ライフサイエンスコーパス: ankle

1 d elbow) and between-limb joints (finger and ankle).

2 ibuted among motions of the femur, knee, and ankle.

3 acrophages were significantly reduced in the ankles.

4 L-1beta, Cxcl-1, and Cxcl-2 significantly in ankles.

5 le and expression of HIF-1alpha in arthritic ankles.

6 ulders, upper back, wrists/hands, knees, and ankles.

7 ulders, upper back, wrists/hands, knees, and ankles.

8 s (2.1 +/- 0.3 %ID/g), compared with healthy ankles (1.4 +/- 0.3 %ID/g) and forepaws (1.5 +/- 0.5 %ID

9 ealed enhanced (18)F-FLT uptake in arthritic ankles (2.2 +/- 0.2 percentage injected dose per gram [%

10 The (18)F-FLT uptake in the ankles (3.5 +/- 0.3 %ID/g) reached the maximum observed

11 Foot anaesthesia reduced ankle adaptation to external force perturbations during

12 included anatomical changes of the foot and ankle, altering the moment arms and control of the muscl

13 ted hallux, an elongated tarsus, and derived ankle and calcaneocuboid joints.

14 phological counterpart of the autopod (wrist/ankle and digits) in living fishes.

15 The ankle and foot are commonly injured during sporting acti

16 acilitates climbing with a bipedally adapted ankle and foot by positioning the climber closer to the

17 of injection, no tissue disease in the foot/ankle and quadriceps, and no evidence of viral persisten

18 the foot; the score was intermediate in the ankle and upper leg and was nonsignificant in the arm, i

19 tal muscle weakness, most severely affecting ankle and wrist dorsiflexion.

20 orothymidine ((18)F-FLT) uptake in arthritic ankles and carcinomas between dynamic and static PET mea

21 mmunohistochemical staining of the arthritic ankles and forepaws revealed a strong correlation with t

22 The (18)F-FLT uptake in the ankles and forepaws was quantified on the basis of the P

23 d (18)F-FLT uptake was measured in arthritic ankles and in CT26 colon carcinomas when the mice breath

24 ast, apoptosis of Ly6C(+) macrophages in the ankles and popliteal lymph nodes, decreased migration of

25 joints as those assessed for the PsA-44 plus ankles and toe PIP joints).

26 m a weight which is attached directly to the ankle, and a one-minute dynamic knee extension protocol

27 e following examinations: hip (femur), knee, ankle, and computed tomographic (CT) angiography of the

28 12, 0.0046, 0.0014, and 0.047 for hip, knee, ankle, and CT angiography, respectively, while in the ca

29 cuits exist for motor pools controlling hip, ankle, and foot muscles, revealing a variable circuit ar

30 y mechanical hyperalgesia in the ipsilateral ankle, and secondary mechanical and heat hyperalgesia in

31 s, decreased migration of monocytes into the ankles, and a reduction of CCL2 were identified followin

32 marily involving her bilateral hands, wrist, ankles, and feet.

33 ng the left and right hands, wrists, elbows, ankles, and knees.

34 , a strong MMR signal was seen in the knees, ankles, and toes of arthritic mice.

35 tarflexion torque at 30% of maximum at three ankle angles.

36 r of the dorsalis pedis and posterior tibial ankle arteries.

37 ts imply that derived aspects of the hominin ankle associated with bipedalism remain compatible with

38 forearm, wrist, scaphoid bone, clavicle, or ankle at age 6-13 y.

39 ed greater joint swelling in the ipsilateral ankle at days 3 and 7 postinfection, and this correlated

40 e with sprains when treated with a removable ankle brace and self-regulated return to activities.

41 89 patients) with critical limb ischemia and ankle brachial index >/=1.4 who underwent lower extremit

42 persisted after excluding participants with ankle brachial index >1.4 only as well as in subgroups d

43 Participants were free of PAD, defined as an ankle brachial index <0.9 or >1.4 at baseline, and had c

44 of 470 cases of incident PAD, defined as an ankle brachial index <0.9 or >1.4, were identified.

45 e cumulative incidence of PAD, defined by an ankle brachial index <0.90 or a confirmed PAD event, wit

46 that defined peripheral artery disease as an ankle brachial index (ABI) lower than or equal to 0.90.

47 months and changes in circulating PC levels, ankle brachial index (ABI), and walking impairment quest

48 oderate arsenic exposure and incident PAD by ankle brachial index (ABI).

