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1 cal angiotensin II production and the type 2 angiotensin receptor.
2 asoconstriction via the activation of type 1 angiotensin receptors.
3 (2)-adrenergic) and G(alpha)(q)-coupled (AT1 angiotensin) receptors.
4 rphisms (SNPs) in the angiotensinogen (AGT), angiotensin receptor 1 (AGTR1), and angiotensin receptor
5 n (AGT), angiotensin receptor 1 (AGTR1), and angiotensin receptor 2 (AGTR2) genes were evaluated for
6 ytometric analysis and Western blot revealed angiotensin receptor 2 (AT(2)) expression in T and NK ce
8 ing from elevated angiotensin II (AngII) and angiotensin receptor 2 (AT2R) producing increased plasma
9 1)-AA), stimulates sEng production via AT(1) angiotensin receptor activation in pregnant mice but not
11 covered that a circulating autoantibody, the angiotensin receptor agonistic autoantibody (AT(1)-AA),
12 rials, however, suggested the benefit of the angiotensin receptor and angiotensin-converting enzyme b
14 embranes, which is distinctly different from angiotensin receptors and key proteases processing angio
15 colleagues present evidence that implicates angiotensin receptors and the relocation of beta-catenin
16 tural basis of the distinct functions of the angiotensin receptors, and may guide the design of new s
18 f angiotensin-converting enzyme inhibitor or angiotensin receptor antagonist (88.3% versus 86.6%), an
20 ll three isoforms of TGF-beta), losartan (an angiotensin receptor antagonist), or a combination of th
21 Heart Failure) trial demonstrated that a new angiotensin receptor antagonist-neprilysin inhibitor was
23 angiotensin converting enzyme inhibitors or angiotensin receptor antagonists in early life can preve
24 ay lead to the development of a new class of angiotensin receptor antagonists with activities biased
25 rs, calcium channel blockers, diuretics, and angiotensin receptor antagonists), smoking status, alcoh
26 stem inhibition with dual blockade, ACEI and angiotensin receptor antagonists, on renal volume and ki
27 of angiotensin-converting enzyme inhibitors/angiotensin receptor antagonists, use of beta-blocker, a
29 n and termination of signaling of the type I angiotensin receptor (AT(1)-R) can lead to dynamic chang
32 ampsia have autoantibodies that activate the angiotensin receptor, AT1, and that autoantibody-mediate
33 r (lpr) mice lacking the major murine type 1 angiotensin receptor (AT1A); lpr mice develop a generali
34 of beta-arrestins in cross-talk between the angiotensin receptor (AT1aR) and a member of the transie
36 ange from time 0 to 180 min; the response to angiotensin receptor blockade (ARB) was defined as the c
38 odocytes, which was prevented by concomitant angiotensin receptor blockade application and TRPC6 knoc
39 hat inhibition of TGF-beta signaling through angiotensin receptor blockade can attenuate CS-induced l
41 erglycemia and the antiproteinuric effect of angiotensin receptor blockade or angiotensin-converting
42 injections (RR 0.45; 95% CI 0.24-0.83), and angiotensin receptor blockade vs placebo (RR 0.65; 95% C
43 ous angiotensin II test showed that complete angiotensin receptor blockade was achieved only in the h
45 combination therapy with NEP-inhibitors and angiotensin-receptor-blockade, which has been shown bein
46 ]: 1.04 to 1.10) and discharge ACE inhibitor/angiotensin receptor blocker (ARB) in LV dysfunction (64
47 tensin-converting enzyme (ACE) inhibitor and angiotensin receptor blocker (ARB) on atherosclerotic ev
49 ensin-converting enzyme inhibitor (ACEI) and angiotensin receptor blocker (ARB) should be used for se
50 ed in increased cancer risk, with a focus on angiotensin receptor blocker (ARB) therapy, as recent pu
51 giotensin-converting enzyme inhibitor (ACEI)/angiotensin receptor blocker (ARB) use and mortality in
52 otensin-converting enzyme (ACE) inhibitor or angiotensin receptor blocker (ARB) use, ESA use, dialysi
53 sing angiotensin-converting enzyme inhibitor/angiotensin receptor blocker (beta=0.36, P<0.001; CI: 0.
