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1 iodontal disease and chronic CAD as assessed angiographically.
2 E did not demonstrate any neovascularization angiographically.
3 networks have been detected in human tumors angiographically.
4 ention in-stent tissue, and (3) was not seen angiographically.
5 tratify cardiac risk have not been validated angiographically.
6 rabbits; arterial occlusions were documented angiographically.
7 eous coronary intervention that are not seen angiographically.
8 cute myocardial infarction (MI) were studied angiographically; 1,848 patients had coronary artery dis
10 31) underwent coronary angiography; 781 were angiographically acceptable; 454 (58% of eligible) patie
13 e is particularly helpful in intermediate or angiographically ambiguous lesions in the absence of non
18 but also in 12 (15%) of 80 segments without angiographically apparent coronary disease (p = 0.004, a
19 e suppression and correlated the presence of angiographically apparent plaque with (18)F-FDG uptake i
21 ents suffering severe late rejection develop angiographically apparent TxCAD rapidly and must be moni
23 sease (prior myocardial infarction or proven angiographically) between September 2011 and November 20
24 raphic score, as measured histologically and angiographically, compared with vehicle or empty viral v
25 ous coronary interventions for patients with angiographically complex lesions confers additive long-t
28 , 1.52; 95% confidence interval, 1.17-1.98), angiographically confirmed angina (1.91; 1.59-2.29), cor
29 ression analyses utilizing the data from 256 angiographically confirmed CAD patients and 250 non-CAD
30 atients from Kuopio University Hospital with angiographically confirmed CHD and 250 age-, gender-, an
31 e patients with ACS and 12 CSA patients with angiographically confirmed coronary artery disease and 9
33 ult volunteers and seven patients with X-ray angiographically confirmed coronary artery disease under
34 ry intima-media thickness, 442 patients with angiographically confirmed coronary artery disease, and
36 plasma leptin and prognosis in patients with angiographically confirmed coronary atherosclerosis.
38 We included adults aged 18-85 years with angiographically confirmed large vessel occlusion stroke
39 45 years +/- 10.4 [standard deviation]) with angiographically confirmed Moyamoya (n = 8) or internal
41 plicated in only one death in a patient with angiographically-confirmed PE at initial presentation.
42 lood sera was reported previously to predict angiographically defined advanced coronary artery diseas
43 domly assigned 423 postmenopausal women with angiographically defined atherosclerosis (321 women had
45 predictive power of the same methodology for angiographically defined CAD using plasma samples from g
46 n the HSP70-2 gene +1267A>G polymorphism and angiographically defined CAD within an Iranian populatio
50 med an association between 9p21 and CAD with angiographically defined cases and control subjects (poo
54 sk factors with the presence and severity of angiographically defined coronary atherosclerosis was an
57 ficant burden of ischemia remains even after angiographically defined successful revascularization.
58 iabetic nonsmoking patients (n = 4,811) with angiographically defined, clinically significant CAD (>
59 y was to investigate the association between angiographically-defined CAD and periodontal disease.
61 in women with myocardial infarction without angiographically demonstrable obstructive coronary arter
62 ferent myocardial diseases in the absence of angiographically demonstrable stenosis of the epicardial
63 t mortality in 985 consecutive patients with angiographically demonstrated CAD (stenosis >/=70%).
64 uals with stable presentation and those with angiographically demonstrated clean coronaries are not a
66 n evaluating the physiologic significance of angiographically detectable coronary artery stenoses via
69 dial perfusion defects on SPECT studies with angiographically detected CAD and with human expert visu
71 ory of complex lesions and specifically with angiographically detected ICT and decreased TIMI flow.
72 to test the ability to predict the extent of angiographically determined coronary artery disease (CAD
74 nd CABG arms of the all-comers SYNTAX trial, angiographically determined ICR has a detrimental impact
77 dy of 734 type 2 diabetes patients (322 with angiographically diagnosed CAD and 412 with no evidence
79 l determinants of mortality in patients with angiographically diagnosed ischemic or nonischemic cardi
80 a are from examinations of 375 patients with angiographically diagnosed PE who participated in the Pr
81 t late lumen loss was 0.27 mm (SD 0.37), and angiographically discernable vasomotion was documented i
83 sion showed that log score but not number of angiographically diseased vessels significantly predicte
84 e CAD, we monitored 167 stable patients with angiographically documented 3-vessel CAD (average follow
88 (age 63+/-10 years, 230 men) of whom 289 had angiographically documented coronary artery disease (> o
89 otal of 95 patients with angina pectoris and angiographically documented coronary artery disease were
90 e 2 diabetes mellitus and clinically stable, angiographically documented coronary artery disease were
92 therapy; these men had angina (determined by angiographically documented coronary artery disease).
