ライフサイエンスコーパス: ankle-brachial index

1 ernal carotid intima-media thickness, or the ankle brachial index.

2 ned from cardiovascular risk factors and the ankle brachial index.

3 dized testing for the presence of PAD by the ankle-brachial index.

4 lesterol, HDL cholesterol, leg symptoms, and ankle-brachial index.

5 ences in carotid intima-medial thickness and ankle-brachial index.

6 mean postprocedural increase of 0.23 in the ankle-brachial index.

7 l carotid artery intima-media thickness, and ankle-brachial index.

8 ignificant change in resting or postexercise ankle/brachial indexes.

9 matic PAD (12 men, age 67 +/- 10 years, mean ankle brachial index 0.62 +/- 0.13) were studied.

10 le, 53% diabetic, 64% with tissue loss, mean ankle-brachial index 0.41, and mean toe pressure 26 mm H

11 68 +/- 10 years) with mild-to-moderate PAD (ankle-brachial index 0.69 +/- 0.14) had their most sympt

12 PAD was defined based on an ankle.brachial index .0.90.

13 Technicians measured ankle-brachial index 6 years apart in 2,298 participants

14 Of 125 limbs with noncompressible ankle brachial index, 72 (57.6%) anterior tibial and 80

15 PAD was defined as ankle brachial index (ABI) < or =0.90 or history of lowe

16 ticipants included 332 men and women with an ankle brachial index (ABI) <0.90 and 212 with ABI 0.90 t

17 ticipants included 392 men and women with an ankle brachial index (ABI) <0.90 and 249 with ABI 0.90 t

18 The ankle brachial index (ABI) and measures of upper and low

19 disease (PAD), specific leg symptoms and the ankle brachial index (ABI) are cross-sectionally related

20 erotic progression was assessed by computing ankle brachial index (ABI) at baseline (1,582 participan

21 s of subclinical atherosclerosis such as the ankle brachial index (ABI) could improve risk prediction

22 A low ankle brachial index (ABI) indicates atherosclerosis and

23 The ankle brachial index (ABI) is a noninvasive, reliable me

24 that defined peripheral artery disease as an ankle brachial index (ABI) lower than or equal to 0.90.

25 on of chronic kidney disease (CKD) with high ankle brachial index (ABI) measurement and to compare it

26 sought to determine the association of high ankle brachial index (ABI) measurements with left ventri

27 associations of low (<0.90) and high (>1.40) ankle brachial index (ABI) with risk of all-cause and ca

28 months and changes in circulating PC levels, ankle brachial index (ABI), and walking impairment quest

29 mance predict mortality independently of the ankle brachial index (ABI).

30 oderate arsenic exposure and incident PAD by ankle brachial index (ABI).

31 purpose of this study is to determine if an ankle-brachial index (ABI) >or=1.40 is associated with r

32 -sectional association of PAD, defined as an ankle-brachial index (ABI) <0.9, and renal insufficiency

33 The authors studied associations between ankle-brachial index (ABI) and subclinical atheroscleros

34 Ankle-brachial index (ABI) and TCO2 measurements were ob

35 ssible arteries (PCA) to those with a normal ankle-brachial index (ABI) and those with peripheral art

36 reviewed the evidence on the use of resting ankle-brachial index (ABI) as a screening test for PAD o

37 atherosclerotic progression, measured by the ankle-brachial index (ABI) at 3 consecutive time points

38 invasive test for diagnosis of LE-PAD is the ankle-brachial index (ABI) at rest and typically an ABI

39 s to determine whether use of an alternative ankle-brachial index (ABI) calculation method improves m

40 The ankle-brachial index (ABI) is widely used in the clinica

41 Patients were eligible if they had an ankle-brachial index (ABI) of 0.80 or less or had underg

42 The ankle-brachial index (ABI) provides information on both

43 ed associations of borderline and low normal ankle-brachial index (ABI) values with functional declin

44 ine the association of both a low and a high ankle-brachial index (ABI) with incident cardiovascular

45 dentify genetic variants associated with the ankle-brachial index (ABI), a noninvasive measure of PAD

46 on between PAD, defined by low values of the ankle-brachial index (ABI), and future CVD risk has been

47 Ankle-brachial index (ABI), coronary artery calcificatio

48 esence of plaque, intima media thickness and ankle-brachial index (ABI), for N = 549.