54 : high dose, 0.55; low dose, 0.72; both ACEi/angiotensin receptor blocker and beta-blocker: high dose
55 ate, angiotensin-converting enzyme inhibitor/angiotensin receptor blocker and oral anti-diabetic agen
57 n angiotensin-converting enzyme inhibitor or angiotensin receptor blocker at discharge, have little r
59 ) administration of ultrahigh dosages of the angiotensin receptor blocker candesartan on the progress
60 clinical trial, comparing the effects of the angiotensin receptor blocker candesartan with placebo in
62 ted, whereas acute HF and patients receiving angiotensin receptor blocker had higher plasma Ang II wi
63 been demonstrated that Valsartan (Val) as an angiotensin receptor blocker has renoprotective effects,
64 uggest that aliskiren was as effective as an angiotensin receptor blocker in attenuating this measure
66 n angiotensin-converting enzyme inhibitor or angiotensin receptor blocker in persons with CKD to impr
70 ng the effects of aliskiren combined with an angiotensin receptor blocker on intermediate markers of
71 etermine the effects of pretreatment with an angiotensin receptor blocker on left ventricular (LV) fu
72 giotensin receptor neprilysin inhibitor with angiotensin receptor blocker on Management Of heart fail
73 ting the renoprotective benefit of adding an angiotensin receptor blocker or a mineralocorticoid rece
74 e of angiotensin-converting enzyme inhibitor/angiotensin receptor blocker or beta-blocker and 60- to
76 from the combination of an ACE-inhibitor and angiotensin receptor blocker therapy in patients with va
77 giotensin-converting enzyme inhibitor and/or angiotensin receptor blocker therapy is the standard of
78 angiotensin-converting enzyme inhibitor and angiotensin receptor blocker therapy on patients who hav
79 d angiotensin-converting enzyme inhibitor or angiotensin receptor blocker therapy, 100% beta-blocker
80 d angiotensin-converting enzyme inhibitor or angiotensin receptor blocker therapy, 35% beta-blocker t
81 d angiotensin-converting enzyme inhibitor or angiotensin receptor blocker therapy, 51% beta-blocker t
87 r angiotensin-converting enzyme inhibitor or angiotensin receptor blocker usage during continuous flo
88 d angiotensin converting enzyme inhibitor or angiotensin receptor blocker usage were not significantl
89 y angiotensin-converting enzyme inhibitor or angiotensin receptor blocker use at discharge was associ
90 .5%, angiotensin-converting enzyme inhibitor/angiotensin receptor blocker use in 24.9+/-1.9%, and asp
91 and angiotensin-converting enzyme inhibitor/angiotensin receptor blocker use in patients with an Ame
92 s in angiotensin-converting enzyme inhibitor/angiotensin receptor blocker use or anticoagulation for
93 , angiotensin-converting enzyme inhibitor or angiotensin receptor blocker use, and nonsmoking status-
94 Angiotensin-converting enzyme inhibitor or angiotensin receptor blocker use, beta-blocker use, anti
95 and angiotensin-converting enzyme inhibitor/angiotensin receptor blocker use-is well suited to provi
96 were randomly assigned to receive either the angiotensin receptor blocker valsartan (titrated to 320
97 rketed by Novartis as Entresto) combines the angiotensin receptor blocker valsartan and the neprilysi
99 etic subjects not treated with ACE inhibitor/angiotensin receptor blocker who had the TT-ID-AA/AC gen
100 loped by scientists at Novartis, combines an angiotensin receptor blocker with a neprilysin inhibitor
101 angiotensin converting enzyme inhibitor, and angiotensin receptor blocker), and N-terminal probrain n
102 tensin-converting enzyme inhibitor (ACEi, or angiotensin receptor blocker), beta-blocker, or both dru
