ライフサイエンスコーパス: aneurysm

1 nts, 25 (53.8%) had more than 1 intracranial aneurysm.

2 which was initially thought to be an aortic aneurysm.

3 CI, 10.3%-15.9%) had at least 1 intracranial aneurysm.

4 s frequently accompanied by ascending aortic aneurysm.

5 t had discordance of aortic valve and aortic aneurysm.

6 on for rupture of superior mesenteric artery aneurysm.

7 e models of sterile inflammation and carotid aneurysm.

8 graphy confirmed a complete exclusion of the aneurysm.

9 intracranial imaging who had an intracranial aneurysm.

10 only used pan-MMP tracer in murine models of aneurysm.

11 etection of inflammation and MMP activity in aneurysm.

12 egulation of NLRX1 in brain injury following aneurysm.

13 ity were shown by autoradiography in carotid aneurysm.

14 ivotally in many diseases including arterial aneurysms.

15 of very small (</=3 mm) and small (</=5 mm) aneurysms.

16 3 expression and affects atherosclerosis and aneurysms.

17 de for patients with ruptured and unruptured aneurysms.

18 equent stabilization of existing plaques and aneurysms.

19 As) constitute 14% to 20% of visceral artery aneurysms.

20 popular treatment modality for many types of aneurysms.

21 it resulted in a complete embolization of 5 aneurysms.

22 y to KD and the formation of coronary artery aneurysms.

23 inal necrosis and multiple visceral arterial aneurysms.

24 ctive surgery for descending thoracic aortic aneurysms.

25 n, flow diversion, is now available to treat aneurysms.

26 imited by its inability to occlude wide-neck aneurysms.

27 wide-necked proximal internal carotid artery aneurysms.

28 effective endovascular treatment of cerebral aneurysms.

29 technique for the repair of abdominal aortic aneurysms.

30 rysms (86.2%), and partial embolization in 4 aneurysms (13.8%), respectively.

31 per 1000 patient days were as follows: 0.04 aneurysms (14 unique cohorts; n=1827 fistulas), 0.11 inf

32 ysms (83.3%), and partial embolization in 11 aneurysms (16.7%).

33 patients (43.2%), and 24 of 128 intracranial aneurysms (18.8%) were in the posterior communicating or

34 Only saccular or broad-based aneurysms 2 mm or larger in greatest dimension were incl

35 vs 163 of 564 patients without intracranial aneurysm (28.9%; P < .001).

36 s, 5, 10, and 8 described the growth rate of aneurysms 3 mm and smaller, 5 mm and smaller, and 7 mm a

37 s were reported in 7, 11, and 13 studies for aneurysms 3 mm and smaller, 5 mm and smaller, and 7 mm a

38 2 years, 3 of the 93 patients with LV apical aneurysms (3%) died suddenly or of heart failure, but 22

39 se causes, the ratio ranged from 1.4 (aortic aneurysm: 3.5 vs 5.1 deaths per 100000 persons) to 4.2 (

40 s significantly associated with intracranial aneurysm: 42 of 78 patients with intracranial aneurysm (

41 In 29 aneurysms (43.9%), the effect of the procedure was confi

42 Intracranial aneurysms 5 mm or larger occurred in 32 of 74 patients (

43 neurysm: 42 of 78 patients with intracranial aneurysm (53.8%) had a smoking history vs 163 of 564 pat

44 A complete embolization was performed in 55 aneurysms (83.3%), and partial embolization in 11 aneury

45 DSA, complete embolization was present in 25 aneurysms (86.2%), and partial embolization in 4 aneurys

46 In 6 aneurysms (9.1%), re-embolization was necessary and it r

47 After the endovascular treatment of the aneurysm, a full recovery of cranial nerve function was

48 s (MMPs) play a key role in abdominal aortic aneurysm (AAA) development.

49 The role of resolvins in abdominal aortic aneurysm (AAA) has not been established.

50 Abdominal aortic aneurysm (AAA) is a common aortic disease with a progres

51 RATIONALE: Abdominal aortic aneurysm (AAA) is a complex disease with both genetic an

52 Abdominal aortic aneurysm (AAA) is a degenerative disease characterized b

53 Abdominal aortic aneurysm (AAA) is a major cause of morbidity and mortali

54 An abdominal aortic aneurysm (AAA) is a permanent and irreversible dilation

55 d tryptophan metabolism and abdominal aortic aneurysm (AAA) is unknown.