94 2.3 years) with chronic stable angina due to angiographically documented coronary artery disease, all
95 of oxidized LDL are strongly associated with angiographically documented coronary artery disease, par
96 oronary atherosclerosis in 825 patients with angiographically documented coronary artery disease.
98 trial was performed recruiting patients with angiographically documented coronary disease (n=96) and
102 suspected renovascular hypertension and (b) angiographically documented hemodynamically significant
104 re measured in 405 consecutive patients with angiographically documented multivessel coronary disease
105 valuable prognostic marker in patients with angiographically documented single- and double-vessel di
106 de polymorphism rs2383206 and CAD defined as angiographically documented stenosis greater than 50% in
108 d patients with type 2 diabetes mellitus and angiographically documented, stable coronary disease to
110 our weeks later, vasoreactivity was assessed angiographically during infusion of acetylcholine (Ach)
111 further medical therapy (307), and 781 were angiographically eligible for random allocation; 454 of
113 The observed close correlation between an angiographically established parameter of flow-dependent
116 owing heart transplantation do not result in angiographically evident acceleration of transplant CAD.
118 a sensitive but not a specific indicator of angiographically evident atherosclerosis; sensitivity is
119 cations and to determine the relationship of angiographically evident complications to elevations of
120 the association of cardiac risk factors and angiographically evident coronary artery disease with co
122 otic changes, even before the development of angiographically evident endothelial dysfunction; theref
125 ath, revascularization of the target lesion, angiographically evident thrombosis, or myocardial infar
126 esized that combination therapy would reduce angiographically evident thrombus (AET) and would increa
127 myocardial infarction in 41.4% of the cases, angiographically evident thrombus in 20.9%, and abrupt o
128 inine, compared to placebo-treated controls; angiographically evident vascularity in the ischemic lim
129 single image, now affords us the ability to angiographically examine the parts of the retina previou
130 ed in situ for 5 days, at which time animals angiographically exhibiting thrombus were randomly assig
131 patients without acute MI who were assessed angiographically for coronary artery disease (CAD) and w
132 revious treatment were included and observed angiographically for up to 18 months and clinically for
135 ce has demonstrated a modest benefit with an angiographically guided approach; but patients having ne
137 guided approach was superior to the standard angiographically-guided approach for percutaneous revasc
141 the photosensitizer verteporfin was assessed angiographically in CNV lesions, to determine the optima
144 to distal microembolization or spasm, and/or angiographically inapparent dissection or residual steno
146 und (IVUS)-guided strategy for patients with angiographically indeterminate left main coronary artery
147 r ultrasound is an accurate method to assess angiographically indeterminate lesions of the LMCA.
148 conducted IVUS studies on 214 patients with angiographically indeterminate LMCA lesions, and deferra
150 associated with a higher probability that an angiographically intermediate coronary stenosis is funct
151 Evidence: A recommendation to perform FFR in angiographically intermediate stenoses in the absence of
153 ysfunction in arteries not yet clinically or angiographically involved in thromboangiitis obliterans.
155 ery prevalence, 48% versus 24%; P<0.001) and angiographically (mean Japan-chronic total occlusion sco
156 tin plus niacin provides marked clinical and angiographically measurable benefits in patients with co
157 esions responsible for follow-up events were angiographically mild at baseline (mean [+/-SD] diameter
159 ble for unanticipated events were frequently angiographically mild, most were thin-cap fibroatheromas
162 age, 53.0+/-10.1 years) with chest pain and angiographically nonsignificant coronary artery disease
163 patients during cardiac catheterization with angiographically normal anterior descending arteries.