105 We evaluated the ability of irbesartan, an angiotensin receptor blocker, and lipoic acid, an antiox
106 n patients receiving higher doses of ACEi or angiotensin receptor blocker, beta-blocker, or both (haz
107 an angiotensin-converting enzyme inhibitor, angiotensin receptor blocker, calcium channel blocker, o
108 DMT (angiotensin-converting enzyme inhibitor/angiotensin receptor blocker, or beta-blocker) at baseli
109 ngiotensin-converting enzyme inhibitor or an angiotensin receptor blocker, should generally be includ
110 converting enzyme inhibitors or losartan, an angiotensin receptor blocker, will decrease the rate of
117 rd ratio for eplerenone versus placebo, ACEi/angiotensin receptor blocker: high dose, 0.67; low dose,
118 tly less likely after dual treatment with an angiotensin-receptor blocker (ARB) and an angiotensin-co
119 otensin-converting enzyme (ACE) inhibitor or angiotensin-receptor blocker (ARB) for patients with lef
120 erting-enzyme inhibitor (lisinopril) plus an angiotensin-receptor blocker (telmisartan) or lisinopril
121 st orally active direct renin inhibitor, the angiotensin-receptor blocker losartan, and their combina
122 in-converting enzyme inhibitor) or losartan (angiotensin-receptor blocker) in FSGS mice stimulated th
124 angiotensin-converting enzyme inhibitors or angiotensin receptor blockers (18.2% vs 16.9%, p = 1.000
125 for angiotensin-converting enzyme inhibitors/angiotensin receptor blockers (66% versus 68%; P=0.04) a
126 1.73 m(-2)), greater renal vasodilation with angiotensin receptor blockers (approximately 145 mL x mi
127 ensin-converting enzyme inhibitors (ACEI) or angiotensin receptor blockers (ARB) are generally well t
128 nsin-converting enzyme inhibitors (ACEI) and angiotensin receptor blockers (ARB) doses on outcomes in
129 ensin-converting enzyme inhibitors (ACEI) or angiotensin receptor blockers (ARB) initiated after myoc
130 otensin-converting enzyme inhibitors (ACEI), angiotensin receptor blockers (ARB), beta-blockers (BB),
132 nin angiotensin system pathways suggest that angiotensin receptor blockers (ARBs) are ideal drugs to
133 nsin-converting enzyme inhibitors (ACEIs) or angiotensin receptor blockers (ARBs) are widely prescrib
134 nsin-converting enzyme inhibitors (ACEIs) or angiotensin receptor blockers (ARBs) are widely prescrib
136 ertensive patients (n = 469), patients using angiotensin receptor blockers (ARBs) did not show a decl
137 PURPOSE OF REVIEW: As their introduction, angiotensin receptor blockers (ARBs) have been widely pr
138 ensin-converting enzyme (ACE) inhibitors and angiotensin receptor blockers (ARBs) improves left ventr
139 tensin-converting enzyme (ACE) inhibitors or angiotensin receptor blockers (ARBs) in patients with ty
140 ensin-converting enzyme (ACE) inhibitors and angiotensin receptor blockers (ARBs) in preventing the n
142 sin-converting enzyme inhibitors (ACEis) and angiotensin receptor blockers (ARBs) may increase the ri
143 in-converting enzyme inhibitors (ACE-Is) and angiotensin receptor blockers (ARBs) on the composite of
144 iotensin-converting enzyme (ACE) inhibitors, angiotensin receptor blockers (ARBs), and direct renin (
145 iotensin converting enzyme (ACE) inhibitors, angiotensin receptor blockers (ARBs), beta blockers, and
146 otensin-converting enzyme inhibitors (ACEI), angiotensin receptor blockers (ARBs), beta-blockers, and
147 target the renin angiotensin system, such as angiotensin receptor blockers (ARBs), have been associat
150 angiotensin-converting enzyme inhibitors or angiotensin receptor blockers (HR, 0.90; 95% CI, 0.79-1.
