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1 transgenic for the SARS-CoV receptor (human angiotensin-converting enzyme 2).
2 n system, angiotensin-converting enzyme, and angiotensin-converting enzyme 2.
3 e developed transgenic mice expressing human angiotensin-converting enzyme 2, a functional receptor f
4 tudies revealed a preferential expression of angiotensin-converting enzyme 2 (ACE-2), the functional
8 ns of SARS-S with the receptor for SARS-CoV, angiotensin converting enzyme 2 (ACE2); (ii) SSAA09E1 {[
11 is the only group I coronavirus known to use angiotensin-converting enzyme 2 (ACE2) as its receptor.
14 the ligand and key binding-site residues of angiotensin-converting enzyme 2 (ACE2) from its homologu
20 The membrane-associated carboxypeptidase angiotensin-converting enzyme 2 (ACE2) is an essential r
22 ent study showed that directed expression of angiotensin-converting enzyme 2 (ACE2) on cells previous
24 n S likely increase recognition of the mouse angiotensin-converting enzyme 2 (ACE2) receptor not only
25 oprotein (S400-600), which overlaps with the angiotensin-converting enzyme 2 (ACE2) receptor-binding
26 by adipocytes, can be catabolized by adipose angiotensin-converting enzyme 2 (ACE2) to form Ang(1-7).
27 ry syndrome (SARS-CoV) utilizes the receptor angiotensin-converting enzyme 2 (ACE2) to infect cells.
30 l host binding via the cell surface receptor angiotensin-converting enzyme 2 (ACE2), as well as the s
31 mploy the same receptor for host cell entry, angiotensin-converting enzyme 2 (ACE2), but it is largel
34 demic in 2002 and 2003, binds to a receptor, angiotensin-converting enzyme 2 (ACE2), through the rece
35 interface between the RBD and its receptor, angiotensin-converting enzyme 2 (ACE2), to assess their
43 ive arm of the renin angiotensin system, the angiotensin-converting enzyme 2 (ACE2)/angiotensin-(1-7)
51 on with enhanced BP, decreased expression of angiotensin converting enzyme 2, and increased expressio
52 eptapeptide generated by catalytic action of angiotensin-converting enzyme-2 angiotensin A or directl
53 ) exchanger 3, agonists of components of the angiotensin-converting enzyme 2/angiotensin(1-7)/Mas rec
54 protein with approximately 50% homology with angiotensin converting enzyme 2, but without a catalytic
55 BD, infected cells expressing human receptor angiotensin-converting enzyme 2, but with 90 to 95% less
57 part, could be attributable to a decrease in angiotensin-converting enzyme 2 expression observed in H
58 g their affinity for a viral receptor, human angiotensin-converting enzyme 2 (hACE-2), and their sens
59 at binds to its receptor glycoprotein, human angiotensin converting enzyme 2 (hACE2), and mediates vi
60 complexed with their common receptor, human angiotensin-converting enzyme 2 (hACE2), and proposed th
61 d that transgenic (Tg) mice expressing human angiotensin-converting enzyme 2 (hACE2), the receptor fo
62 ce that express the SARS-CoV receptor (human angiotensin-converting enzyme 2 [hACE2]) in airway and o
63 essing a functional SARS-CoV receptor (human angiotensin-converting enzyme 2) in a dose-dependent man
65 was lower in fetal growth restriction in an angiotensin-converting enzyme 2 knockout mouse model cha
67 ght to determine whether circulating soluble angiotensin-converting enzyme 2 (sACE2) is increased in
68 renin-angiotensin system and is a homolog of angiotensin-converting enzyme 2, sharing approximately 5
69 mAbs significantly blocked RBD-Fc binding to angiotensin-converting enzyme 2, suggesting that their e
71 omain (RBD), which mediates virus binding to angiotensin-converting enzyme 2, the functional receptor
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