56 ed for treatments to reduce abdominal aortic aneurysm (AAA) progression.

57 sically frail patients with abdominal aortic aneurysm (AAA) randomized to either early endovascular a

58 to 100 years who underwent abdominal aortic aneurysm (AAA) repair (n = 71,422), pulmonary resection

59 after open and endovascular abdominal aortic aneurysm (AAA) repair are each well described separately

60 D) predicts mortality after abdominal aortic aneurysm (AAA) repair.

61 ventitial DCN is reduced in abdominal aortic aneurysm (AAA) resulting in vessel wall instability ther

62 ed in literature are due to abdominal aortic aneurysm (AAA) rupture into the left renal vein.

63 ain control during elective abdominal aortic aneurysm (AAA) surgery.

64 and endovascular repair of abdominal aortic aneurysm (AAA), cost may be an important factor in choos

65 the event of rupture of an abdominal aortic aneurysm (AAA), mortality is very high.

66 Abdominal aortic aneurysms (AAAs) are a deadly pathology with strong sexu

67 Abdominal aortic aneurysms (AAAs) represent a potentially life-threatenin

68 soform of alpha-actin, cause thoracic aortic aneurysms, acute aortic dissections, and occlusive vascu

69 Remodeling of abdominal aortic aneurysms after EVAR is not uniform.

70 ctive and safe treatment method for cerebral aneurysms, although it carries the risk of some complica

71 nths after the procedure, showed an excluded aneurysm and a patent covered stent.

72 ced arterial fitness and an elevated risk of aneurysm and aortic rupture.

73 ed data on genes involved in thoracic aortic aneurysm and attempts to explain divergent hypotheses of

74 Sporadic aortic aneurysm and dissections (AADs) are common vascular dise

75 omotes hypertension, atherogenesis, vascular aneurysm and impairs post-ischemic cardiac remodeling th

76 (99m)Tc-RYM1 binding to aneurysm and its specificity were shown by autoradiograp

77 f choice for young patients with aortic root aneurysm and normal or near-normal aortic cusps.

78 ve in preclinical models of abdominal aortic aneurysm and show great potential for clinical translati

79 (99m)Tc-RYM1 binding to aneurysm and specificity were evaluated by quantitative

80 rtly after parturition from ascending aortic aneurysm and spontaneous hemorrhage.

81 5.7%); in total, 384 patients (41.7%) had an aneurysm and/or a dissection by the time of FMD diagnosi

82 Patients with FMD have a high prevalence of aneurysm and/or dissection prior to or at the time of FM

83 location, and outcomes of FMD patients with aneurysm and/or dissection to those of patients without.

84 d morbidity in patients with FMD who have an aneurysm and/or dissection, it is recommended that every

85 Thoracic aortic aneurysms and acute aortic dissections (TAADs) occur as

86 enTAC (Genetically Triggered Thoracic Aortic Aneurysms and Cardiovascular Conditions) is a longitudin

87 enTAC (Genetically Triggered Thoracic Aortic Aneurysms and Cardiovascular Conditions) Registry (53% m

88 atherosclerotic plaques and abdominal aortic aneurysms and correlated with decreased expression of pr

89 atherosclerotic plaques and abdominal aortic aneurysms and correlated with decreased expression of pr

90 tic alterations for familial thoracic aortic aneurysms and dissections (TAAD) are missense mutations

91 Thoracic aortic aneurysms and dissections (TAAD) represent a substantial

92 Heritable thoracic aortic aneurysms and dissections (TAAD), including Marfan syndr

93 Thoracic aortic diseases, including aneurysms and dissections of the thoracic aorta, are a m

94 onditions that predispose to thoracic aortic aneurysms and dissections, including MFS.

95 1 locus associated with both thoracic aortic aneurysms and dissections.

96 5% of families with familial thoracic aortic aneurysms and dissections.

97 ible for a large family with thoracic aortic aneurysms and dissections.

98 flammation in patients with abdominal aortic aneurysms and predicts the rate of aneurysm growth and c

99 Aortic aneurysms and prophylactic aortic surgery are more commo

100 iting the progression of atherosclerosis and aneurysms and protecting them from rupture.

101 Aneurysms and rupture did not occur with norepinephrine-

102 ischemia as a consequence of coronary artery aneurysms and stenosis.

103 ow-shear-rate conditions in abdominal aortic aneurysms and thoracic aortic dissections to thrombosis

104 n the RF group, 2 patients developed a false aneurysm, and 1 patient needed surgical repair.