49 on urine cadmium, potential confounders, and ankle brachial index determinations in the follow-up exa

50 tors of poorer cognitive performance were an ankle brachial index greater than 1.30 (OR, 18.56 [95% C

51 indirect measures of arterial stiffness, an ankle brachial index greater than 1.30 and increased blo

52 if they had intermittent claudication and an ankle brachial index of <0.85, or if they had a prior pe

53 age of 74.4 (6.6) years, and had a mean (SD) ankle brachial index of 0.67 (0.18).

54 h critical limb ischemia and noncompressible ankle brachial index results, the prevalence of occlusiv

55 Approximately 20% of patients undergoing ankle brachial index testing for critical limb ischemia

56 otid intimal medial thickness, stenosis, and ankle brachial index) and risk of dementia, CHD, and tot

57 Of 125 limbs with noncompressible ankle brachial index, 72 (57.6%) anterior tibial and 80

58 s postoperatively with physical examination, ankle brachial index, duplex, and a quality-of-life ques

59 Cell therapy significantly increased ankle brachial index, increased transcutaneous oxygen te

60 patients) precisely estimated the changes in ankle brachial index, transcutaneous oxygen tension, res

61 he United States were evaluated by screening ankle brachial indices <0.9 for peripheral artery diseas

62 ears or older with a venous leg ulcer and an ankle brachial pressure index of at least 0.8, and were

63 brachial flow-mediated dilation >5% change, ankle-brachial index >0.9 and <1.3, high-sensitivity C-r

64 en urinary BPA levels (in tertiles) and PAD (ankle-brachial index < 0.9, n = 63) using logistic regre

65 kle-brachial index was used to diagnose PAD (ankle-brachial index </= 0.9).

66 Patients were enrolled based on an abnormal ankle-brachial index </=0.80 or a previous lower extremi

67 Patients age 35 to 85 years with an ankle-brachial index </=0.95 and without clinically reco

68 Incident PAD was determined by an ankle-brachial index <0.9 assessed at 2 subsequent exami

69 percentile for age, sex, and ethnicity; and ankle-brachial index <0.9.

70 ssible arteries (PCA) to those with a normal ankle-brachial index (ABI) and those with peripheral art

71 reviewed the evidence on the use of resting ankle-brachial index (ABI) as a screening test for PAD o

72 invasive test for diagnosis of LE-PAD is the ankle-brachial index (ABI) at rest and typically an ABI

73 s to determine whether use of an alternative ankle-brachial index (ABI) calculation method improves m

74 Patients were eligible if they had an ankle-brachial index (ABI) of 0.80 or less or had underg

75 dentify genetic variants associated with the ankle-brachial index (ABI), a noninvasive measure of PAD

76 esence of plaque, intima media thickness and ankle-brachial index (ABI), for N = 549.

77 the coronary artery calcium (CAC) score, the ankle-brachial index (ABI), high-sensitivity C-reactive

78 severity were established by the use of the ankle-brachial index (ABI).

79 model without B-type natriuretic peptide and ankle-brachial index (C statistic, 0.79; 95% CI, 0.75-0.

80 isk factors, B-type natriuretic peptide, and ankle-brachial index (model 6) yielded modest improvemen

81 sted with carotid intima-media thickness and ankle-brachial index (two other measures of subclinical

82 participants with PAD, independently of the ankle-brachial index and other confounders.

83 0%, 6%, 2.6 (95% CI, 1.4-4.8), and 39.2; for ankle-brachial index criteria, 0.6%, 9%, 5%, 2.3 (95% CI

84 hen compared with patients enrolled based on ankle-brachial index criteria.

85 compared with patients enrolled based on the ankle-brachial index criterion.

86 Moreover, patients with lower ankle-brachial index had (1) a more delayed reactive hyp

87 tivity C-reactive protein <2 mg/L and normal ankle-brachial index had DLRs >0.80.

88 score than carotid intima-media thickness or ankle-brachial index in subjects without and with CKD (H

89 The ankle-brachial index in the Viabahn group significantly

90 Of these patients, 47.5% underwent ankle-brachial index measurement, 38.7% duplex ultrasoun

91 n=3787) had a history of claudication and an ankle-brachial index of <0.85 or prior revascularization

92 ting vascular obstruction of 50% or greater, ankle-brachial index of less than 0.90, or physician-dia

93 ast 50%), or coronary artery disease with an ankle-brachial index of less than 0.90.

