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1 nding of the complex contribution of CtsB to angiogenesis.
2 herapy represents a proven strategy to treat angiogenesis.
3 thylation and other PTMs in regulating tumor angiogenesis.
4 l miRNA-based therapeutic strategies against angiogenesis.
5 othelial cell proliferation to promote tumor angiogenesis.
6 o decreased the weights of tumors and cancer angiogenesis.
7 rmation, all of which are important steps in angiogenesis.
8 x and has a crucial role in fibrogenesis and angiogenesis.
9 tified regulatory pathway that could promote angiogenesis.
10 ase of reactive oxygen species and decreased angiogenesis.
11 ing and therefore as important regulators of angiogenesis.
12  have implications for alcohol-induced tumor angiogenesis.
13 ls in driving physiological and pathological angiogenesis.
14  by endothelium and are implicated in tumour angiogenesis.
15  are crucial for a complete understanding of angiogenesis.
16 et activation and inducing cell invasion and angiogenesis.
17 that contributes to tumour cell invasion and angiogenesis.
18 ility of targeting RUNX1 in aberrant retinal angiogenesis.
19 f adverse myocardial remodeling and promotes angiogenesis.
20 ous biochemical factors on tumour growth and angiogenesis.
21  elevated blood vessel sprouting and in vivo angiogenesis.
22 nt and maintenance of the vasculature and in angiogenesis.
23 ating apoptosis, inflammation, fibrosis, and angiogenesis.
24 PlGF pathways in the regulation of placental angiogenesis.
25 aracterized of accelerated postnatal retinal angiogenesis.
26 s emphasize that eNOS-derived NO can promote angiogenesis.
27 Endothelial metabolism is a key regulator of angiogenesis.
28 ngly promoted endothelial cells invasion and angiogenesis.
29 ibroblast and myofibroblast accumulation and angiogenesis.
30 ical for regulation of inflammation mediated angiogenesis.
31 othelial cells controls zebra fish embryonic angiogenesis.
32  the TLR9 ligand class C, ODN2395, increased angiogenesis.
33 ing, mediates immune responses, and triggers angiogenesis.
34 colonic surgery, possibly via its effects on angiogenesis.
35 endothelial fatty acid metabolism and tumour angiogenesis.
36 yme in glutamine catabolism, markedly blunts angiogenesis.
37 d from trophoblasts is involved in placental angiogenesis.
38 fic biomarkers, many of which have a role in angiogenesis.
39  suggesting a role for Cavin-2 in pathogenic angiogenesis.
40 autophagy in endothelial cells and attenuate angiogenesis.
41 endothelial growth factor, a major driver of angiogenesis.
42  tissue progenitor cell differentiation, and angiogenesis.
43 ealing in diabetic mice through facilitating angiogenesis.
44 ssing the extracellular matrix and promoting angiogenesis.
45 evaluating biliary mass, liver fibrosis, and angiogenesis.
46 ific alpha3 knockout mice displayed impaired angiogenesis.
47 bility and turnover in blood vessels impacts angiogenesis.
48 diates ischemic tissue repair via beneficial angiogenesis.
49 mor efficacy of bevacizumab, an inhibitor of angiogenesis.
50 he current available animal models of ocular angiogenesis.
51 lation of cell migration, proliferation, and angiogenesis.
52 ional pathway contributing to VEGF-dependent angiogenesis.
53 ndothelial cell proliferation during retinal angiogenesis.
54 l-cell adhesion, which are all requisite for angiogenesis.
55 ed a potential role of PTP1B in VEGF-induced angiogenesis.
56 ing several genes important for invasion and angiogenesis.
57 icellular, strand-like invasion required for angiogenesis.
58 s well as enhanced fibrinolysis and impaired angiogenesis.
59 ream effectors and are pivotal regulators of angiogenesis.
60 validating modulation of paxillin to inhibit angiogenesis.
61 cal-recognition motifs for cell adhesion and angiogenesis.
62 and stromal cells, and iii) cancer-regulated angiogenesis.
63 ecreased lung compliance, and decreased lung angiogenesis.
64 rway smooth muscle hyperplasia and increased angiogenesis.
65  investigated the role of IPMK in regulating angiogenesis.
66 ys including hedgehog, and mTOR pathways and angiogenesis.
67 is conceptually similar to hypoxia-triggered angiogenesis.
68 lls typically rely on aerobic glycolysis for angiogenesis.
