction, brought about by elimination of anesthetic drugs from their CNS site(s) of action.

23 tudies is clear and continuing to mount that anesthetic drugs given at the right time and in sufficie

24 e relaxation, and to minimize the amounts of anesthetic drugs given to infants and sick children.

25 oduction of newer less-toxic, shorter-acting anesthetic drugs has reduced the requirement for muscle

26 Many anesthetic drugs have been shown to disrupt conscious re

27 anges in practice and the development of new anesthetic drugs have influenced the use of muscle relax

28 This is a consequence of ideal mixing of the anesthetic drugs in the membrane fluid phase and exclusi

29 have shown that prolonged administration of anesthetic drugs, including ketamine, isoflurane, nitrou

30 lly diverse group of sedative, hypnotic, and anesthetic drugs, including the volatile anesthetic isof

31 ulk of reported complications are related to anesthetic drug-induced respiratory depression or airway

32 Malignant hyperthermia (MH) is an anesthetic-drug-induced, life-threatening hypermetabolic

33 novel ways of reversing the effects of some anesthetic drugs (inhalational anesthetics and muscle re

34 (GABA(A)Rs), and it is assumed that once the anesthetic drug is eliminated, the activity of GABA(A)Rs

35 support for the antidepressant effect of an anesthetic drug, ketamine, by Inverse-Frequency Analysis

36 the deactivation of NsVBa, whereas the local anesthetic drug lidocaine was shown to antagonize NsVBa

37 eceptor (NMDAR) antagonists are dissociative anesthetics, drugs of abuse, and are of therapeutic inte

38 us be understood as a differential effect of anesthetic drugs on thalamic nuclei with disparate spati

39 many to investigate the neurotoxic effect of anesthetic drugs on the developing brain.

40 ainly question and undermine the safe use of anesthetic drugs, particularly in pediatric anesthesia,

41 Use of enantiomeric local anesthetic drugs permits a safer and wider range of post

42 ions (10-15 Hz) induced by the commonly used anesthetic drug propofol are synchronized between the th

43 ors, such as the influence of vasoactive and anesthetic drugs, total muscular relaxation, or the pres

44 ry of epilepsy, previous SE, type of SE, and anesthetic drug used were not associated with functional

45 tations these monitors can be used to reduce anesthetic drug utilization and turnover time.

46 Propofol, a sedative and anesthetic drug was chosen as a model lipophilic drug in

47 troencephalogram background suppression with anesthetic drugs) was tested.

48 anesthetics and consequences of exposure to anesthetic drugs will likely require the evaluation and

49 iate seizure activity and the interaction of anesthetic drugs with AEDs.