94 active protein, family history of ASCVD, and ankle-brachial index recommendations by the American Col

95 , brachial flow-mediated dilation was 0.024, ankle-brachial index was 0.036, carotid intima-media thi

96 ears, 73% were male, and the median baseline ankle-brachial index was 0.78.

97 The ankle-brachial index was used to diagnose PAD (ankle-bra

98 Creatinine, age, and ankle-brachial index were among the top predictors of at

99 ripheral revascularization, smoking, and the ankle-brachial index were predictive of ALI.

100 dysfunction, microalbuminuria, and a reduced ankle-brachial index) in 2680 Framingham Study participa

101 is, pain score, pain-free walking distance, ankle-brachial index, and transcutaneous oxygen measurem

102 condary outcomes quality of life, rest pain, ankle-brachial index, and transcutaneous oxygen pressure

103 CVD mortality, adjusting for age, sex, race, ankle-brachial index, body mass index, smoking, comorbid

104 and subclinical disease measures, including ankle-brachial index, carotid intimal-medial thickness,

105 Coronary artery calcium, ankle-brachial index, high-sensitivity CRP, and family h

106 clusion of carotid intima-media thickness or ankle-brachial index, inclusion of the coronary artery c

107 lantar flexion inversely correlated with the ankle-brachial index, indicating that patients with more

108 Improvements in ankle-brachial index, Rutherford class, and quality of l

109 ation, but noninvasive measures, such as the ankle-brachial index, show that asymptomatic PAD is seve

110 djusted for age, sex, race, body mass index, ankle-brachial index, smoking, physical activity, and co

111 l carotid artery intima-media thickness, and ankle-brachial index.

112 abetic patients on hemodialysis, we measured ankle-brachial pressure index (ABix) and evaluated miner

113 Ankle-brachial pressure index increased from 0.75 to 0.9

114 PAD was defined based on an ankle.brachial index .0.90.

115 ice exhibited a 36% loss in torque about the ankle but mdx mice exhibited a greater torque loss of 73

116 N/METHODS: Volunteer's legs were measured at ankle, calf and thigh following guidance from British nu

117 on among various subsections of the foot and ankle can be difficult, in large part due to a lack of o

118 imeter by drawing regions of interest around ankles, carcinomas, and muscle tissue.

119 measured using goniometry, measuring active ankle combined plantarflexion and dorsiflexion and combi

120 pain, lower extremity (hips, knees, and feet/ankles combined) pain, and no pain.

121 fold elevation in ROS expression in inflamed ankles compared with the ankles of healthy controls.

122 er of spirochetes in the joints and inflamed ankles compared with the infected wild-type (WT) mice, s

123 feedback, the direction of vestibular-evoked ankle compensatory responses was also reversed.

124 y presents with delayed motor milestones and ankle contractures.

125 acupuncture at 'distal' body sites, near the ankle contralesional to the more affected hand; and (iii

126 An additional ankle DF/PF exertion task was performed.

127 reas were found to be more active during the ankle DF/PF task when compared with the active balance s

128 When compared to the ankle DF/PF task, the active balance simulation task eli

129 g both the active balance simulation and the ankle DF/PF tasks, the bilateral fusiform gyrus and midd

130 that Twa hunter-gatherers use extraordinary ankle dorsiflexion (>45 degrees ) during climbing, simil

131 50 DM1 subjects, we measured the strength of ankle dorsiflexion (ADF) and then obtained a needle biop

132 , significant increases were observed in (1) ankle dorsiflexion amplitude and speed; (2) ankle planta

133 Administration of a SAS accelerated ankle dorsiflexion in both groups, but more so in the pa

134 tract was affected, as reflected by delayed ankle dorsiflexion reaction times.

135 on the CMTPedS (mean [SD], 21.5 [8.9]), with ankle dorsiflexion strength and functional hand dexterit

136 During the ankle dorsiflexion task, HSP patients had an average 19

137 was measured during a precision grip and an ankle dorsiflexion task, respectively.