69  CIB1 in regulating cancer cell survival and angiogenesis, although the mechanisms involved have rema
70    The inhibition of inflammation-associated angiogenesis ameliorates inflammatory diseases by reduci
71 e show that Cavin-2 is required for in vitro angiogenesis and also for endothelial cell proliferation
72 t mice (miR106b-93-25(-/-)) showed decreased angiogenesis and arteriogenesis correlating with increas
73  we determined that IL-12 disruption rescued angiogenesis and arteriogenesis in type 2 diabetic mice.
74 xpressed in retinal endothelial cells during angiogenesis and barrier formation.
75 reported to be required for normal forebrain angiogenesis and BBB function in mouse embryos, but the
76                                          CNS angiogenesis and blood-brain barrier integrity are contr
77 D4 signalling in vitro and recapitulates CNS angiogenesis and blood-CNS-barrier defects caused by imp
78 f the gliomas, is characterized by extensive angiogenesis and by an inflammatory microenvironment tha
79  glutathione-dependent oxidoreductase drives angiogenesis and cancer progression by promoting TGM2-de
80 ating cardiac remodeling, and (3) increasing angiogenesis and cell survival to protect against and li
81 F-Axxxb from the PT, inhibiting infundibular angiogenesis and diminishing lactotroph (LT) VEGFR2 expr
82 utionary functional redundancy to facilitate angiogenesis and ensure maintenance of uterine blood flo
83 ects in a VEGF-independent in vitro model of angiogenesis and exerts an additive antiangiogenic respo
84 epatic stellate cells in vitro, we evaluated angiogenesis and fibrosis gene expression.
85 ing MIF-shRNA grew more rapidly with greater angiogenesis and had macrophages localizing to the tumor
86 ly member that plays a pivotal role in brain angiogenesis and in ensuring a tight blood-brain barrier
87             The disruption of IL-12 promotes angiogenesis and increases blood flow recovery in obese
88 sive behaviour of endothelial cells to drive angiogenesis and increases invasiveness of cancer cells
89 ble factor)-1alpha is a major determinant of angiogenesis and induces VEGF transcription.
90        IGFBP7 abrogates tumors by inhibiting angiogenesis and inducing cancer-specific senescence and
91                                              Angiogenesis and inflammation are regarded as important
92 ibited primary tumor growth, inhibited tumor angiogenesis and inflammatory cell recruitment and, most
93 en-activated protein kinase signaling, tumor angiogenesis and inflammatory cell recruitment.
94 lin plays a crucial role in fibrogenesis and angiogenesis and is an important protein for tumor growt
95 vidence that IL-33 perpetuates inflammation, angiogenesis and lesion proliferation, which are critica
96  identify novel genes that control sprouting angiogenesis and lineage specification of blood vessels.
97                                   Controlled angiogenesis and lymphangiogenesis are essential for tis
98 rowth factor (VEGF)-D is capable of inducing angiogenesis and lymphangiogenesis through signaling via
99  proteolytic activity of APN promotes cancer angiogenesis and metastasis making it an important targe
100 cer, partly by driving metabolic adaptation, angiogenesis and metastasis through upregulation of hypo
101  take a center stage in promoting both tumor angiogenesis and metastatic spread.
102  mesenchymal stem cells (MSCs) can stimulate angiogenesis and modify immune function in experimental
103 AT3 signaling by VEGF165a and hence inhibits angiogenesis and perfusion recovery in PAD muscle.
104 novel role for EMCN as a potent regulator of angiogenesis and point to its potential as a new therape
105 stress and beta-adrenergic signaling promote angiogenesis and prostate cancer progression.
106 ance providing new mechanistic insights into angiogenesis and providing better understanding of glyci
107  Glioblastomas are lethal cancers defined by angiogenesis and pseudopalisading necrosis.
108 mpted to find the associations between tumor angiogenesis and radiomic imaging features from PET/MRI.
109 eased orthotopic tumor growth, reduced tumor angiogenesis and recruitment of inflammatory cells, and
110 ific signals in mammalian CNS ECs to promote angiogenesis and regulate the BBB.
