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1 with primary open angle glaucoma or primary angle closure glaucoma.
2 omy should be considered in the treatment of angle closure glaucoma.
3 55 had primary angle closure and 263 primary angle-closure glaucoma.
4 ular pressure 30 mm Hg or greater or primary angle-closure glaucoma.
5 0% (n = 148) with OAG and 1.0% (n = 16) with angle-closure glaucoma.
6 hat antidepressants may increase the risk of angle-closure glaucoma.
7 il dilation is a known risk factor for acute angle-closure glaucoma.
8 re defined according to the first coding for angle-closure glaucoma.
9 ain complications being severe hyperopia and angle-closure glaucoma.
10 r 2013 on the topics open-angle glaucoma and angle-closure glaucoma.
11 rior chamber angles that predisposed them to angle-closure glaucoma.
12 subjects (0.3%, 95% CI, 0.1-0.4) had primary angle-closure glaucoma.
13 utic strategy for certain types of secondary angle-closure glaucoma.
14 eliminate any pupillary block due to primary angle-closure glaucoma.
15 wo forms of glaucoma: open-angle glaucoma or angle-closure glaucoma.
16 origin are the main risk factors for primary angle-closure glaucoma.
17 NNia substrain of mouse (DBA) with inherited angle-closure glaucoma.
18 omic variations and underlying mechanisms in angle-closure glaucoma.
19 ns/eye volume ratio, and a high incidence of angle-closure glaucoma.
20 spondents (14.6%; 95% CI, 9.9%-19.3%), acute angle-closure glaucoma; 11 of 216 respondents (5.1%; 95%
22 eyes of 154 patients consisting of 40 acute angle-closure glaucoma (AACG) eyes, 40 fellow eyes of AA
23 Recent studies underscore the importance of angle-closure glaucoma (ACG) as a cause of world blindne
24 gnosed in 3.1% (95% CI = 2.5, 3.8%), primary angle-closure glaucoma (ACG) in 0.59% (95% CI = 0.35, 0.
25 nterior chamber (AC) depth and the attack of angle-closure glaucoma (ACG) in eyes with the recent ons
30 ase represents the first report of worsening angle closure glaucoma and choroidal detachments over an
31 old Chinese female was diagnosed of primary angle closure glaucoma and had bilateral laser periphera
32 pressure (IOP) in patients with PAC, primary angle closure glaucoma, and acute angle closure crisis.
34 syndrome predisposes to both open-angle and angle-closure glaucoma, and to capsular rupture, zonular
35 The IOP reductions obtained in patients with angle closure glaucoma are often more pronounced than th
37 CG) eyes, 40 fellow eyes of AACG, 42 chronic angle-closure glaucoma (CACG) eyes, 40 primary angle-clo
38 of glaucoma reside in Asia, and with primary angle-closure glaucoma carrying a higher rate of visual
39 an origin have a higher frequency of primary angle-closure glaucoma compared with those of European o
41 nd significant visual comorbidities, such as angle closure glaucoma, cystic macular edema, and exudat
42 The DBA/2J mouse is a model for secondary angle-closure glaucoma, due to iris atrophy and pigment
43 , GAS7, FOXC1, ATXN2, TXNRD2); PACG (primary angle-closure glaucoma (EPDR1, CHAT, GLIS3, FERMT2, DPM2
45 ulcers of 1 year's duration developed acute angle-closure glaucoma following the appearance of new m
46 s and knowledge of retinal detachment, acute angle-closure glaucoma, giant cell arteritis, and centra
47 d definitions to detect and diagnose primary angle-closure glaucoma has resulted in difficulties in i
48 t surgery in patients with acute and chronic angle-closure glaucoma have been well studied and are ge
49 International investigations into primary angle-closure glaucoma have demonstrated reproducible ev
50 oma--primary open-angle glaucoma and primary angle-closure glaucoma--have different risk factors.
51 ngs into discussion a new clinical entity of angle closure glaucoma in nanophthalmos accompanied by o
52 To report clinical features of bilateral angle-closure glaucoma in a patient with nanophthalmic e
63 2 with IOP higher than 35 mm Hg, and 1 with angle-closure glaucoma not attributed to the study drug
65 primary open-angle glaucoma, chronic primary angle-closure glaucoma or pseudoexfoliation glaucoma wer
66 pseudoexfoliative glaucoma (PXG) and Primary Angle Closure Glaucoma (PACG) are scarce in the Sub-Saha
67 on the morbidity and progression to primary angle closure glaucoma (PACG) in White Caucasian individ
71 pen angle glaucoma (POAG), 27 (4.2%) primary angle closure glaucoma (PACG), and 5 (0.8%) normal tensi
72 with primary angle closure (PAC) and primary angle closure glaucoma (PACG), and to compare their diag
73 imary open angle glaucoma (POAG) and primary angle closure glaucoma (PACG)] patients and matched heal
74 s and TIMPs in the aqueous humour of primary angle-closure glaucoma (PACG) eyes were measured and com
77 laucomatous VF loss in patients with primary angle-closure glaucoma (PACG) using pointwise linear reg
78 POAG), 0.39% (95% CI, 0.34-0.45) for primary angle-closure glaucoma (PACG), and 0.15% (95% CI, 0.07-0
79 imary open-angle glaucoma (POAG) and primary angle-closure glaucoma (PACG), and projected the number
80 Glaucoma cases were classified as primary angle-closure glaucoma (PACG); 1 of the 4 forms of open-
81 rmal-tension glaucoma [NTG], 1.5%]), primary angle-closure glaucoma (PACG, 0.4%), pseudoexfoliation g
83 foliative glaucoma (PXG) and primary chronic angle closure glaucoma (PCAG) patients was 19.22 mmHg, 2
84 cus associated with nanophthalmos or with an angle-closure glaucoma phenotype, and the identification
85 our understanding of the pathophysiology of angle-closure glaucoma, pigmentary glaucoma, and a varie
86 g a higher rate of visual morbidity, primary angle-closure glaucoma poses an important public health
87 70-year-old female with a history of primary angle closure glaucoma presented with 4 mm of proptosis,
88 eful in the clinical management of eyes with angle closure glaucoma, recent studies show that the dec
89 f the first reported cases of severe chronic angle- closure glaucoma secondary to ciliary body cysts
90 ic and surgical management of severe chronic angle- closure glaucoma secondary to ciliary body cysts
91 odigital dysplasia, with progressive chronic angle- closure glaucoma secondary to ciliary body cysts
93 mice develop a progressive form of secondary angle-closure glaucoma that appears to be initiated by i
95 he highest cost category among patients with angle-closure glaucoma, whereas office visits was the hi
97 erfluoropropene (C3F8) may produce secondary angle-closure glaucoma with or without pupillary block.
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