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1 nding of the complex contribution of CtsB to angiogenesis.
2 herapy represents a proven strategy to treat angiogenesis.
3 thylation and other PTMs in regulating tumor angiogenesis.
4 l miRNA-based therapeutic strategies against angiogenesis.
5 othelial cell proliferation to promote tumor angiogenesis.
6 o decreased the weights of tumors and cancer angiogenesis.
7 rmation, all of which are important steps in angiogenesis.
8 x and has a crucial role in fibrogenesis and angiogenesis.
9 tified regulatory pathway that could promote angiogenesis.
10 ase of reactive oxygen species and decreased angiogenesis.
11 ing and therefore as important regulators of angiogenesis.
12 have implications for alcohol-induced tumor angiogenesis.
13 ls in driving physiological and pathological angiogenesis.
14 by endothelium and are implicated in tumour angiogenesis.
15 are crucial for a complete understanding of angiogenesis.
16 et activation and inducing cell invasion and angiogenesis.
17 that contributes to tumour cell invasion and angiogenesis.
18 ility of targeting RUNX1 in aberrant retinal angiogenesis.
19 f adverse myocardial remodeling and promotes angiogenesis.
20 ous biochemical factors on tumour growth and angiogenesis.
21 elevated blood vessel sprouting and in vivo angiogenesis.
22 nt and maintenance of the vasculature and in angiogenesis.
23 ating apoptosis, inflammation, fibrosis, and angiogenesis.
24 PlGF pathways in the regulation of placental angiogenesis.
25 aracterized of accelerated postnatal retinal angiogenesis.
26 s emphasize that eNOS-derived NO can promote angiogenesis.
27 Endothelial metabolism is a key regulator of angiogenesis.
28 ngly promoted endothelial cells invasion and angiogenesis.
29 ibroblast and myofibroblast accumulation and angiogenesis.
30 ical for regulation of inflammation mediated angiogenesis.
31 othelial cells controls zebra fish embryonic angiogenesis.
32 the TLR9 ligand class C, ODN2395, increased angiogenesis.
33 ing, mediates immune responses, and triggers angiogenesis.
34 colonic surgery, possibly via its effects on angiogenesis.
35 endothelial fatty acid metabolism and tumour angiogenesis.
36 yme in glutamine catabolism, markedly blunts angiogenesis.
37 d from trophoblasts is involved in placental angiogenesis.
38 fic biomarkers, many of which have a role in angiogenesis.
39 suggesting a role for Cavin-2 in pathogenic angiogenesis.
40 autophagy in endothelial cells and attenuate angiogenesis.
41 endothelial growth factor, a major driver of angiogenesis.
42 tissue progenitor cell differentiation, and angiogenesis.
43 ealing in diabetic mice through facilitating angiogenesis.
44 ssing the extracellular matrix and promoting angiogenesis.
45 evaluating biliary mass, liver fibrosis, and angiogenesis.
46 ific alpha3 knockout mice displayed impaired angiogenesis.
47 bility and turnover in blood vessels impacts angiogenesis.
48 diates ischemic tissue repair via beneficial angiogenesis.
49 mor efficacy of bevacizumab, an inhibitor of angiogenesis.
50 he current available animal models of ocular angiogenesis.
51 lation of cell migration, proliferation, and angiogenesis.
52 ional pathway contributing to VEGF-dependent angiogenesis.
53 ndothelial cell proliferation during retinal angiogenesis.
54 l-cell adhesion, which are all requisite for angiogenesis.
55 ed a potential role of PTP1B in VEGF-induced angiogenesis.
56 ing several genes important for invasion and angiogenesis.
57 icellular, strand-like invasion required for angiogenesis.
58 s well as enhanced fibrinolysis and impaired angiogenesis.
59 ream effectors and are pivotal regulators of angiogenesis.
60 validating modulation of paxillin to inhibit angiogenesis.
61 cal-recognition motifs for cell adhesion and angiogenesis.
62 and stromal cells, and iii) cancer-regulated angiogenesis.
63 ecreased lung compliance, and decreased lung angiogenesis.
64 rway smooth muscle hyperplasia and increased angiogenesis.
65 investigated the role of IPMK in regulating angiogenesis.
66 ys including hedgehog, and mTOR pathways and angiogenesis.
67 is conceptually similar to hypoxia-triggered angiogenesis.
68 lls typically rely on aerobic glycolysis for angiogenesis.