105 initial DS angiographic results negative for aneurysm, and two DSA examinations within 10 days.

106 e incidence and severity of abdominal aortic aneurysms, and caused aortic arch ruptures and dissectio

107 han that for serious liver disease, cerebral aneurysms, and inguinal hernias but less than that for u

108 HCM patients with LV apical aneurysms are at high risk for arrhythmic sudden death a

109 RATIONALE: Atherosclerosis and aneurysms are leading causes of mortality worldwide.

110 centuries, physicians have recognized aortic aneurysms as an acute threat to life.

111 Young adults with aortic root aneurysms associated with genetic syndromes are ideal ca

112 Purpose To quantify the rate of detection of aneurysms at follow-up digital subtraction angiography (

113 ysm repair, registered in the Dutch Surgical Aneurysm Audit between 2014 and 2015 were included.

114 Bronchial artery aneurysm (BAA) is a rare condition with a reported preva

115 r each sex separately, with abdominal aortic aneurysms being assessed for aneurysm repair by either e

116 ith a nonruptured infrarenal aortic or iliac aneurysm between September 2012 and June 2014.

117 sional campaign to fund screening for aortic aneurysms brought the disease to national attention and

118 ir of ruptured anterior communicating artery aneurysms by clip ligation (n = 14) or coiling (n = 9).

119 roptosis with Nec-1s stabilizes pre-existing aneurysms by diminishing inflammation and promoting conn

120 igh packing density of coils in the lumen of aneurysms can decrease the risk of recurrence.

121 including 2 patients with an aortic mycotic aneurysm, caused significant morbidity.

122 lasm in the acetylcysteine group, and aortic aneurysm, contusion, forearm fracture, and worsening IPF

123 A change of the aneurysm cross-sectional over 10% was considered signifi

124 The summary rate of aneurysm detection for subsequent DSA was calculated by

125 The summary aneurysm detection rate at subsequent DSA was 1.6% (95%

126 reduced arterial wall stiffness and hampered aneurysm development.

127 d as a common molecular signature for aortic aneurysm development.

128 garding vessel wall biology in the course of aneurysm development.

129 Baseline abdominal aortic aneurysm diameter (P<0.0001) and current smoking habit (

130 Data on the aneurysm diameter at the time of repair were extracted f

131 (85% male, 73.1+/-7.2 years) had a baseline aneurysm diameter of 49.6+/-7.7 mm, and USPIO enhancemen

132 x (number of devices >2) + 0.016 x (maximum aneurysm diameter).

133 rt study, 342 patients with abdominal aortic aneurysm (diameter >/=40 mm) were classified by the pres

134 The presence of intracranial aneurysm did not vary with location of extracranial FMD

135 es, minimally invasive methods that approach aneurysms endovascularly through small groin incisions h

136 r endovascular treatment of abdominal aortic aneurysms (EVAR) is mainly aimed at detection of endolea

137 Coronary artery aneurysms evolve dynamically over time, usually reaching

138 th USPIO enhancement have increased rates of aneurysm expansion (3.1+/-2.5 versus 2.5+/-2.4 mm/year,

139 t does not provide independent prediction of aneurysm expansion or clinical outcomes in a model incor

140 tic rupture, rapid aortic growth (>10 mm/y), aneurysm formation (>/=6 cm), organ or limb ischemia, or

141 es resolvins inhibit murine abdominal aortic aneurysm formation and increase M2 macrophage polarizati

142 used Apoe(-/-) and Ldlr(-/-) mice attenuated aneurysm formation and progression within the ascending,

143 allenge-induced aortic diameter enlargement, aneurysm formation, dissection and aortic rupture.

144 ta and its large branches, is complicated by aneurysm formation, dissection, and arterial occlusions.

145 . (2017) report that, in experimental aortic aneurysm formation, neutralization of interleukin-1beta

146 Using a mouse model of aneurysm formation, we showed that the combination of Mm

147 ated that the lack of RIP3 in mice prevented aneurysm formation.

148 p CT scans revealed a total exclusion of the aneurysm from the systemic circulation, resolution of th

149 al aortic aneurysms and predicts the rate of aneurysm growth and clinical outcome.

150 harmacological treatment to effectively slow aneurysm growth or prevent rupture.

151 essed whether USPIO-enhanced MRI can predict aneurysm growth rates and clinical outcomes.