49 the coronary artery calcium (CAC) score, the ankle-brachial index (ABI), high-sensitivity C-reactive

50 systolic blood pressure ratio, also known as ankle-brachial index (ABI), in RA patients.

51 C score predicted amputation better than the ankle-brachial index (ABI).

52 tibial, and brachial arteries to obtain the ankle-brachial index (ABI).

53 aluated by history and by measurement of the ankle-brachial index (ABI).

54 severity were established by the use of the ankle-brachial index (ABI).

55 , using different methods of calculating the ankle-brachial index (ABI).

56 ary artery calcium score (CAC score, >0), or ankle-brachial index (ABI, <0.90).

57 1) young healthy subjects (YH) (n = 10; mean ankle-brachial index [ABI] 1.0 +/- 0.1, mean age 30 +/-

58 Plasma B2M correlated with ankle brachial index and functional capacity.

59 years, the 403 patients showed a significant ankle brachial index and toe brachial index deterioratio

60 isk factors, an inverse relationship between ankle-brachial index and cardiovascular events was obser

61 We assessed ankle-brachial index and hip bone mineral density, follo

62 participants with PAD, independently of the ankle-brachial index and other confounders.

63 and tertiary end points included changes in ankle-brachial index and quality-of-life assessments.

64 otid intimal medial thickness, stenosis, and ankle brachial index) and risk of dementia, CHD, and tot

65 adjusted for age, sex, race, comorbidities, ankle brachial index, and other confounders.

66 y angiography, positron emission tomography, ankle-brachial index, and B-mode ultrasound.

67 fasting blood glucose, periodontal disease, ankle-brachial index, and carotid intima-media thickness

68 ted for age, sex, race, comorbid conditions, ankle-brachial index, and other confounders.

69 ed for age, gender, race, comorbidities, the ankle-brachial index, and other potential confounders.

70 is, pain score, pain-free walking distance, ankle-brachial index, and transcutaneous oxygen measurem

71 condary outcomes quality of life, rest pain, ankle-brachial index, and transcutaneous oxygen pressure

72 were independent of CVD risk factors or the ankle-brachial index, and VEGF treatment of ECs in vitro

73 sure, reduced vascular compliance, decreased ankle-brachial indexes, and adventitial thickening.

74 The mean+/-SD ankle brachial index at baseline was 0.63+/-0.16.

75 artery disease (PAD) identified by screening ankle-brachial index benefit from preventive therapies t

76 CVD mortality, adjusting for age, sex, race, ankle-brachial index, body mass index, smoking, comorbid

77 ery calcium, carotid intima-media thickness, ankle-brachial index, brachial flow-mediated dilation, h

78 ical success was defined as improved resting ankle brachial index by > or =0.10, relief of resting pa

79 model without B-type natriuretic peptide and ankle-brachial index (C statistic, 0.79; 95% CI, 0.75-0.

80 and subclinical disease measures, including ankle-brachial index, carotid intimal-medial thickness,

81 Secondary measures, including DeltaPWT, ankle-brachial index, claudication onset time, and quali

82 Analyses were adjusted for age, sex, race, ankle brachial index, comorbidities, and other confounde

83 ace, body mass index, physical activity, the ankle brachial index, comorbidities, and other confounde

84 Adjusting for age, sex, ankle brachial index, comorbidities, and other potential

85 pported angioplasty for CLI and LLC improves ankle brachial indexes comparable to tibial bypass, heal

86 MRA index, % wall volume, and ankle-brachial index correlated with most functional mea

87 0%, 6%, 2.6 (95% CI, 1.4-4.8), and 39.2; for ankle-brachial index criteria, 0.6%, 9%, 5%, 2.3 (95% CI

88 hen compared with patients enrolled based on ankle-brachial index criteria.