69 CIB1 in regulating cancer cell survival and angiogenesis, although the mechanisms involved have rema
70 The inhibition of inflammation-associated angiogenesis ameliorates inflammatory diseases by reduci
71 e show that Cavin-2 is required for in vitro angiogenesis and also for endothelial cell proliferation
72 t mice (miR106b-93-25(-/-)) showed decreased angiogenesis and arteriogenesis correlating with increas
73 we determined that IL-12 disruption rescued angiogenesis and arteriogenesis in type 2 diabetic mice.
75 reported to be required for normal forebrain angiogenesis and BBB function in mouse embryos, but the
77 D4 signalling in vitro and recapitulates CNS angiogenesis and blood-CNS-barrier defects caused by imp
78 f the gliomas, is characterized by extensive angiogenesis and by an inflammatory microenvironment tha
79 glutathione-dependent oxidoreductase drives angiogenesis and cancer progression by promoting TGM2-de
80 ating cardiac remodeling, and (3) increasing angiogenesis and cell survival to protect against and li
81 F-Axxxb from the PT, inhibiting infundibular angiogenesis and diminishing lactotroph (LT) VEGFR2 expr
82 utionary functional redundancy to facilitate angiogenesis and ensure maintenance of uterine blood flo
83 ects in a VEGF-independent in vitro model of angiogenesis and exerts an additive antiangiogenic respo
85 ing MIF-shRNA grew more rapidly with greater angiogenesis and had macrophages localizing to the tumor
86 ly member that plays a pivotal role in brain angiogenesis and in ensuring a tight blood-brain barrier
88 sive behaviour of endothelial cells to drive angiogenesis and increases invasiveness of cancer cells
92 ibited primary tumor growth, inhibited tumor angiogenesis and inflammatory cell recruitment and, most
94 lin plays a crucial role in fibrogenesis and angiogenesis and is an important protein for tumor growt
95 vidence that IL-33 perpetuates inflammation, angiogenesis and lesion proliferation, which are critica
96 identify novel genes that control sprouting angiogenesis and lineage specification of blood vessels.
98 rowth factor (VEGF)-D is capable of inducing angiogenesis and lymphangiogenesis through signaling via
99 proteolytic activity of APN promotes cancer angiogenesis and metastasis making it an important targe
100 cer, partly by driving metabolic adaptation, angiogenesis and metastasis through upregulation of hypo
102 mesenchymal stem cells (MSCs) can stimulate angiogenesis and modify immune function in experimental
104 novel role for EMCN as a potent regulator of angiogenesis and point to its potential as a new therape
106 ance providing new mechanistic insights into angiogenesis and providing better understanding of glyci
108 mpted to find the associations between tumor angiogenesis and radiomic imaging features from PET/MRI.
109 eased orthotopic tumor growth, reduced tumor angiogenesis and recruitment of inflammatory cells, and
111 among brain regions for its distinct pace of angiogenesis and selective vulnerability to hemorrhage d
112 suggesting that the contribution of EETs to angiogenesis and subsequent tumor growth may be attribut
114 roles in disease, including KSHV-associated angiogenesis and the survival and growth of primary effu
115 therapeutic level of NO necessary to promote angiogenesis and to protect ECs against hydrogen peroxid
116 (miR93) has been shown to favorably modulate angiogenesis and to reduce tissue loss in genetic PAD mo
118 Endothelial LKB1 may regulate endothelial angiogenesis and tumor growth by modulating Sp1-mediated
120 y, chemotaxis and cytokine biosynthesis, and angiogenesis and vascular development in (adjusted P<0.1
121 l growth factor (VEGF) is the main driver of angiogenesis and vascular permeability via VEGF receptor
124 relationship between regional expressions of angiogenesis and VEGF, and localized radiomic features f
126 sarcomas in the context of current models of angiogenesis and, in light of recent clinical trial data
127 or behaviors (proliferation, metastasis, and angiogenesis) and differential sensitivities to standard
128 ntributes to IFNalpha-mediated inhibition of angiogenesis, and disruption of this network results in
132 imental functions by promoting tumor growth, angiogenesis, and subsequent metastasis development.
133 highlight the contributions of inflammation, angiogenesis, and the ECM to both "healthy" and "unhealt
137 e cell growth, proliferation, and migration; angiogenesis; apoptosis; vascular tone; host defenses; a
139 tion, as well as models with induced corneal angiogenesis, are widely used to investigate the molecul
140 e polarization in ischemic muscle to enhance angiogenesis, arteriogenesis, and perfusion recovery in
141 nic acid pathway in macrophage polarization, angiogenesis, arteriogenesis, and perfusion recovery.