151 angiotensin-converting enzyme inhibitors or angiotensin receptor blockers (ie, renin-angiotensin sys
152 itors (captopril, fosinopril, ramipril), and angiotensin receptor blockers (losartan, candesartan).
153 for angiotensin-converting enzyme inhibitors/angiotensin receptor blockers (median, 85%; interquartil
155 angiotensin-converting enzyme inhibitors or angiotensin receptor blockers (p = 0.029), higher biliru
157 tensin-converting enzyme inhibitors [ACEIs], angiotensin receptor blockers [ARBs], and beta-blockers
158 nsin-converting enzyme inhibitors [ACEIs] or angiotensin receptor blockers [ARBs], and cilostazol) an
159 iotensin-converting enzyme inhibitors and/or angiotensin receptor blockers achieves only partial reno
160 angiotensin-converting enzyme inhibitors or angiotensin receptor blockers and 82% were receiving bet
161 expression is ameliorated by antiproteinuric angiotensin receptor blockers and angiotensin-converting
162 ith angiotensin-converting enzyme inhibitors/angiotensin receptor blockers and beta-blockers and an o
163 use angiotensin-converting enzyme inhibitors/angiotensin receptor blockers and beta-blockers compared
164 Angiotensin-converting enzyme inhibitors or angiotensin receptor blockers and beta-blockers were pre
167 angiotensin-converting enzyme inhibitors or angiotensin receptor blockers at admission), beta-blocke
168 of angiotensin-converting enzyme inhibitors/angiotensin receptor blockers at discharge decreased in
169 f angiotensin-convering enzyme inhibitors or angiotensin receptor blockers during the study and a his
170 cardiovascular examples, such as the use of angiotensin receptor blockers for chronic heart failure,
171 angiotensin-converting enzyme inhibitors or angiotensin receptor blockers for secondary prevention a
172 (3) angiotensin-converting enzyme inhibitor/angiotensin receptor blockers for systolic dysfunction,
173 he therapeutic benefits of beta-blockers and angiotensin receptor blockers given the emerging concept
174 angiotensin-converting enzyme inhibitors or angiotensin receptor blockers has become a crucial eleme
175 Specific benefits beyond those of other angiotensin receptor blockers have been claimed for tele
176 nd clinical data support the hypothesis that angiotensin receptor blockers have beneficial effects on
177 angiotensin-converting enzyme inhibitors and angiotensin receptor blockers have demonstrated benefici
178 vitro, specific actions not shared by other angiotensin receptor blockers have not yet been convinci
180 rdial infarction patients suggest a role for angiotensin receptor blockers in patients with heart fai
181 ngiotensin-converting enzyme inhibitors, and angiotensin receptor blockers in patients with heart fai
183 ngiotensin-converting enzyme inhibitors, and angiotensin receptor blockers increased by 23%, 57%, 31%
184 ensin converting enzyme (ACE) inhibitors and angiotensin receptor blockers increases the likelihood o
185 w warranted to establish the extent to which angiotensin receptor blockers may provide antiinflammato
186 and angiotensin-converting enzyme inhibitors/angiotensin receptor blockers on outcome in patients wit
187 for angiotensin-converting enzyme inhibitors/angiotensin receptor blockers therapy, 1.08 (95% confide
189 angiotensin-converting enzyme inhibitors or angiotensin receptor blockers was indicated in 18.1% of