89 compared with patients enrolled based on the ankle-brachial index criterion.

90 S) 1999 to 2004 who underwent measurement of ankle brachial index, CRP, and fasting glucose and insul

91 sidered major progression, which was a -0.30 ankle brachial index decrease for LV-PAD and a -0.27 toe

92 on urine cadmium, potential confounders, and ankle brachial index determinations in the follow-up exa

93 s postoperatively with physical examination, ankle brachial index, duplex, and a quality-of-life ques

94 icity, comorbid conditions, body mass index, ankle-brachial index, education, leg symptoms, cigarette

95 ses and disease severity measures, including ankle-brachial index, forced expiratory volume, and exer

96 tors of poorer cognitive performance were an ankle brachial index greater than 1.30 (OR, 18.56 [95% C

97 indirect measures of arterial stiffness, an ankle brachial index greater than 1.30 and increased blo

98 cle characteristics, greater declines in the ankle brachial index, greater declines in lower extremit

99 89 patients) with critical limb ischemia and ankle brachial index >/=1.4 who underwent lower extremit

100 persisted after excluding participants with ankle brachial index >1.4 only as well as in subgroups d

101 brachial flow-mediated dilation >5% change, ankle-brachial index >0.9 and <1.3, high-sensitivity C-r

102 Moreover, patients with lower ankle-brachial index had (1) a more delayed reactive hyp

103 tivity C-reactive protein <2 mg/L and normal ankle-brachial index had DLRs >0.80.

104 or a measure of subclinical atherosclerosis (ankle brachial index) had little impact on these associa

105 Coronary artery calcium, ankle-brachial index, high-sensitivity CRP, and family h

106 Coronary artery calcium, ankle-brachial index, high-sensitivity CRP, and family h

107 The ankle-brachial index improved significantly (0.33+/-0.05

108 Mean ankle-brachial indexes improved from 0.53 +/- 0.25 to 0.

109 score than carotid intima-media thickness or ankle-brachial index in subjects without and with CKD (H

110 The ankle-brachial index in the Viabahn group significantly

111 dysfunction, microalbuminuria, and a reduced ankle-brachial index) in 2680 Framingham Study participa

112 common carotid intimal-medial thickness, and ankle-brachial index) in 5,172 US adults without clinica

113 clusion of carotid intima-media thickness or ankle-brachial index, inclusion of the coronary artery c

114 Ankle brachial indexes increased for all groups (CLI = 0

115 Cell therapy significantly increased ankle brachial index, increased transcutaneous oxygen te

116 lantar flexion inversely correlated with the ankle-brachial index, indicating that patients with more

117 Using the single criterion of an ankle brachial index less than 0.9 to define PAD, the pr

118 D risk: high-sensitivity C-reactive protein, ankle-brachial index, leukocyte count, fasting blood glu

119 .90, and severe prevalent PAD was defined as ankle brachial index </= 0.70, with both definitions als

120 Prevalent PAD was defined as ankle brachial index </= 0.90, and severe prevalent PAD

121 The mean prevalence of PAD (defined as an ankle brachial index </=0.9) was 5.5% (SE, 0.47%).

122 eripheral arterial disease was defined as an ankle brachial index <0.9 in at least 1 leg.

123 Participants were free of PAD, defined as an ankle brachial index <0.9 or >1.4 at baseline, and had c

124 of 470 cases of incident PAD, defined as an ankle brachial index <0.9 or >1.4, were identified.

125 Participants were 384 men and women with an ankle brachial index <0.90 followed for a median of 47 m

126 e cumulative incidence of PAD, defined by an ankle brachial index <0.90 or a confirmed PAD event, wit

127 articipants were 370 men and women with PAD (ankle brachial index <0.90) and 231 without PAD.

128 pheral arterial disease (PAD) was defined by ankle brachial index <0.90, coronary artery calcificatio

129 PAD participants (n=465) had an ankle brachial index <0.90.

130 s-sectional analysis, 6,653 subjects with an ankle brachial index <1.40 were analyzed.

