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1 appropriate direction to compensate for the anisometropia.
2 refractive error, irregular astigmatism, or anisometropia.
3 ation was not associated with astigmatism or anisometropia.
4 e corneal power can lead to great amounts of anisometropia.
5 ibromas, most commonly because of ptosis and anisometropia.
6 idual amblyopia resulting from strabismus or anisometropia.
7 ignificant association with the magnitude of anisometropia.
8 >or=0.5 D of hyperopic, myopic, or cylinder anisometropia.
9 c anisometropia and >or=2 to 3 D of cylinder anisometropia.
10 dditional monkeys developed persistent axial anisometropias.
12 r than +3 D, astigmatism 2 D or more, and/or anisometropia 2 D or more, were found in 25.6% of the ch
13 R lines) occurred, with >or=1 D of hyperopic anisometropia and >or=2 to 3 D of cylinder anisometropia
14 ed to assess variations in the prevalence of anisometropia and aniso-astigmatism by age group, with l
15 We describe the profile and associations of anisometropia and aniso-astigmatism in a population-base
17 logistic regression used to compare odds of anisometropia and aniso-astigmatism with refractive stat
18 ia, particularly during the first 1.3 years; anisometropia and astigmatism also increased with age.
19 y to show an independent association between anisometropia and both spherical ametropia and astigmati
21 ulation, there is a high prevalence of axial anisometropia and corneal/axial aniso-astigmatism, assoc
24 tropia [n = 18]); strabismus [n = 22]); both anisometropia and strabismus [n = 32]) completed the occ
25 nt hyperopia and astigmatism, in addition to anisometropia and strabismus, were the major amblyogenic
28 opic eyes of children with strabismus and/or anisometropia, and the associated poor stereoacuity prob
29 tween the 2 eyes, in addition to strabismus, anisometropia, and/or visual axis obstruction; bilateral
30 8 high-light-reared monkeys developed myopic anisometropias, and in 6 of these monkeys, the form-depr
32 es were based on spherical equivalent (SEQ), anisometropia, astigmatism, and age (corrected for gesta
38 en macular thickness and magnitude of myopic anisometropia, axial length, and visual acuity (VA) were
39 the use of stringent inclusion criteria for anisometropia, because otherwise a large proportion of t
40 nted to the dominant eye of patients without anisometropia, consistent with classical strabismic supp
42 TMV was simulated with 1.5 diopters (D) of anisometropia (dominant eye at distance, nondominant eye
43 iopter contact lenses in one eye to simulate anisometropia during the first months of life, macaques
44 mal random-dot stereopsis, and hypermetropic anisometropia each pose a significant risk for the devel
45 y similar (treated eyes: F = 0.31, P = 0.74; anisometropia: F = 0.61, P = 0.59), but significantly di
46 as more useful as a screener, especially for anisometropia for which it was 91% sensitive and 92% spe
47 ucoma in the treated eye (n = 9) had greater anisometropia (glaucoma, median -8.25 D; IQR -11.38, -5.
51 5 D), astigmatism (> 0.5 D of cylinder), and anisometropia (> 1.0 D between eyes) increased with age.
52 ical equivalent of < +3.00 D and significant anisometropia had a 7.8-fold increased risk for accommod
53 ies on the treatment of exotropia related to anisometropia have demonstrated less favorable outcomes.
56 logy of the MFS was esotropia in 58 (92.1%), anisometropia in 2 (3.2%), and exotropia in 3 patients (
57 ) made amblyopic by artificial strabismus or anisometropia in early life, as well as two visually nor
59 ical equivalent anisometropia or cylindrical anisometropia in identifying preschoolers with unilatera
63 rly monocular abnormalities of strabismus or anisometropia is proportional to the loss in cortical af
64 t hyperopic meridian; astigmatism </=1.50 D; anisometropia </=1.0 D) and emmetropic status were deter
65 ian of at least 1 eye, astigmatism </=1.5 D, anisometropia </=1.0 D) or emmetropia (hyperopia </=1.0
67 than 1 line less than with full correction; anisometropia measured less than 1.0 diopter; and an app
69 tigmatism were independently associated with anisometropia (myopes, P < 1.0E-61; hyperopes, P < 1.0E-
70 ivariable analysis: sex, age at examination, anisometropia, myopic and hyperopic refractive error (>/
71 yopia associated with strabismus (n = 32) or anisometropia (n = 20) or associated with both anisometr
72 blyopia associated with strabismus (n = 34), anisometropia (n = 23), and both anisometropia and strab
73 n (5-17 years old) with strabismus (n = 31), anisometropia (n = 29), or both conditions (n = 29).
74 10%), occlusion from ptosis (n = 9, 43%), or anisometropia (n = 9, 43%), or a combination of factors
75 s of age, mean +/- SD age 5.2 +/- 1.4 years (anisometropia [n = 18]); strabismus [n = 22]); both anis
76 D) or more, astigmatism of 1.0 D or more, or anisometropia of 0.5 D or more were present in 91% of ch
77 nisometropia of more than 0.5 D, cylindrical anisometropia of more than 0.25 D, vertical and horizont
78 al amblyopia increased significantly with SE anisometropia of more than 0.5 D, cylindrical anisometro
79 e is more accurate than spherical equivalent anisometropia or cylindrical anisometropia in identifyin
81 4), astigmatism (OR, 5.7; 95% CI, 2.5-12.7), anisometropia (OR, 27.8; 95% CI, 11.2-69.3), and strabis
82 roximately 20/63) resulting from strabismus, anisometropia, or both were enrolled into a randomized c
87 errors (myopia, hyperopia, astigmatism, and anisometropia; P<0.00001 for each) were associated indep
89 th amblyopia risk factors (eg, strabismus or anisometropia), patching improved visual acuity of the a
90 Astigmatism ranged from 15.8% to 45.2%, and anisometropia ranged from 2.8% to 8.1%, depending on age
91 dominant eye of strabismic patients without anisometropia retained suppressive input from crossed bu
95 o showed disruption of SA at lower values of anisometropia than were associated with increases in IAD
96 Monovision is therefore a form of acquired anisometropia that causes a superimposition of an in-foc
100 rs, specifically hyperopia, astigmatism, and anisometropia, varied by group, with the highest rate of
109 D, lid-suture = -1.0 to -10.25 D) of myopic anisometropia were comparable in both treatment groups.
111 ous sensory manipulations (e.g., strabismus, anisometropia), which have been reported to alter the pe
112 the previously noted increased prevalence of anisometropia with age occurs later in hyperopes than in
113 ions between the prevalence and magnitude of anisometropia with age, sex, spherical power, and cylind
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