190 angiotensin-converting enzyme inhibitors or angiotensin receptor blockers with well controlled BP an
191 ing angiotensin-converting enzyme inhibitors/angiotensin receptor blockers, and 4.5 million not recei
192 angiotensin-converting enzyme inhibitors or angiotensin receptor blockers, and aldosterone antagonis
193 aneurysmal disease, including beta-blockers, angiotensin receptor blockers, and angiotensin-convertin
194 ns, angiotensin-converting enzyme inhibitors/angiotensin receptor blockers, and beta-blockers, respec
195 th angiotensin-converting enzyme inhibitors, angiotensin receptor blockers, and calcium antagonists y
197 of angiotensin-converting enzyme inhibitors/angiotensin receptor blockers, and more sunscreen use in
198 angiotensin-converting enzyme inhibitors or angiotensin receptor blockers, and P2Y12 antagonists) re
199 angiotensin-converting enzyme inhibitors or angiotensin receptor blockers, and statins (combination
200 rs, angiotensin-converting enzyme inhibitors/angiotensin receptor blockers, and statins after acute m
201 rs, angiotensin-converting enzyme inhibitors/angiotensin receptor blockers, and statins reduces cardi
202 tency is approximately equivalent to that of angiotensin receptor blockers, angiotensin-converting en
203 angiotensin-converting enzyme inhibitors or angiotensin receptor blockers, antiplatelet drugs, beta-
204 used in the clinic AT(1)R antagonist drugs (angiotensin receptor blockers, ARBs, or sartans) at prev
205 angiotensin-converting enzyme inhibitors or angiotensin receptor blockers, beta blockers, calcium ch
206 of angiotensin-converting enzyme inhibitors/angiotensin receptor blockers, beta-blockers, aldosteron
207 angiotensin-converting enzyme inhibitors or angiotensin receptor blockers, beta-blockers, aldosteron
208 Angiotensin-converting enzyme inhibitors, angiotensin receptor blockers, beta-blockers, and aldost
209 ns, angiotensin-converting enzyme inhibitors/angiotensin receptor blockers, beta-blockers, and dual a
210 ns, angiotensin-converting enzyme inhibitors/angiotensin receptor blockers, beta-blockers, and dual a
211 sed angiotensin-converting enzyme inhibitors/angiotensin receptor blockers, beta-blockers, and statin
212 (angiotensin-converting enzyme inhibitors or angiotensin receptor blockers, beta-blockers, spironolac
214 -blockers, angiotensin-converting inhibitors/angiotensin receptor blockers, statins, diabetic treatme
215 angiotensin-converting enzyme inhibitors or angiotensin receptor blockers, which together explained
227 ing angiotensin-converting enzyme inhibitors/angiotensin receptors blockers, beta-blockers, and devic
228 (ACE) inhibitors (1.08, 1.02-1.15, p=0.008), angiotensin-receptor blockers (1.16, 1.07-1.25, p=0.0002
232 ensin-converting-enzyme (ACE) inhibitors and angiotensin-receptor blockers (ARBs) decreases proteinur
234 ensin-converting enzyme (ACE) inhibitors and angiotensin-receptor blockers (ARBs) reduce cardiovascul
235 tensin-converting enzyme (ACE) inhibitors or angiotensin-receptor blockers (ARBs) who filled brand-na
236 ensin-converting-enzyme (ACE) inhibitors and angiotensin-receptor blockers (ARBs), 3-hydroxy-3-methyl
237 allergic myocardial infarction, anaphylaxis, angiotensin-receptor blockers (ARBs), beta-adrenergic bl
239 ean BP reduction 12.9/7.7 mm Hg; p < 0.003), angiotensin-receptor blockers (mean BP reduction 13.3/7.
