ライフサイエンスコーパス: ankylosis

1 ve defects, but without clinical evidence of ankylosis.

2 metaplasia, formation of syndesmophytes, and ankylosis.

3 induced apoptosis could play a role in tooth ankylosis.

4 tion, extra-articular fibroplasia, and joint ankylosis.

5 everity index, hindpaw swelling, and hindpaw ankylosis.

6 BMP-2 increase the development of transient ankylosis.

7 hange in the joint space width, erosions and ankylosis.

8 urfaces was often associated with a layer of ankylosis (44%, versus 8% of the ePTFE group).

9 cid conditioning promoted significantly more ankylosis (50%) compared with non-acid conditioning (6.3

10 were stained for identification of new bone, ankylosis and cementum formation.

11 cess of the procedure: complications such as ankylosis and root resorption up to the tooth exfoliatio

12 ion but also are implicated in causing tooth ankylosis and root resorption.

13 d cartilage degradation, bone erosion, joint ankylosis, and deformities in Tnfip6-null animals.

14 infraocclusion, overretained primary teeth, ankylosis, and/or slow orthodontic tooth movement, sugge

15 responsible for the phenotype of progressive ankylosis (ank) in the mouse.

16 Mutation at the mouse progressive ankylosis (ank) locus causes a generalized, progressive

17 tion progressing to a state of near complete ankylosis by day 30.

18 riable micrognathia, temporomandibular joint ankylosis, cleft palate, and a characteristic "question-

19 However, BMPe developed more ankylosis compared with both CONe and CONf while BMPf wa

20 Ankylosis compromised regeneration in sites receiving PG

21 astic prosthesis is a reliable treatment for ankylosis, especially in recurrent cases.

22 ligament, and associated root resorption and ankylosis following surgical implantation of rhBMP-2 in

23 Mutations in the progressive ankylosis gene (Ank/ANKH) cause surprisingly different s

24 nt or recessive mutations in the progressive ankylosis gene ANKH have been linked to familial chondro

25 rs can be moved by orthodontic force without ankylosis, however, at a slower rate compared with those

26 rprisingly, results indicated the absence of ankylosis; however, a marked increase in cementum format

27 he genes involved in determining the rate of ankylosis in AS are likely to be different from those in

28 stic TMJ prostheses for the treatment of TMJ ankylosis in Chinese patients with severe mandibular def

29 Temporomandibular joint (TMJ) ankylosis is a refractory disease that is difficult to p

30 BMP-induced tooth ankylosis is controversial and is poorly understood.

31 The underlying mechanism of tooth ankylosis is unclear.

32 lex with or near the targeted regions of the ankylosis mRNA through UV-induced crosslinking of the an

33 Specific regions of the 3' and 5' UTRs of ankylosis mRNA were targeted by antisense PNAs transport

34 o destruction of the articular cartilage and ankylosis of the joints.

35 Ankylosis or root resorption were not observed.

36 Fifteen patients had sacroiliac disease (ankylosis or sclerosis) on computed tomography scans.

37 No evidence of root resorption, ankylosis, or untoward inflammation was seen.

38 meniscal cells, have elevated expression of ankylosis progressive analog (ANKH) and ectonucleotide p

39 for matrix-gla-protein (MGP), fetuin-A, and ankylosis protein (Ank) as well as alkaline phosphatase

40 fic alkaline phosphatase (TNAP), progressive ankylosis protein (ANK), and ectonucleotide pyrophosphat

41 Ankylosis protein (Ank), which transports intracellular

42 ey PP(i) regulators, genes ALPL, progressive ankylosis protein (ANKH), and ectonucleotide pyrophospha

43 asia (CMD), mental retardation, deafness and ankylosis syndrome (MRDA).

44 bsequently found to have a congenital stapes ankylosis syndrome that included hyperopia, a hemicylind

45 n Tomb I displays a conspicuous case of knee ankylosis that is conclusive evidence of lameness.

46 No significant increase in tooth root ankylosis was found among the treatment groups when comp

47 One instance of root resorption and ankylosis was noted with EMD+CAF.

48 No significant new cementum formation or ankylosis was noted.

49 see if such mineral deposition and resulting ankylosis were occurring in the periodontium as well.

50 tions observed in these families with stapes ankylosis without symphalangism are predicted to disrupt