138 leg MTR correlated strongly with strength of ankle dorsiflexion, measured with the Medical Research C

139 or center of pressure movements generated by ankle dorsiflexor (DF) and plantarflexor (PF) movements,

140 EMG recordings were made from right ankle dorsiflexor and right wrist extensor muscles.

141 ncreases beta and gamma oscillatory drive to ankle dorsiflexor motor neurons and that it improves toe

142 med slow triangular ramp contractions of the ankle dorsiflexors in the absence and presence of tonic

143 task (perturbing force field applied to the ankle during swing using a robotized orthosis) on 2 cons

144 ective with exoskeletons worn on one or both ankles, during a variety of walking conditions, during r

145 Ankle elasticity also allows the hip to power economical

146 Our model shows that ankle elasticity can use passive dynamics to aid push-of

147 ther walking is powered by the ankle or hip, ankle elasticity may aid walking economy by reducing col

148 We found that the key requirement for using ankle elasticity to achieve economical gait is the prope

149 human walking can be reduced by an unpowered ankle exoskeleton.

150 the animal work together; we found knee and ankle extension are the principal drivers of speed on a

151 after birth, almost all ( approximately 80%) ankle extensor motoneurons recorded in whole-cell config

152 The woman is hyperextensible and thinks her ankles feel weak.

153 ntensity of the lower back, hips, knees, and ankles/feet using the visual analog scale, categorizing

154 cerebral palsy who had been prescribed fixed ankle-foot orthoses as an example.

155 The main findings indicate that ankle-foot orthoses exert significant effects on coronal

156 early swing phase using an electrohydraulic ankle-foot orthosis.

157 We obtain simulations of an amputee using an ankle-foot prosthesis by simultaneously optimizing human

158 orous experimental test of this idea wherein ankle-foot prosthesis push-off work was incrementally va

159 r back pain was prevalent (63%), followed by ankle/foot (53%), knee (49%), and hip (31%) pain; 26% ha

160 r back (8.9%), knee (8.8%), neck (8.6%), and ankle/foot (6.8%).

161 2]), knee (OR 1.83 [95% CI 1.10, 3.02]), and ankle/foot (OR 1.82 [95% CI 1.05, 3.16]) (adjusted for s

162 istribution, namely, the shoulder, knee, and ankle/foot.

163 represented as a spatial field of isometric ankle force.

164 s of rehabilitation after immobilization for ankle fracture are unclear.

165 Patients with isolated ankle fracture presenting to fracture clinics in 7 Austr

166 Relevance: Among older adults with unstable ankle fracture, the use of close contact casting compare

167 for patients with isolated and uncomplicated ankle fracture.

168 for patients with isolated and uncomplicated ankle fracture.

169 r than 60 years with acute, overtly unstable ankle fracture.

170 Importance: Ankle fractures cause substantial morbidity in older per

171 Fourteen ankles from seven men (mean age, 32 years +/- 12 [standa

172 At 6 months, casting resulted in ankle function equivalent to that with surgery (OMAS sco

173 This enables accurate blinded estimation of ankle function purely from motor neuron information.

174 ance (MR) imaging of the brain and the right ankle had been performed 3 years previously.

175 Baseline ankle IENFD and 30-day cutaneous regeneration after thig

176 Lateral ankle injuries without radiographic evidence of a fractu

177 en with radiograph fracture-negative lateral ankle injuries.

178 We investigated this foot-ankle interplay during walking by adding stiffness to th

179 c technology to powered prosthetic knees and ankles is limited by the lack of a robust control strate

180 and negative work distal to the shank (i.e., ankle joint and all foot structures), these structures r

181 tions, physiological tremor increases as the ankle joint becomes plantarflexed.

182 istinct traumatic bone marrow lesions of the ankle joint can be diagnosed on noncalcium images recons

183 oxidative damage in the ipsilateral foot and ankle joint compared to wild-type mice which was indepen

184 Forces were applied to the ankle joint during the early swing phase using an electr

185 transmission and improve the control of the ankle joint in children with cerebral palsy.

186 Physical therapy or exercise that targets ankle joint mobility may lead to improvement in calf mus

187 ous structures on the plantar surface of the ankle joint of Confuciusornis may indicate a more crouch

188 athetic and peptidergic fiber density in the ankle joint synovium.

189 th the fascia of the inoculated foot and the ankle joint, and DCIR deficiency skewed the CHIKV-induce

190 uded open fractures, fractures involving the ankle joint, contraindication to nailing, or inability t

191 ol and intrinsic mechanical stiffness of the ankle joint, respectively.

192 g platform whose axis was collinear with the ankle joint.

193 d's adjuvant (CFA) was injected into the rat ankle joint.

194 The ankle joints are the first and most severely affected jo

195 In contrast to findings for the ankle joints, numbers of NOS2(+) and CD206(+) macrophage

196 ponses to mechanical stimulation of knee and ankle joints.