111 among brain regions for its distinct pace of angiogenesis and selective vulnerability to hemorrhage d
112  suggesting that the contribution of EETs to angiogenesis and subsequent tumor growth may be attribut
113                                Developmental angiogenesis and the maintenance of the blood-brain barr
114  roles in disease, including KSHV-associated angiogenesis and the survival and growth of primary effu
115 therapeutic level of NO necessary to promote angiogenesis and to protect ECs against hydrogen peroxid
116 (miR93) has been shown to favorably modulate angiogenesis and to reduce tissue loss in genetic PAD mo
117 eceptor 2 (PAR2) that plays pivotal roles in angiogenesis and tumor development.
118    Endothelial LKB1 may regulate endothelial angiogenesis and tumor growth by modulating Sp1-mediated
119 pathologies are characterized by deregulated angiogenesis and unstable blood vessels.
120 y, chemotaxis and cytokine biosynthesis, and angiogenesis and vascular development in (adjusted P<0.1
121 l growth factor (VEGF) is the main driver of angiogenesis and vascular permeability via VEGF receptor
122 e many endothelial cell functions, including angiogenesis and vascular permeability.
123                                              Angiogenesis and vascular remodeling are essential for t
124 relationship between regional expressions of angiogenesis and VEGF, and localized radiomic features f
125 rlecan domain I have the potential to induce angiogenesis and wound healing.
126 sarcomas in the context of current models of angiogenesis and, in light of recent clinical trial data
127 or behaviors (proliferation, metastasis, and angiogenesis) and differential sensitivities to standard
128 ntributes to IFNalpha-mediated inhibition of angiogenesis, and disruption of this network results in
129  We assessed anastomotic leakage, mortality, angiogenesis, and inflammation.
130  co-opt macrophages to promote their growth, angiogenesis, and metastasis.
131 ey nodal point in coordinating tumor growth, angiogenesis, and metastatic spread in ccRCC.
132 imental functions by promoting tumor growth, angiogenesis, and subsequent metastasis development.
133 highlight the contributions of inflammation, angiogenesis, and the ECM to both "healthy" and "unhealt
134 processes such as oxidative phosphorylation, angiogenesis, and the p53 pathway.
135 2), inflammation (Cxcl5, 10, IL6, Ccl2), and angiogenesis (Angpt2) genes.
136 , and promoted microvessel sprouting from an angiogenesis animal model.
137 e cell growth, proliferation, and migration; angiogenesis; apoptosis; vascular tone; host defenses; a
138 etailed mechanisms of IFNalpha-mediated anti-angiogenesis are not completely understood.
139 tion, as well as models with induced corneal angiogenesis, are widely used to investigate the molecul
140 e polarization in ischemic muscle to enhance angiogenesis, arteriogenesis, and perfusion recovery in
141 nic acid pathway in macrophage polarization, angiogenesis, arteriogenesis, and perfusion recovery.
142 (-/-) to wild-type ischemic muscle decreased angiogenesis, arteriogenesis, and perfusion, whereas tra
143 R93 in miR106b-93-25(-/-) PAD mice increased angiogenesis, arteriogenesis, and the extent of perfusio
144 enhanced vascular permeability and increased angiogenesis as compared with those implanted in wild-ty
145  in the pancreas remarkable up-regulation of angiogenesis as downstream of the HIF signaling pathway
146 to the inhibitory effect of SFN on HCC tumor angiogenesis as well as tumor growth, and indicate that
147 l cells-1 and in vivo with the Matrigel plug angiogenesis assay in mice.
148                        Transwell chamber and angiogenesis assays were conducted to verify the effect
149 activity of ZO-1 in both ex vivo and in vivo angiogenesis assays.
150    Herein, we demonstrate that MSA inhibited angiogenesis at 2 microM, which falls in the range of mo
151 nce have been shown to specifically bind the angiogenesis biomarker alpha V beta 3 integrin.
152 owed that (1) Glycine significantly promoted angiogenesis both in vitro and in vivo and effectively p
153 pertrophic and hypoxic signals can stimulate angiogenesis, both of which stimulated SRC-2 expression
154 , KSHV miR-K6-5p expedites cell invasion and angiogenesis by activating the c-Met pathway.
155 of eosinophilia, epithelial hyperplasia, and angiogenesis by immunohistochemistry and RT-PCR.
156 K, via its product IP5, negatively regulates angiogenesis by inhibiting VEGF expression.
157  this study, we examined the role of EMCN in angiogenesis by modulating EMCN levels both in vivo and
158 iated ischemic tissue repair and therapeutic angiogenesis by studying their miRNA content and uptake.
