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1 s and secretion similar to the action of the angiotensin type-1 receptor.
2 These data suggest that angiotensin II via angiotensin type 1 receptor activation contributes to or
4 de in drinking water; SP+TRX, n = 7) or with angiotensin type 1 receptor antagonist (AT1RA; losartan
5 Treatment in WT mice on an HF diet with the angiotensin type 1 receptor antagonist to downregulate P
7 studies have shown that neonatal delivery of angiotensin type 1 receptor antisense (AT(1)R-AS) in a r
8 irally mediated gene-transfer approach using angiotensin type 1 receptor antisense (AT1R-AS) could be
9 Gs (QM) results in 2-3-fold higher levels of angiotensin type 1 receptor (AT(1)R) densities in transi
10 the NADPH oxidase subunit, gp91phox, and the angiotensin type 1 receptor (AT-1R) and decreased eNOS i
12 of CRPtg+/0/apoE-/- mice contained increased angiotensin type 1 receptor (AT1-R) transcripts and disp
13 d to identify regions on the 1253 nucleotide angiotensin type-1 receptor (AT1) mRNA that are accessib
18 protein synthesis were mediated through the angiotensin type 1 receptor (AT1R), insofar as an AT1R a
19 9), prorenin receptor (PRR; P = 0.0004), and angiotensin type 1 receptor (AT1R, P = 0.006) and type 2
20 release of stress hormones via activation of angiotensin type 1 receptors (AT1R) within the SFO, we d
22 angiotensinogen (AGT, M235T), ACE (I/D), and angiotensin type 1 receptor (ATR1, A1166C) as predictors
24 inonucleoside, in the presence or absence of angiotensin type 1 receptor blocker (ARB), on GEN sprout
25 enTgMK mice, and treatment with losartan [an angiotensin type 1 receptor blocker (ARB)] ameliorated c
26 on is also demonstrated by the failure of an angiotensin type 1 receptor blocker, candesartan, to nor
27 the effects of topical reformulations of the angiotensin type 1 receptor blockers losartan and valsar
28 eceptors abolished the beneficial effects of angiotensin type 1 receptor blockers, suggesting a role
29 ctions of angiotensin II are mediated by the angiotensin type-1 receptor, but recent evidence strongl
32 to enter the perivascular space and activate angiotensin type 1 receptors in PVMs, leading to product
34 tudy was undertaken to compare the effect of angiotensin type 1 receptor inhibition on the progressio
35 he Edn type a receptor, Edn type b receptor, angiotensin type 1 receptor, mineralocorticoid receptor,
36 the angiotensin type-2 receptor opposes the angiotensin type-1 receptor, particularly by promoting v
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