197 ritis, including erosion of the bones in the ankle joints.

198 ysis and determination of cytokine levels in ankle joints.

199 rosion were assessed at the wrist, knee, and ankle joints.

200 age and high resolution from spinal nerve to ankle level: four slabs per leg, each with 35 axial slic

201 Major amputations (at or above the ankle) limit functional independence, and their preventi

202 ed by USH2 causative genes assemble into the ankle link complex (ALC) at the hair cell stereociliary

203 y membrane complex in photoreceptors and the ankle link of the stereocilia in hair cells.

204 ct to assemble a multiprotein complex at the ankle link region of the mechanosensitive stereociliary

205 ZD7 is a second scaffolding component of the ankle-link complex.

206 mouse hair cells that PDZD7 localizes to the ankle-link region, overlapping with usherin, whirlin, an

207 ensity and whirlin localizes to both tip and ankle-link regions.

208 mplex, composed of USH2A, GPR98 and WHRN, to ankle links in developing cochlear hair cells, likely th

209 ntial role in organizing the USH2 complex at ankle links in developing cochlear hair cells.

210 The localization of PDZD7 to the ankle links of cochlear hair bundles also relies on USH2

211 s indispensable for USH2 complex assembly at ankle links, indicating the potential transport and/or a

212 tely 1 mum of the shaft, the location of the ankle links, is enriched in the lipid phosphatase PTPRQ

213 been reported to form hair cell stereocilia ankle-links, harmonin localizes to the stereocilia upper

214 ell as the nerves supplying the muscles, and ankle mobility limitations.

215 include testing for neuropathy and improving ankle mobility.

216 itioned place preference (CPP) combined with ankle monoarthritis as a condition of persistent nocicep

217 metatarsalians retained the crocodylian-like ankle morphology and hindlimb proportions of stem archos

218 eristic curve of 0.973, 0.813, and 0.758 for ankle mortise, talar dome, and talar body/head, respecti

219 r secondary outcomes: quality of life, pain, ankle motion, mobility, and patient satisfaction.

220 condary outcomes were quality of life, pain, ankle motion, mobility, complications, health resource u

221 cations is the relationship between range of ankle movement (ROAM), neuropathy, and the clinical seve

222 Range of ankle movement was measured using goniometry, measuring

223 Ankle movement were measured when the subjects walked on

224 d a spring as it is stretched and relaxed by ankle movements when the foot is on the ground, helping

225 posturing of the right leg and during paced ankle movements.

226 Of the 135 children who underwent ankle MRI, 4 (3.0%; 95% CI, 0.1%-5.9%) demonstrated MRI-

227 results suggest that vestibular influence on ankle muscle control is adjusted rapidly in sensorimotor

228 demonstrate that the vestibular influence on ankle muscles during locomotion can be adapted independe

229 myelinated afferent fibers for antagonistic ankle muscles.

230 in (mBSA), but not vehicle challenge, in the ankle of previously immunized mice produced time-depende

231 activity was recorded bilaterally around the ankles of each limb and used to compare vestibulo-muscul

232 ression in inflamed ankles compared with the ankles of healthy controls.

233 phorylated serine 558 on TACC3 bound to the "ankle" of clathrin.

234 Whether walking is powered by the ankle or hip, ankle elasticity may aid walking economy b

235 Either the ankle or the thigh may be reliable alternatives, only to

236 are inaccessible, the cuff is placed at the ankle or the thigh, but this common practice has never b

237 of agreement of -6.3/13.1 mm Hg) contrary to ankle or thigh noninvasive blood pressure (mean bias of

238 ent conditions: dorsiflexion of the dominant ankle; or flexion of the dominant wrist.

239 Sixty-one patients wore foot/ankle orthoses, 19 required walking assistance or suppor

240 Accident and Emergency Department with right ankle pain after an inversion injury and underwent plain

241 ith cutaneous feedback reduced adaptation to ankle perturbations during walking.

242 the hips shifted in the posterior direction (ankle plantar flexion).

243 luences the force-generating capacity of the ankle plantar flexors during push-off.

244 ankle dorsiflexion amplitude and speed; (2) ankle plantarflexion amplitude, speed, and duration; and

245 e with CMT2A and CMT4C exhibited the weakest ankle plantarflexion and dorsiflexion strength, as well

246 pic coefficient) for two passively mobilized ankle positions.