152 Hepatic artery aneurysms (HAAs) constitute 14% to 20% of visceral arter

153 A genetic predisposition to thoracic aortic aneurysm has been established, and gene discovery in aff

154 .78; 95% CI: 1.51 to 2.10), abdominal aortic aneurysm (HR: 1.72; 95% CI: 1.34 to 2.21), and nonfatal

155 g are 2 novel approaches to abdominal aortic aneurysm imaging evaluated in clinical trials.

156 moment of treatment decision of the ruptured aneurysm improved model performance.

157 The prevalence of intracranial aneurysm in patients with fibromuscular dysplasia (FMD)

158 inical benefit of screening for intracranial aneurysm in patients with FMD has yet to be proven, thes

159 moplegia secondary to a traumatic dissecting aneurysm in the cavernous segment of internal carotid ar

160 otid artery (ICA) together with a dissecting aneurysm in the cavernous segment.

161 The prevalence of intracranial aneurysm in women diagnosed with FMD is significantly hi

162 To examine the prevalence of intracranial aneurysm in women diagnosed with FMD.

163 The management of abdominal aortic aneurysm in women needs improvement.

164 s of childhood that leads to coronary artery aneurysms in approximately 25% of untreated cases.

165 To investigate the pathogenesis of aortic aneurysms in MFS, we generated a vascular model derived

166 after initial DSA with results negative for aneurysms in subjects with perimesencephalic (PM) nonane

167 Extradural aneurysms in the internal carotid artery were included;

168 Seven days after aneurysm induction by elastase perfusion, mice were rand

169 pression at early and late time points after aneurysm induction.

170 ed macrophages reduced aortic dilation after aneurysm induction.

171 ernal carotid artery were included; fusiform aneurysms, infundibulae, and vascular segments with unce

172 he rupture of a partially thrombosed mycotic aneurysm into the biliary tree.

173 nding of the pathogenesis of thoracic aortic aneurysm is quite limited.

174 laparotomy in patients with abdominal aortic aneurysm is safe and effectively prevents the developmen

175 ssess whether the stability of murine aortic aneurysms is associated with the homogeneity of pulse wa

176 ) patients with left ventricular (LV) apical aneurysms is being identified with increasing frequency.

177 gnosis and appropriate therapy of dissecting aneurysms is necessary for good clinical outcomes in cas

178 of follow-up DSA for detection of causative aneurysms is very low.

179 h multiple efferent arteries, dolichoectatic aneurysms, large aneurysms with mass effect, when there

180 or endovascular coiling, such as wide-necked aneurysms located at branch points of major vessels, lar

181 m repair (EVAR) for mycotic abdominal aortic aneurysms (MAAAs).

182 which (4.8%) were identified with LV apical aneurysms; mean age was 56 +/- 13 years, and 69% were ma

183 ding atrial fibrillation (Northwest), aortic aneurysm (Midwest), and endocarditis (Mountain West and

184 Prognosis for women with abdominal aortic aneurysm might be worse than the prognosis for men.

185 (apoE(-/-)) mice with CaCl2-induced carotid aneurysm (n = 11).

186 In the LB group, stroke (n=1), a false aneurysm (n=1), and phrenic nerve palsy (n=1) were obser

187 An incidental finding of a bronchial artery aneurysm necessitates prompt treatment.

188 n cardiovascular pathologies, such as aortic aneurysm, new knowledge on the involvement of cell-matri

189 as flow diversion, are associated with high aneurysm occlusion rates and have become a popular treat

190 Aneurysm occurred in 200 patients (21.7%) and dissection

191 giography revealed a single bronchial artery aneurysm of 9 mm in diameter, abutting the esophageal wa

192 Two animals had occlusion of hTPV with aneurysm of main pulmonary artery.

193 We report a case of pseudo aneurysm of splenic artery developed after an episode of

194 dissected segment of ICA, and the dissecting aneurysm of the cavernous segment was successfully manag

195 An aneurysm of the superior mesenteric artery (SMA) with a

196 ients had to be aged 60 years or older, have aneurysms of at least 5.5 cm in diameter, and deemed sui

197 mortality in patients after repair of aortic aneurysms of the descending thoracic aorta thoracic endo

198 -year-old man who developed multiple mycotic aneurysms of the right hepatic artery and massive spleni

199 this is the first case reported of infected aneurysms of visceral arteries caused by Group B strepto

200 atment, intracranial haemorrhage or residual aneurysm on 1-year imaging.