159 document that TSP-4 mediates upregulation of angiogenesis by TGF-beta1.
160 a mechanistic insight into the regulation of angiogenesis by TLRs and confirm a central role of Fli1
161 role during embryo development and regulates angiogenesis, cardiovascular activity, and energy metabo
162 B, which are critical in cell proliferation, angiogenesis, development of metastasis, and survival.
163 s accompanied by reduced VEGF expression and angiogenesis, diminished numbers of PD-L1(+) cancer cell
164 or immune response, protection from hypoxia, angiogenesis, DNA repair, cell migration and invasivenes
165      FGFBP1 enhances FGF signaling including angiogenesis during cancer progression and is upregulate
166  findings showing that atypical E2Fs promote angiogenesis during fetal development in mice and zebraf
167 ion of EMCN in vivo led to the impairment of angiogenesis during normal retinal development in vivo.
168 harmacological inhibition of eNOS attenuates angiogenesis during tissue repair, resulting in delayed
169 ate development and is primarily achieved by angiogenesis - endothelial cell sprouting from pre-exist
170 he relevance of this pathway to VEGF-induced angiogenesis, especially in diabetic wound healing.
171            Maintenance of pregnancy requires angiogenesis, establishment of the correct cellular mili
172 ve oil consumption, is a strong inhibitor of angiogenesis ex vivo and in vivo.
173                                              Angiogenesis factor levels were compared between respond
174                              Nineteen plasma angiogenesis factors (angiopoietin 2; hepatocyte growth
175 diotracer and blood-sample tests to quantify angiogenesis factors (fibroblast growth factor, vascular
176 tify changes in a broad panel of circulating angiogenesis factors after bland transcatheter arterial
177                          Conclusion Multiple angiogenesis factors demonstrated significant upregulati
178                                     Fourteen angiogenesis factors showed statistically significant ch
179 s expressed by mast cells, and mitigated neo-angiogenesis formation in vitro.
180                      Animal models of ocular angiogenesis: from development to pathologies.
181 hanistic link between LVAD support, abnormal angiogenesis, gastrointestinal angiodysplasia, and bleed
182 abolishes well-known apelin effects, such as angiogenesis, glucose tolerance, and vasodilatation.
183                                              Angiogenesis, growth and metastasis in ovarian tumour xe
184  that lal(-/-) ECs facilitated in vivo tumor angiogenesis, growth, and metastasis, largely by stimula
185                                    Targeting angiogenesis has emerged as a promising strategy for can
186 ace of immune cells, but its contribution to angiogenesis has not been studied.
187                                              Angiogenesis has traditionally been viewed from the pers
188  To identify material properties that impact angiogenesis, here we have developed an in vitro model w
189                                   RATIONALE: Angiogenesis improves perfusion to the ischemic tissue a
190 (VEGFA) is a key factor in the regulation of angiogenesis in adipose tissue.
191 our surprise, atypical E2Fs suppressed tumor angiogenesis in all three cancer models, which is in a s
192                                    Disrupted angiogenesis in aortic rings from VimKO mice and in endo
193  AND Deletion of IPMK in fibroblasts induces angiogenesis in both in vitro and in vivo models.
194 showed that CXCL12 dose-dependently enhanced angiogenesis in chorioallantoic membranes (CAMs) and HUV
195  (PGE2) is associated with proliferation and angiogenesis in colorectal tumours.
196 r, our findings suggest that RPS15A promotes angiogenesis in HCCs by enhancing Wnt/beta-catenin induc
197 transcription (STAT)-3 signaling to decrease angiogenesis in human and experimental PAD.
198 ining showed decreased fibrosis and improved angiogenesis in hypoxic groups compared with controls.
199 ells are a promising cell type for enhancing angiogenesis in ischemic cardiovascular tissues.
200  interactions that influence tissue-specific angiogenesis in mammals.
201 c-RGD conjugate 3 strongly inhibited in vivo angiogenesis in Matrigel plug assays in FVB mice.
202 se disorders, highlighting the need to study angiogenesis in more detail.
203 yze biochemical and biomechanical drivers of angiogenesis in murine corneal neovessels.
204 endothelial cell activation and pathological angiogenesis in our previous study, but the mechanisms b
205 lycolysis, which is crucial for pathological angiogenesis in proliferative retinopathies.