131 en urinary BPA levels (in tertiles) and PAD (ankle-brachial index < 0.9, n = 63) using logistic regre

132 kle-brachial index was used to diagnose PAD (ankle-brachial index </= 0.9).

133 Patients were enrolled based on an abnormal ankle-brachial index </=0.80 or a previous lower extremi

134 We defined PAD as an ankle-brachial index </=0.90.

135 Patients age 35 to 85 years with an ankle-brachial index </=0.95 and without clinically reco

136 Incident PAD was determined by an ankle-brachial index <0.9 assessed at 2 subsequent exami

137 Incident PAD was defined as a new onset of ankle-brachial index <0.9 assessed at regular examinatio

138 percentile for age, sex, and ethnicity; and ankle-brachial index <0.9.

139 PAD was defined as a baseline ankle-brachial index <0.9.

140 PAD, which is defined as an ankle-brachial index <0.90 at the baseline visit, was di

141 PAD was defined as an ankle-brachial index <0.90 in either leg.

142 When any history of CVD or an ankle-brachial index <0.90 were added to the model, SS r

143 in patients with claudication demonstrating ankle/brachial index <0.8.

144 Ankle brachial index measurement was performed at the ba

145 Of these patients, 47.5% underwent ankle-brachial index measurement, 38.7% duplex ultrasoun

146 Along with coronary artery calcium scanning, ankle-brachial index measurement, and carotid artery ult

147 isk factors, B-type natriuretic peptide, and ankle-brachial index (model 6) yielded modest improvemen

148 if they had intermittent claudication and an ankle brachial index of <0.85, or if they had a prior pe

149 Plasma was collected from PAD patients (ankle brachial index of <0.90; n=45) and subjects with r

150 age of 74.4 (6.6) years, and had a mean (SD) ankle brachial index of 0.67 (0.18).

151 Non-PAD participants (n=292) had an ankle brachial index of 0.90 to 1.30.

152 n=3787) had a history of claudication and an ankle-brachial index of <0.85 or prior revascularization

153 ting vascular obstruction of 50% or greater, ankle-brachial index of less than 0.90, or physician-dia

154 ast 50%), or coronary artery disease with an ankle-brachial index of less than 0.90.

155 ted vasodilation (assessed as the FMD%), the ankle-brachial index, or autopsy.

156 cant differences in claudication onset time, ankle-brachial index, or quality-of-life measurements be

157 active protein, family history of ASCVD, and ankle-brachial index recommendations by the American Col

158 h critical limb ischemia and noncompressible ankle brachial index results, the prevalence of occlusiv

159 Improvements in ankle-brachial index, Rutherford class, and quality of l

160 ation, but noninvasive measures, such as the ankle-brachial index, show that asymptomatic PAD is seve

161 Change in ankle-brachial index showed a similar relation (p for qu

162 , sex, race, comorbidities, body mass index, ankle brachial index, smoking, and walking exercise freq

163 djusted for age, sex, race, body mass index, ankle-brachial index, smoking, physical activity, and co

164 ent for age, sex, race, body mass index, the ankle-brachial index, smoking, physical activity, releva

165 Approximately 20% of patients undergoing ankle brachial index testing for critical limb ischemia

166 en groups in secondary end points, including ankle-brachial index, toe-brachial index, pain relief, w

167 patients) precisely estimated the changes in ankle brachial index, transcutaneous oxygen tension, res

168 sted with carotid intima-media thickness and ankle-brachial index (two other measures of subclinical

169 Ankle-brachial index values >or=1.40 have been associate

170 event was not increased, even at the lowest ankle-brachial index values, and was the same as in a pa

171 Ankle brachial index was measured, and participants repo

172 , brachial flow-mediated dilation was 0.024, ankle-brachial index was 0.036, carotid intima-media thi

173 ears, 73% were male, and the median baseline ankle-brachial index was 0.78.

174 The ankle-brachial index was used to diagnose PAD (ankle-bra

175 Creatinine, age, and ankle-brachial index were among the top predictors of at

176 ripheral revascularization, smoking, and the ankle-brachial index were predictive of ALI.