201 s proximity with blood, molecular imaging of aneurysm optimally requires highly sensitive tracers wit

202 uted to PFO with an associated atrial septal aneurysm or large interatrial shunt, the rate of stroke

203 ted to PFO, with an associated atrial septal aneurysm or large interatrial shunt, to transcatheter PF

204 gen content, without signs of calcification, aneurysm or stenosis.

205 (freedom from immune response or infection, aneurysm, or mechanical failure, and incidence of advers

206 mostly manifesting as endocarditis, infected aneurysms, or infected vascular prostheses.

207 attempts to explain divergent hypotheses of aneurysm origin.

208 large cohort of HCM patients with LV apical aneurysms over long-term follow-up.

209 evelopment of specific treatments for aortic aneurysms over time and more broadly addresses how medic

210 ear event rate in 1,847 HCM patients without aneurysms (p < 0.001).

211 and flutter, rheumatic heart disease, aortic aneurysm, peripheral arterial disease, endocarditis, and

212 hree who received deferred treatment (aortic aneurysm, pneumonia, and unknown cause); all four deaths

213 f the exact site of communication, length of aneurysm, proximal and distal extent of the affected seg

214 s hospitalized for ruptured abdominal aortic aneurysms (rAAA) by conducting a retrospective analysis

215 OC at onset and delayed cerebral ischemia or aneurysm rebleeding.

216 The primary analysis compared total and aneurysm-related deaths in groups until June 30, 2015 (m

217 The primary analysis compared total and aneurysm-related deaths in groups until mid-2015 in the

218 0.0275), all-cause mortality (P=0.0635), and aneurysm-related mortality (P=0.0590).

219 the study was to compare long-term total and aneurysm-related mortality in physically frail patients

220 However, overall aneurysm-related mortality was significantly lower in th

221 ortality; and 5.82, 1.64-20.65, p=0.0064 for aneurysm-related mortality).

222 for total mortality; and 0.47, 0.23-0.93 for aneurysm-related mortality, p=0.031), but beyond 8 years

223 s ineligible for open repair, but can reduce aneurysm-related mortality.

224 With these modalities, many large or complex aneurysms remain difficult to treat.

225 ost-of-rescue hospitals for abdominal aortic aneurysm repair ($60456 vs $23261; P < .001), colectomy

226 tions, prolonged hospital stay [endovascular aneurysm repair (EVAR) </=4 d, open surgical repair (OSR

227 st between open repair (OR) and endovascular aneurysm repair (EVAR) for mycotic abdominal aortic aneu

228 AAA) randomized to either early endovascular aneurysm repair (EVAR) or no-intervention.

229 ent decades with a shift toward endovascular aneurysm repair (EVAR).

230 alve replacement (n = 3223), an endovascular aneurysm repair (n = 12633), or a percutaneous left vent

231 stence and outcomes of open abdominal aortic aneurysm repair (OAR) and carotid endarterectomy (CEA) p

232 bdominal aortic aneurysms being assessed for aneurysm repair by either endovascular repair (EVAR) or

233 Persistent aortic remodeling after aneurysm repair could place the patient at risk for endo

234 atients undergoing elective abdominal aortic aneurysm repair through a midline laparotomy (Clinical.T

235 e 2013, it takes a revolutionary approach to aneurysm repair through minimally invasive techniques.

236 (colectomy, lung resection, abdominal aortic aneurysm repair, coronary artery bypass graft, aortic va

237 atients undergoing elective abdominal aortic aneurysm repair, registered in the Dutch Surgical Aneury

238 ormation in patients undergoing endovascular aneurysm repair.

239 ty (operative mortality review) after intact aneurysm repair.

240 centage of patients who had abdominal aortic aneurysm-repair without intraoperative complications, po

241 urysm ruptures, 126 (36.8%) abdominal aortic aneurysm repairs, and 48 (14.0%) deaths occurred.

242 Hepatic artery aneurysms represent a significant risk for hemorrhage an

243 Patients with aneurysms require life-long and uninterrupted cardiology

244 n for midaortic syndrome and multiple aortic aneurysms, respectively underwent renal transplantation.

245 s with USPIO enhancement had higher rates of aneurysm rupture or repair (47.3% versus 35.6%; 95% conf

246 ociated with reduced event-free survival for aneurysm rupture or repair (P=0.0275), all-cause mortali

247 The primary end point was the composite of aneurysm rupture or repair.

248 hen XY females were exposed to testosterone, aneurysm rupture rates were striking.

