ライフサイエンスコーパス: aneurysmal subarachnoid hemorrhage

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1 ic stiffness occur during the early phase of aneurysmal subarachnoid hemorrhage.

2 een used to prevent cerebral vasospasm after aneurysmal subarachnoid hemorrhage.

3 mprove the outcome among patients with acute aneurysmal subarachnoid hemorrhage.

4 er craniotomy among good-grade patients with aneurysmal subarachnoid hemorrhage.

5 oped as a tool for risk stratification after aneurysmal subarachnoid hemorrhage.

6 the epidemiology of types of stroke, such as aneurysmal subarachnoid hemorrhage and cerebral vein thr

7 agnosing cerebral vasospasm in patients with aneurysmal subarachnoid hemorrhage and for guiding trans

8 se-wave velocity were improved between acute aneurysmal subarachnoid hemorrhage and stable state (p <

9 n ischemic stroke, intracerebral hemorrhage, aneurysmal subarachnoid hemorrhage, and traumatic brain

10 Early lactate and glucose levels after aneurysmal subarachnoid hemorrhage are associated with d

11 cantly contribute to poor outcomes following aneurysmal subarachnoid hemorrhage (aSAH).

12 ay be used to prevent early rebleeding after aneurysmal subarachnoid hemorrhage, but anticoagulation

13 anal-modified protein levels in humans after aneurysmal subarachnoid hemorrhage correlate with the de

14 model to predict 60-day case fatality after aneurysmal subarachnoid hemorrhage developed from the In

15 The care of patients with aneurysmal subarachnoid hemorrhage has evolved significa

16 ignificant morbidity and mortality following aneurysmal subarachnoid hemorrhage; however, the effect

17 ologies including migraine, ischemic stroke, aneurysmal subarachnoid hemorrhage, intracerebral hemato

18 diagnosis as primary cerebral vasculitis and aneurysmal subarachnoid hemorrhage is common because of

19 Aneurysmal subarachnoid hemorrhage often leads to death

20 ated at two time points: on admission (acute aneurysmal subarachnoid hemorrhage phase) and at least 2

21 Cerebral vasospasm following aneurysmal subarachnoid hemorrhage (SAH) has devastating

22 cause ischemic neurological damage following aneurysmal subarachnoid hemorrhage (SAH) have remained e

23 Acute aneurysmal subarachnoid hemorrhage (SAH) is a complex mu

24 Aneurysmal subarachnoid hemorrhage (SAH) is a potentiall

25 thophysiology of early ischemic injury after aneurysmal subarachnoid hemorrhage (SAH) is not understo

26 Mortality and morbidity can be reduced if aneurysmal subarachnoid hemorrhage (SAH) is treated urge

27 In this prospective observational study of aneurysmal subarachnoid hemorrhage (SAH) patients, we ex

28 Aneurysmal subarachnoid hemorrhage (SAH) remains a devas

29 Significance statement: Aneurysmal subarachnoid hemorrhage (SAH)--strokes involv

30 he most severe and pervasive consequences of aneurysmal subarachnoid hemorrhage (SAH).

31 ge phase) and at least 21 days later (stable aneurysmal subarachnoid hemorrhage state).

32 n is associated with poor outcome, but after aneurysmal subarachnoid hemorrhage, this has not been in

33 Consecutive patients with aneurysmal subarachnoid hemorrhage treated with clipping

34 At baseline, the severity of aneurysmal subarachnoid hemorrhage was assessed clinical

35 rovide early identification of patients with aneurysmal subarachnoid hemorrhage who are at high risk

36 at risk for delayed cerebral ischemia after aneurysmal subarachnoid hemorrhage with hemoglobin 7-13

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