142 (-/-) to wild-type ischemic muscle decreased angiogenesis, arteriogenesis, and perfusion, whereas tra
143 R93 in miR106b-93-25(-/-) PAD mice increased angiogenesis, arteriogenesis, and the extent of perfusio
144 enhanced vascular permeability and increased angiogenesis as compared with those implanted in wild-ty
145 in the pancreas remarkable up-regulation of angiogenesis as downstream of the HIF signaling pathway
146 to the inhibitory effect of SFN on HCC tumor angiogenesis as well as tumor growth, and indicate that
150 Herein, we demonstrate that MSA inhibited angiogenesis at 2 microM, which falls in the range of mo
152 owed that (1) Glycine significantly promoted angiogenesis both in vitro and in vivo and effectively p
153 pertrophic and hypoxic signals can stimulate angiogenesis, both of which stimulated SRC-2 expression
157 this study, we examined the role of EMCN in angiogenesis by modulating EMCN levels both in vivo and
158 iated ischemic tissue repair and therapeutic angiogenesis by studying their miRNA content and uptake.
160 a mechanistic insight into the regulation of angiogenesis by TLRs and confirm a central role of Fli1
161 role during embryo development and regulates angiogenesis, cardiovascular activity, and energy metabo
162 B, which are critical in cell proliferation, angiogenesis, development of metastasis, and survival.
163 s accompanied by reduced VEGF expression and angiogenesis, diminished numbers of PD-L1(+) cancer cell
164 or immune response, protection from hypoxia, angiogenesis, DNA repair, cell migration and invasivenes
165 FGFBP1 enhances FGF signaling including angiogenesis during cancer progression and is upregulate
166 findings showing that atypical E2Fs promote angiogenesis during fetal development in mice and zebraf
167 ion of EMCN in vivo led to the impairment of angiogenesis during normal retinal development in vivo.
168 harmacological inhibition of eNOS attenuates angiogenesis during tissue repair, resulting in delayed
169 ate development and is primarily achieved by angiogenesis - endothelial cell sprouting from pre-exist
170 he relevance of this pathway to VEGF-induced angiogenesis, especially in diabetic wound healing.
175 diotracer and blood-sample tests to quantify angiogenesis factors (fibroblast growth factor, vascular
176 tify changes in a broad panel of circulating angiogenesis factors after bland transcatheter arterial
181 hanistic link between LVAD support, abnormal angiogenesis, gastrointestinal angiodysplasia, and bleed
182 abolishes well-known apelin effects, such as angiogenesis, glucose tolerance, and vasodilatation.
184 that lal(-/-) ECs facilitated in vivo tumor angiogenesis, growth, and metastasis, largely by stimula
188 To identify material properties that impact angiogenesis, here we have developed an in vitro model w
191 our surprise, atypical E2Fs suppressed tumor angiogenesis in all three cancer models, which is in a s
194 showed that CXCL12 dose-dependently enhanced angiogenesis in chorioallantoic membranes (CAMs) and HUV
196 r, our findings suggest that RPS15A promotes angiogenesis in HCCs by enhancing Wnt/beta-catenin induc
198 ining showed decreased fibrosis and improved angiogenesis in hypoxic groups compared with controls.
204 endothelial cell activation and pathological angiogenesis in our previous study, but the mechanisms b
206 r pregnancy complications and placental anti-angiogenesis in response to Aroclor 1254 (A-1254), a mix
212 s using rAAV1-mediated CRISPR/Cas9 abrogates angiogenesis in the mouse models of oxygen-induced retin
213 genesis, in retinal endothelium and abrogate angiogenesis in the mouse models of oxygen-induced retin
214 t adenosine receptor A2A drives pathological angiogenesis in the oxygen-induced retinopathy mouse mod
216 nt on pathological angiogenesis.Pathological angiogenesis in the retina is a major cause of blindness
219 mevastatin stimulated epithelialization and angiogenesis in vivo Mevastatin also reversed FPP-mediat
220 logy to silence VEGFR2, a major regulator of angiogenesis, in retinal endothelium and abrogate angiog
225 determinant of the magnitude and duration of angiogenesis induced by vascular endothelial growth fact
226 (ECs) are widely used to study mechanisms of angiogenesis, inflammation, and endothelial permeability
227 d in many physiological processes, including angiogenesis, inflammatory responses, and vascular stabi
228 tition model had to be rejected, whereas the angiogenesis inhibition and proliferation inhibition mod
230 represses thrombospondin-1 (TSP1), a potent angiogenesis inhibitor, through epigenetic regulation.