164 s: 85 patients with chest pain syndromes and angiographically normal arteries (group 1); 21 patients
165 Coronary vasodilatory reserve was higher in angiographically normal arteries in patients with chest
166 vasodilatory reserve values in patients with angiographically normal arteries who had atypical chest
167 able coronary artery disease (CAD) (n = 17), angiographically normal coronary arteries (n = 8), and f
168 rs (healthy control subjects), patients with angiographically normal coronary arteries (patient contr
169 rding the range of normal values obtained in angiographically normal coronary arteries in patients wi
170 /- 10 years) with angina-like chest pain and angiographically normal coronary arteries underwent exer
171 onary artery disease and in 12 subjects with angiographically normal coronary arteries who were free
175 ssels to be distinguished from subjects with angiographically normal coronary arteries, with a specif
181 ltrasound was performed on 121 patients with angiographically normal LMCAs to determine the lower ran
182 ore stenting in the culprit vessel and in an angiographically normal nonculprit vessel in patients wi
183 iography for assessment of stable angina had angiographically normal or near normal coronary arteriog
187 n vascular pathology, diabetic patients with angiographically normal retinas have been found to exhib
188 a deleterious effect of gamma-irradiation on angiographically normal uninjured reference segments in
189 city and MPR ratios between poststenotic and angiographically normal vascular beds were comparably re
193 algorithm for noninvasive identification of angiographically obstructive three-vessel and/or left ma
197 agnetic resonance images in 21 patients with angiographically proved dural AV fistula of the cavernou
198 nd pulmonary angiograms of 104 patients with angiographically proved PE were reviewed by two nuclear
201 teen patients with chronic stable angina and angiographically proven CAD (>70% stenosis in at least 1
208 tors, duration of follow-up, the presence of angiographically proven obstructive CAD (>/=50% stenosis
210 d men and women aged 60 years and older with angiographically proven stable ischaemic heart disease o
212 nsfection, VEGF-transfected animals had more angiographically recognizable collateral vessels (angiog
213 .43 mL x min(-1) x 100 g(-1), P<0.001), more angiographically recognizable collateral vessels (angios
216 risk factor analysis can rule in or rule out angiographically severe disease, i.e., three-vessel and/
218 udication or ischemic pain while at rest and angiographically significant atherosclerotic lesions to
219 4-year cardiovascular risk in women without angiographically significant CAD (hazard ratio 1.41, 95%
221 uited in a case-control study: 250 cases had angiographically significant CAD (stenosis > or =70%), a
222 ere stratified by the presence or absence of angiographically significant CAD at study entry, in wome
223 aphy, determination of pretest likelihood of angiographically significant CAD by the invasive angiogr
224 na typicality-based pretest probabilities of angiographically significant CAD derived from invasive c
225 wer in predicting the severity and extent of angiographically significant CAD in symptomatic patients
226 higher accuracy for detecting patients with angiographically significant CAD than the analysis of wa
227 ignificant CAD at study entry, in women with angiographically significant CAD, the metabolic syndrome
229 calcium scores in a model for prediction of angiographically significant coronary artery disease (CA
230 have higher sensitivity for the detection of angiographically significant coronary artery disease, wh
231 ise, which may be observed in the absence of angiographically significant coronary artery stenosis.
232 omatic cardiac transplant recipients without angiographically significant coronary disease, FFR and t
234 <0.0001) or the clinical model combined with angiographically significant coronary stenosis (P=0.0007
235 e hospitalized for chest pain but who had no angiographically significant coronary-artery obstruction
241 cent) of the 35 women with chest pain and no angiographically significant stenosis had decreases in t
243 e aim of this study was to determine whether angiographically silent early coronary intimal thickenin
244 ant coronary abnormalities with OCT that are angiographically silent in children with a history of co
247 ary artery disease (CAD) and 8 patients with angiographically smooth coronary arteries (normal), we i
249 ovascular resistance (HMR) immediately after angiographically successful PCI predicts MVI at cardiova
250 n develop microvascular injury (MVI) despite angiographically successful primary percutaneous coronar
254 ransient ischemic attack or stroke caused by angiographically verified 50 to 99 percent stenosis of a
255 ciate significantly with the age of onset of angiographically verified coronary artery disease (hazar
256 randomly assigned a total of 309 women with angiographically verified coronary disease to receive 0.
259 m by ultrasound was greater in patients with angiographically visible calcification (175 degrees +/-
262 nce the behavior of both the small number of angiographically visible lesions and the large number of
264 sus 0.77+/-0.06; P<0.005), reduced number of angiographically visible vessels (angiographic score=0.4
266 he area of diffuse vascular leakage measured angiographically were significantly larger with DEX impl
267 prospective cohort of 985 patients diagnosed angiographically with severe CAD (stenosis > or =70%) an
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