1 y, at baseline and after pilocarpine-induced anisocoria.

2                                     However, anisocoria correlated with RAPD only in subjects with da

3                                  Contraction anisocoria describes a phenomenon in which the pupil of

4           Lastly, the localizing pitfalls of anisocoria--Horner and Raeder syndromes, physiologic ani

5 nvestigate the lateralization of contraction anisocoria in young female and male subjects.

6 ed to ascertain how a clinically significant anisocoria influences the relative afferent pupil defect

7 se results provide evidence that contraction anisocoria is more laterally asymmetric in males than in

8 uch right-side lateralization of contraction anisocoria is much greater in males than in females.

9 l fractures; unexplained neurologic deficit; anisocoria; lateral neck soft tissue injury; clinical su

10 d with narrow palpebral fissure, dysarthria, anisocoria (narrower pupil on the right side), unilatera

11                        Values of contraction anisocoria obtained by stimulating a subject's right or

12  in understanding the potential influence of anisocoria on the RAPD and also greater susceptibility o

13 des with elevated intracranial pressure (new anisocoria or intracranial pressure >20 mm Hg for >/=20

14                                      Induced anisocoria produced a significant change in RAPD from ba

15 ia--Horner and Raeder syndromes, physiologic anisocoria, pupil-involving third nerve palsy, and benig

16 ting the right eye led to larger contraction anisocoria than stimulating the left eye.

17 abetic patients with third-nerve palsies had anisocoria up to 2.5 mm.

18                                  Contraction anisocoria was calculated by subtracting relative constr