231 These results suggest that 3TSR, or other angiogenesis inhibitors, can be repurposed for TSP1 repl
232 pproaches targeting the DNA damage response, angiogenesis inhibitors, immune checkpoint inhibitors, o
234 ly indicator of response to tyrosine kinase (angiogenesis) inhibitors such as pazopanib in ovarian ca
238 We previously reported that ischemia-induced angiogenesis is compromised in type 2 diabetic mice.
240 ameters through a VEGF-independent mechanism.Angiogenesis is essential for solid tumor progression.
242 ar endothelial growth factor (VEGF)-mediated angiogenesis is inhibited upon reduction of NO bioactivi
243 r study adds evidence to the hypothesis that angiogenesis is involved in the initiation of tissue inf
245 transcription factor implicated in sprouting angiogenesis) is required for its VEGF-mediated inductio
246 ng inflammation, glial cell dysfunction, and angiogenesis, its role in the retina and the factors tha
249 ha1 catalytic subunit has been implicated in angiogenesis, little is known about the role played by t
250 3-fold increases in tumor mass and levels of angiogenesis markers in animals injected with TGF-beta1,
251 d alphaSMA), in addition to inflammation and angiogenesis markers in the treatment group (p < 0.05).
252 s chemokines, cytokines, growth factors, and angiogenesis mediators, with CXCL12 among the most signi
257 ssues by combining in vitro neurogenesis and angiogenesis models using a microfluidic platform, which
259 and other diseases dependent on pathological angiogenesis.Pathological angiogenesis in the retina is
260 th VEGF and hypoxia inducible factor related angiogenesis pathways, with special emphasis on DA produ
261 geneity in cancer cell phenotypes, including angiogenesis, proliferation, and stemness, and a minor s
262 oadly promote RNAPII pause release and drive angiogenesis.Promoter proximal RNAPII pausing is a rate-
263 acitinib disrupted adventitial microvascular angiogenesis, reduced outgrowth of hyperplastic intima,
264 the hallmark features of pathologic retinal angiogenesis, reducing neovascular tuft formation and in
265 utic application in regenerative medicine or angiogenesis-related diseases is drawing increasing inte
267 ncept of how the covalent conjugation of two angiogenesis-related small modules may result in novel a
268 erventions that target signaling pathways in angiogenesis relevant to cancer, vascular diseases, and
272 actions participate in neuronal development, angiogenesis, spinal cord injury, viral invasion, and im
276 d (NDP) of a molecular pathway that controls angiogenesis, the Norrin-beta-catenin signaling pathway.
277 le; the role of inflammation in fibrosis and angiogenesis; the factors that contribute to the initiat
279 ential bias of intratumoral heterogeneity of angiogenesis, this study investigated the relationship b
280 s (MVs), activates VEGF receptors and tumour angiogenesis through a unique 90 kDa form of VEGF (VEGF9
281 in vivo studies have shown that DA inhibits angiogenesis through activation of the DA receptor type
282 esenchymal transition, stromal reaction, and angiogenesis through autocrine and paracrine PDGFRbeta s
283 h an AQP3-dependent mechanism that activates angiogenesis, triggers collagen and hyaluronic acid synt
285 D40L, IL-7, CCL25, IL-2RB, IL-15RA, CD6) and angiogenesis (VEGF-A) were significantly increased only
287 immunosuppressive tumor microenvironment and angiogenesis via cytokine production; (ii) functioning a
288 atial relationship between tumor hypoxia and angiogenesis was assessed by an overlap analysis of the
290 cs ischemic/inflammatory neovascularization, angiogenesis was dramatically upregulated in Apoa1bp(-/-
292 n of in vitro and in vivo assays to evaluate angiogenesis, we demonstrated that human limbal epitheli
293 better understanding of glycine function in angiogenesis, which may provide valuable information for
294 ls leads to increased cell proliferation and angiogenesis, which may, in turn, contribute to positive
297 stable molecules, which impair tumor-related angiogenesis with equal or even superior ability as comp
299 nd exosomes from various cellular origins in angiogenesis, with a particular emphasis on the underlyi
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