249 ts BioPARR (Biomechanics based Prediction of Aneurysm Rupture Risk), a software system to facilitate

250 AAA incidences doubled and aneurysms ruptured in XY females.

251 1005+/-280 days), 17 (5.0%) abdominal aortic aneurysm ruptures, 126 (36.8%) abdominal aortic aneurysm

252 l diameter and volume of an abdominal aortic aneurysm sac can be used for temporal monitoring after e

253 verter', can divert blood flow away from the aneurysm sac.

254 d centerline; volume of the abdominal aortic aneurysm sac; and volume from the lowest renal artery to

255 changes in the size of the abdominal aortic aneurysm sack using CT angiography (CTA) after successfu

256 Although aneurysm sealing may be thought as more stable than conv

257 NF-kappaB signaling that is correlated with aneurysm severity in these human subjects.

258 any benefit of reduced apoptosis, increasing aneurysm severity.

259 There was no consistent relationship between aneurysm size and adverse HCM-related events.

260 ange of aneurysm size depends on the initial aneurysm size but not on the time from EVAR.

261 The change of aneurysm size depends on the initial aneurysm size but n

262 ew endo-leaks were associated with increased aneurysm size measured as the largest diameter on the ax

263 The model's predictors were age, aneurysm size, Fisher grade, and World Federation of Neu

264 f endoleak, sac pressure, endoleak size, and aneurysm size.

265 growth and rupture risks, which may vary by aneurysm size.

266 atherosclerotic plaque and abdominal aortic aneurysm stability are poorly understood.

267 rs in maintaining atherosclerotic plaque and aneurysm stability.

268 ess in ligating arteries to treat peripheral aneurysms, surgeons attempted analogous operations on th

269 h comprises a desirable outcome for elective aneurysm surgery, called "Textbook Outcome" (TO).

270 as poorly controlled hypertension, previous aneurysm surgery, splenectomy, acute aortic dissection,

271 the overall quality of the care of elective aneurysm surgery, which subsequently can be used by hosp

272 is a serious complication of thoracic aortic aneurysm (TAA).

273 ic aortopathy (GA) underlies thoracic aortic aneurysms (TAA) in younger adults.

274 e only known risk factor was an intracranial aneurysm that was found on her grandmother's autopsy.

275 e methods to surgery, especially in cases of aneurysms that do not respond to medical therapy.

276 Moreover, there are some types of aneurysms that may not be appropriate for endovascular c

277 and open cerebrovascular surgery for complex aneurysms that may not be suitable for coiling.

278 noid hemorrhage and initial DSA negative for aneurysms, the yield of follow-up DSA for detection of c

279 This allows aortic aneurysms to serve as a case study for exploring shiftin

280 ment failure, defined as: initial failure of aneurysm treatment, intracranial haemorrhage or residual

281 ere performed at approximately 24-48 h after aneurysm treatment.

282 shed in trials comparing it with traditional aneurysm treatments.

283 rgery, splenectomy, acute aortic dissection, aneurysm type, older age, and history of diabetes and sm

284 Unruptured intracranial aneurysms (UIAs) are increasingly diagnosed and are comm

285 Small unruptured intracranial aneurysms (UIAs) are increasingly diagnosed.

286 VAC presented as a large perifoveal isolated aneurysm, unifocal in 12 of 15 eyes, associated with sma

287 sessed for repair of intact abdominal aortic aneurysm using data from study periods after the year 20

288 necroptosis affects progression of existing aneurysm using the RIP1 inhibitors Necrostatin-1 (Nec-1)

289 Thresholds for repair of abdominal aortic aneurysms vary considerably among countries.

290 spindle-shaped and inflammatory cells in the aneurysm wall.

291 e associated with BAV and/or thoracic aortic aneurysm was performed.

292 In patients with abdominal aortic aneurysm, we assessed whether USPIO-enhanced MRI can pre

293 Three aneurysms were detected at follow-up DSA in three of six

294 Sixty-six cerebral aneurysms were embolized with hydrogel coils, which expa

295 vertebral, or suspected intracranial artery aneurysms were reviewed.

296 nt arteries, dolichoectatic aneurysms, large aneurysms with mass effect, when there are technical com

297 anch points of major vessels, large saccular aneurysms with multiple efferent arteries, dolichoectati

298 ty of endovascular treatment of intracranial aneurysms with the use hydrogel coils.

299 c regimes, acting on sealed abdominal aortic aneurysms, with references to real case studies.

300 y (CT) and two DSA examinations negative for aneurysm within 10 days were evaluated for inclusion in