206 r pregnancy complications and placental anti-angiogenesis in response to Aroclor 1254 (A-1254), a mix
207 g a novel pathway that mediates induction of angiogenesis in response to TGF-beta1.
208 key regulator of vascular cell migration and angiogenesis in SM.
209 d effector molecules triggering fibrosis and angiogenesis in SM.
210                    Q8 inhibits developmental angiogenesis in Tg(fli1:EGFP) zebrafish and inhibits hum
211 cruitment and activation of fibroblasts, and angiogenesis in the granulation tissue.
212 s using rAAV1-mediated CRISPR/Cas9 abrogates angiogenesis in the mouse models of oxygen-induced retin
213 genesis, in retinal endothelium and abrogate angiogenesis in the mouse models of oxygen-induced retin
214 t adenosine receptor A2A drives pathological angiogenesis in the oxygen-induced retinopathy mouse mod
215 l mechanism linking IL-6 trans-signaling and angiogenesis in the peritoneal membrane.
216 nt on pathological angiogenesis.Pathological angiogenesis in the retina is a major cause of blindness
217 Here, we investigate whether T cells promote angiogenesis in transplantation.
218 om these cells had reduced ability to induce angiogenesis in vitro.
219  mevastatin stimulated epithelialization and angiogenesis in vivo Mevastatin also reversed FPP-mediat
220 logy to silence VEGFR2, a major regulator of angiogenesis, in retinal endothelium and abrogate angiog
221         This work aimed to study hypoxia and angiogenesis, in vivo and in situ, in patients admitted
222                                              Angiogenesis, in which vascular endothelial growth facto
223              Models of retinal and choroidal angiogenesis, including oxygen-induced retinopathy, lase
224              Targeting factors that regulate angiogenesis, including the potent promoter vascular end
225 determinant of the magnitude and duration of angiogenesis induced by vascular endothelial growth fact
226 (ECs) are widely used to study mechanisms of angiogenesis, inflammation, and endothelial permeability
227 d in many physiological processes, including angiogenesis, inflammatory responses, and vascular stabi
228 tition model had to be rejected, whereas the angiogenesis inhibition and proliferation inhibition mod
229 e of hydroxytyrosol for angio-prevention and angiogenesis inhibition in clinical setting.
230  represses thrombospondin-1 (TSP1), a potent angiogenesis inhibitor, through epigenetic regulation.
231    These results suggest that 3TSR, or other angiogenesis inhibitors, can be repurposed for TSP1 repl
232 pproaches targeting the DNA damage response, angiogenesis inhibitors, immune checkpoint inhibitors, o
233 impact as a noninvasive method for assessing angiogenesis inhibitors.
234 ly indicator of response to tyrosine kinase (angiogenesis) inhibitors such as pazopanib in ovarian ca
235                                              Angiogenesis is a complex, multicellular process that is
236                                              Angiogenesis is a crucial component of the vascular resp
237                                              Angiogenesis is central to both normal and pathologic pr
238 We previously reported that ischemia-induced angiogenesis is compromised in type 2 diabetic mice.
239                                              Angiogenesis is dysregulated in various pathologies, inc
240 ameters through a VEGF-independent mechanism.Angiogenesis is essential for solid tumor progression.
241                                    Sustained angiogenesis is essential for the development of solid t
242 ar endothelial growth factor (VEGF)-mediated angiogenesis is inhibited upon reduction of NO bioactivi
243 r study adds evidence to the hypothesis that angiogenesis is involved in the initiation of tissue inf
244 ivated in multiple cases where sinus venosus angiogenesis is stunted.
245 transcription factor implicated in sprouting angiogenesis) is required for its VEGF-mediated inductio
246 ng inflammation, glial cell dysfunction, and angiogenesis, its role in the retina and the factors tha
247                               During retinal angiogenesis, KO mice exhibit a 50% decrease in endothe
248  properties, immunomodulation, and increased angiogenesis, leading to resistance to therapy.
249 ha1 catalytic subunit has been implicated in angiogenesis, little is known about the role played by t
250 3-fold increases in tumor mass and levels of angiogenesis markers in animals injected with TGF-beta1,
251 d alphaSMA), in addition to inflammation and angiogenesis markers in the treatment group (p < 0.05).
252 s chemokines, cytokines, growth factors, and angiogenesis mediators, with CXCL12 among the most signi
253 n 18 major representative networks including angiogenesis, metabolism, and immunity.