247 ernative ABI method using the lower of the 2 ankle pressures assigned 282 patients to the alternative

248 ay be beneficial when addressing balance and ankle proprioception exercises for the scoliotic populat

249 tery disease in these patients compared with ankle pulse volume recording.

250 Brachial-ankle pulse wave velocity (baPWV) was measured to determ

251 This suggests that powered ankle push-off reduces walking effort primarily through

252 ution, predicting that increasing prosthetic ankle push-off should decrease leading limb collision, t

253 Femoral-ankle PWV was only higher among Abeta-positive participa

254 Brachial-ankle PWV was significantly higher among Abeta-positive

255 ral (femoral-ankle PWV), and mixed (brachial-ankle PWV) vascular beds.

256 and heart-femoral PWV), peripheral (femoral-ankle PWV), and mixed (brachial-ankle PWV) vascular beds

257 ysfunction, which is associated with reduced ankle range of motion (ROM).

258 que patterns from an exoskeleton worn on one ankle reduced metabolic energy consumption by 24.2 +/- 7

259 omposite of new loss of vibratory sensation, ankle reflexes, or light touch).

260 s are smaller at the knee than at the hip or ankle, (respectively).

261 investigate sustainability of the increased ankle ROM after physical therapy has ended or if VLU reo

262 trengthens the calf muscle pump and improves ankle ROM, few studies have investigated the effect of t

263 ficant effects on coronal and sagittal plane ankle rotation; and both sagittal and horizontal plane f

264 Secondary outcomes were the Olerud-Molander Ankle Score (OMAS), quality of life, and complications (

265 per-protocol outcome was the Olerud-Molander Ankle Score at 6 months (OMAS; range, 0-100; higher scor

266 flexion/extension at right finger, elbow and ankle separately.

267 ctures of her femur and wrist; fractured her ankles several times in her late teens; and had occasion

268 that various subsections within the foot and ankle showed disparate work distribution, particularly w

269 es suggest that these injuries may represent ankle sprains rather than growth plate fractures.

270 ed the effect of sway history upon intrinsic ankle stiffness and demonstrated reductions in stiffness

271 he results show that increasing sway reduces ankle stiffness by up to 43% compared to the body-fixed

272 with a movement-dependent change in passive ankle stiffness caused by thixotropic properties of the

273 rol during times when increased sway renders ankle stiffness low.

274 hieve economical gait is the proper ratio of ankle stiffness to foot length.

275 Ankle stiffness was measured by using the same platform

276 By measuring the effect of sway history upon ankle stiffness, the present study determines whether th

277 calf muscles causing the observed changes in ankle stiffness.

278 s of human locomotion indicate that foot and ankle structures can interact in complex ways.

279 Ankle sway was increased by slowly tilting this platform

280 occurred earlier during increased levels of ankle sway.

281 ional dyspnea, chest pain, palpitations, and ankle swelling were reported by 47 (43%), 43 (39%), 10 (

282 istologic signs of arthritis but only slight ankle swelling.

283 The human foot and ankle system is equipped with structures that can produc

284 , and PCA defined as an ABI >/=1.4 and/or an ankle systolic blood pressure >255 mm Hg.

285 tra-arterial measurements at each site (arm, ankle, thigh [if Ramsay sedation scale >4]) and, in case

286 and no evidence of viral persistence in foot/ankle tissues 21 days after infection.

287 ion of CXCR2/CXCR2 ligand gene expression in ankle tissues, and significant and selective expansion o

288 ssenger RNA analysis of inflamed and healthy ankles to confirm our in vivo results.

289 n lower than assuming that the non-amputee's ankle torques are cost-free.

290 ero active work, albeit with relatively high ankle torques.

291 and MR imaging within 1 day following acute ankle trauma.

292 attributed to Purgatorius indicate a mobile ankle typical of arboreal euarchontan mammals generally

293 saminoglycan content in tendon, five cadaver ankles were examined with MR imaging and immunohistologi

294 Ankles were harvested and examined by histology, immunoh

295 th pain on Day 1 (capsaicin cream around the ankle), while the task was performed pain-free for all s

296 g hypoxia noninvasively in vivo in arthritic ankles with PET/MRI using the hypoxia tracers (18)F-fluo

297 Children underwent MRI of both ankles within 1 week of injury.

298 ple bipedal walking model to investigate how ankle work and series elasticity impact economical locom

299 Thus, the model demonstrates how elastic ankle work can reduce the total energetic demands of wal

300 ever, this does not explain why or when this ankle work, whether by muscle or tendon, needs to be per