240 angiotensin-converting enzyme inhibitors or angiotensin-receptor blockers after SAVR for severe AS b
241 angiotensin-converting enzyme inhibitors or angiotensin-receptor blockers after surgical aortic valv
243 (angiotensin-converting enzyme inhibitors or angiotensin-receptor blockers and statins), and adverse
244 (angiotensin-converting enzyme inhibitors or angiotensin-receptor blockers and statins), and adverse
246 re angiotensin-converting enzyme inhibitors, angiotensin-receptor blockers, alpha-blockers, beta-bloc
247 n, angiotensin-converting enzyme inhibitors, angiotensin-receptor blockers, and beta-adrenergic block
248 irm the beneficial effect of ACE-inhibitors, angiotensin-receptor blockers, and diuretics and/or beta
249 of angiotensin-converting enzyme inhibitors, angiotensin-receptor blockers, beta blockers, calcium-ch
250 ng angiotensin-converting-enzyme inhibitors, angiotensin-receptor blockers, beta-blockers, and minera
251 ., angiotensin-converting enzyme inhibitors, angiotensin-receptor blockers, calcium channel blockers,
255 angiotensin-converting enzyme inhibitors or angiotensin receptor blocking agents 85.3% versus 77.4%
256 sms for sexual dimorphism of regional aortic angiotensin receptor expression and AAA formation are un
257 that in this model, expression of the AT(1b) angiotensin receptor gene in the adrenal gland is upregu
259 ngiotensin system (RAS) and the type I (AT1) angiotensin receptor in essential hypertension, we devel
261 e findings suggest that activation of type 1 angiotensin receptors in the glomerulus is sufficient to
262 ies suggest that hyperactivation of brain AT angiotensin receptors is a major pathophysiological fact
263 In murine systems, there is a second type 1 angiotensin receptor isoform, AT1B, and its expression i
264 xa11s, GATA6, TGFbeta2, chemokine ligand 12, angiotensin receptor like 1, cytochrome P450, cadherin5,
265 ponent 1, q subcomponent receptor 1 (C1qr1), angiotensin receptor-like 1 (Agtrl1), and vascular endot
266 that are being probed include, among others, angiotensin receptors, matrix metalloproteinases, integr
268 icacy and safety of LCZ696, a first-in-class angiotensin receptor neprilysin inhibitor (ARNI), in pat
269 eptide and left atrial size suggest that the angiotensin receptor neprilysin inhibitor LCZ696 may red
270 ioN fracTion (PARAMOUNT) trial, in which the angiotensin receptor neprilysin inhibitor LCZ696 reduced
271 ents with heart failure (HF) treated with an angiotensin receptor neprilysin inhibitor lived longer w
274 rtality and Morbidity in Heart Failure), the angiotensin receptor neprilysin inhibitor sacubitril/val
275 on enrolled in the Prospective comparison of angiotensin receptor neprilysin inhibitor with angiotens
278 randomized in the Prospective Comparison of Angiotensin Receptor-Neprilysin Inhibitor With an Angiot
280 GM-HF trial (Prospective Comparison of ARNI [Angiotensin Receptor-Neprilysin Inhibitor] With ACEI [An
282 PARADIGM-HF (Prospective Comparison of ARNI [Angiotensin Receptor-Neprilysin Inhibitor] with ACEI [An
283 alocorticoid receptor antagonists (MRA), and angiotensin receptor-neprilysin inhibitors (ARNI), have
284 ening in the Prospective Comparison of ARNI (angiotensin-receptor-neprilysin inhibitor) with ACEI (an
285 y, we found that activation of ERK1/2 by the angiotensin receptor occurs via both of these distinct p
286 tration of losartan, which blocks all type 1 angiotensin receptors, reduced markers of kidney disease
287 ystems diacylglycerol produced during type 1 angiotensin receptor signaling can be converted to 2-ara
290 th an animal model of PTSD and the selective angiotensin receptor type 1 (AT1) antagonist losartan, w
293 played decreased fibrosis in response to the angiotensin receptor type 1 blocker losartan showed decr
297 ing enzyme (ACE), angiotensinogen (AGT), and angiotensin receptor type I (AGTR1) have been associated
298 fluorescent protein revealed that Kv4.3 and angiotensin receptor type I are located in close proximi
299 with signaling from other receptors such as angiotensin receptors, which also couple to Gq) appears
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