254 P members mediated OS development, including angiogenesis, migration and invasion.
255 enin overexpression decreased proliferation, angiogenesis, migration, and invasion.
256                     Our results from in vivo angiogenesis models and cultured EC document that TSP-4
257 ssues by combining in vitro neurogenesis and angiogenesis models using a microfluidic platform, which
258                              In pathological angiogenesis of human ovarian carcinomas, reduced VEC ex
259 and other diseases dependent on pathological angiogenesis.Pathological angiogenesis in the retina is
260 th VEGF and hypoxia inducible factor related angiogenesis pathways, with special emphasis on DA produ
261 geneity in cancer cell phenotypes, including angiogenesis, proliferation, and stemness, and a minor s
262 oadly promote RNAPII pause release and drive angiogenesis.Promoter proximal RNAPII pausing is a rate-
263 acitinib disrupted adventitial microvascular angiogenesis, reduced outgrowth of hyperplastic intima,
264  the hallmark features of pathologic retinal angiogenesis, reducing neovascular tuft formation and in
265 utic application in regenerative medicine or angiogenesis-related diseases is drawing increasing inte
266 ts potential as a new therapeutic target for angiogenesis-related diseases.
267 ncept of how the covalent conjugation of two angiogenesis-related small modules may result in novel a
268 erventions that target signaling pathways in angiogenesis relevant to cancer, vascular diseases, and
269 r roles in KSHV-induced tumor metastasis and angiogenesis remain largely unclear.
270 gy, and the downstream mechanism of CXCR7 in angiogenesis remains unclear.
271                  Further an in vivo model of angiogenesis, showed a significant reduction of haemoglo
272 actions participate in neuronal development, angiogenesis, spinal cord injury, viral invasion, and im
273                                              Angiogenesis study was evaluated in vitro with human mic
274                               Tumors exploit angiogenesis, the formation of new blood vessels from pr
275 tant physiological stress signal that drives angiogenesis, the formation of new blood vessels.
276 d (NDP) of a molecular pathway that controls angiogenesis, the Norrin-beta-catenin signaling pathway.
277 le; the role of inflammation in fibrosis and angiogenesis; the factors that contribute to the initiat
278                      Inhibiting pathological angiogenesis therefore represents a promising therapeuti
279 ential bias of intratumoral heterogeneity of angiogenesis, this study investigated the relationship b
280 s (MVs), activates VEGF receptors and tumour angiogenesis through a unique 90 kDa form of VEGF (VEGF9
281  in vivo studies have shown that DA inhibits angiogenesis through activation of the DA receptor type
282 esenchymal transition, stromal reaction, and angiogenesis through autocrine and paracrine PDGFRbeta s
283 h an AQP3-dependent mechanism that activates angiogenesis, triggers collagen and hyaluronic acid synt
284 P as a possible druggable target to modulate angiogenesis under pathological conditions.
285 D40L, IL-7, CCL25, IL-2RB, IL-15RA, CD6) and angiogenesis (VEGF-A) were significantly increased only
286                             We also examined angiogenesis (VEGFR2, p-ERK, p-PLCr1/2), hedgehog (Gli1,
287 immunosuppressive tumor microenvironment and angiogenesis via cytokine production; (ii) functioning a
288 atial relationship between tumor hypoxia and angiogenesis was assessed by an overlap analysis of the
289                    We found that NO-mediated angiogenesis was blocked by inhibition of VEGF with sFlt
290 cs ischemic/inflammatory neovascularization, angiogenesis was dramatically upregulated in Apoa1bp(-/-
291                      The initiation phase of angiogenesis was not associated with classical endotheli
292 n of in vitro and in vivo assays to evaluate angiogenesis, we demonstrated that human limbal epitheli
293  better understanding of glycine function in angiogenesis, which may provide valuable information for
294 ls leads to increased cell proliferation and angiogenesis, which may, in turn, contribute to positive
295                                 Coordinating angiogenesis with acquisition of tissue-specific propert
296                       By stimulating corneal angiogenesis with an alkali burn in Tie2-GFP fluorescent
297 stable molecules, which impair tumor-related angiogenesis with equal or even superior ability as comp
298 sed to build simulations of tumor growth and angiogenesis with realistic vessel networks.
299 nd exosomes from various cellular origins in angiogenesis, with a particular emphasis on the underlyi
300  effects on cancer prevention by influencing angiogenesis within Se nutritional levels.

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