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1 te urethral tubularization (hypospadias) and anorectal abnormalities are two common and poorly unders
2 nterior meningocoele, presacral teratoma and anorectal abnormalities constitutes the Currarino triad
3 of the nRO significantly reduced spontaneous anorectal activity when compared to baseline contraction
4 quenced and compared the genomes of clinical anorectal and cervical isolates belonging to serovars E,
5 her isolated or coordinated malformations of anorectal and external genital organs provides a new too
6                  Congenital malformations of anorectal and genitourinary (collectively, anogenital) o
7 ints can account for the association between anorectal and genitourinary defects.
8 ring the anogenital phase causes coordinated anorectal and genitourinary malformations, whereas inact
9 stigating normal and abnormal development of anorectal and genitourinary structures.
10      The clinical treatment of patients with anorectal and pelvic floor dysfunction is often difficul
11 st-effective procedure for the evaluation of anorectal and pelvic floor dysfunction.
12 en shown to cause bifid nose with or without anorectal and renal anomalies (BNAR) syndrome and Manito
13 ceptible to gastric (cardia-antrum section), anorectal, and acute systemic (intravenous challenge) ca
14  important for murine resistance to gastric, anorectal, and acute systemic candidiasis.
15 mance related to types of practice (general, anorectal, and colorectal), levels and types of Board ce
16 ia were demonstrated for measurements of the anorectal angle and anorectal junction during liquid med
17 le plays an integral role in maintaining the anorectal angle.
18     (3) Surgery can correct minor congenital anorectal anomalies by identifying the external sphincte
19 typic variability in constipated patients by anorectal assessments.
20 cleft lip with cleft palate (aOR = 1.23) and anorectal atresia/stenosis (aOR = 1.40).
21 ation of the aorta, cleft lip, cleft palate, anorectal atresia/stenosis, and limb reduction (upper li
22                                              Anorectal biofeedback for children has been proposed, bu
23 (eight with esophageal cancer and eight with anorectal cancer).
24 ents in either cohort), followed by skin and anorectal cancer.
25 r the improved detection of genital tract or anorectal carriage of group B streptococci (GBS) in preg
26 l evaluation suggested no cause, or a benign anorectal cause, of bleeding.
27 sed to identify determinants associated with anorectal chlamydia and gonorrhea.
28 orted anal sex or symptoms is used to manage anorectal Chlamydia trachomatis (chlamydia) and Neisseri
29 testing is a suboptimal control strategy for anorectal chlamydia, as we found a high prevalence in wo
30 % CI, 2.8-75.0], respectively), but not with anorectal chlamydia.
31                                       Of all anorectal chlamydia/gonorrhea cases, 72% (n = 92)/33% (n
32                                      Overall anorectal chlamydia/gonorrhea positivity was 13.4% (n =
33                         Detection of vaginal-anorectal colonization with group B streptococci (GBS) i
34 f the body plan including the urogenital and anorectal complex, and the perineum region.
35 nternal anal sphincter tone is important for anorectal continence.
36  neurons which may be involved in control of anorectal contractions (mediated via the pelvic nerve),
37      Previous research has demonstrated that anorectal contractions in the rat are modulated by activ
38                                              Anorectal contractions were measured by a fluid-filled m
39 ulation sites outside the nRO did not affect anorectal contractions when compared to either (a) the 1
40 e more likely than the rostral nRO to reduce anorectal contractions.
41 t defines criteria for diagnosing functional anorectal disorders (ie, fecal incontinence, anorectal p
42 egories of functional oesophageal, bowel and anorectal disorders, and to the specific FGIDs of IBS, f
43 y, pathophysiology, and management of common anorectal disorders: fecal incontinence (FI), functional
44 cal observations suggest a wider spectrum of anorectal disturbances.
45 ten experience visceral hypersensitivity and anorectal dysfunction.
46 cography) provide a refined understanding of anorectal dysfunctions and identify phenotypes in defeca
47 clinical observations indicate a spectrum of anorectal dysfunctions.
48                            An annual digital anorectal examination may be useful for early detection
49                   Rigid sigmoidoscopy and an anorectal examination were also used to examine symptoma
50 ntinence as well as for patients who require anorectal excision for low-lying malignancy.
51                                     Although anorectal function is transiently somewhat impaired afte
52 sociated with substantial morbidity (loss of anorectal function with a permanent colostomy and a high
53 41 patients with cloacal exstrophy to assess anorectal function, urinary continence, and sexual funct
54 on) and to diagnose problems with bladder or anorectal functioning.
55  symptoms were independently associated with anorectal gonorrhea (odds ratios [ORs], 3.3 [95% confide
56 could be an appropriate control strategy for anorectal gonorrhea, as few infections would be missed.
57  for measurements of the anorectal angle and anorectal junction during liquid medium voiding.
58 al 1.00-8.11) and a distal (</=3 cm from the anorectal junction on magnetic resonance imaging) tumor
59  zones of other mouse tissues (including the anorectal junction) as well as in the gastro-oesophageal
60  on staging MRI, low rectal tumor <2 cm from anorectal junction, and laparoscopic transabdominal post
61 rineal descent during evacuation (factor 1), anorectal location at rest (factor 2), and anal resting
62 oversial aspects of caring for patients with anorectal malformation and offer insights into various m
63                                              Anorectal malformations are uncommon but complex congeni
64                       This results in severe anorectal malformations characterized by an absence of t
65 s in Bmp signaling are one possible cause of anorectal malformations during human embryogenesis.
66                                              Anorectal malformations have been recognized and managed
67 udies, abdominal wall defects in 27 (52.9%), anorectal malformations in 24 (47.1%), and Hirschsprung'
68                                Management of anorectal malformations requires an accurate clinical di
69 nital abnormalities, including malrotations, anorectal malformations, and tracheoesophageal fistula a
70 scribes recent advances in the management of anorectal malformations, including prenatal diagnosis, n
71 tion in postsurgical functional outcomes for anorectal malformations.
72                                              Anorectal manometry and imaging are useful for evaluatin
73 nd validated and high-resolution colonic and anorectal manometry as well as the barostat, despite the
74  were more frequently suggested in IBS-C and anorectal manometry in FC.
75 isorders are distinct or overlap is unclear; anorectal manometry might be used in diagnosis, but heal
76 arectum, degree of soiling/incontinence, and anorectal manometry profile(s).
77 ve assessment included physical examination, anorectal manometry, and anal endosonography.
78 tal pressures, measured with high-resolution anorectal manometry, and rectal balloon expulsion time i
79 constipated patients through high-resolution anorectal manometry.
80 cipal components analysis of high-resolution anorectal manometry.
81  optimal medical and surgical management for anorectal melanoma are needed to improve outcomes.
82                                              Anorectal melanoma is a rare malignant neoplasm with var
83 cterize changes in the surgical treatment of anorectal melanoma over time.
84 ve review was performed of all patients with anorectal melanoma treated at our institution between 19
85 45.8-74.3 years]) with histologically proven anorectal melanoma treated at our institution during a 2
86 abdominoperineal resection (APR) for primary anorectal melanoma, such an aggressive approach may be u
87 nt is not associated with outcome in primary anorectal melanoma.
88 anding and treating disorders of colonic and anorectal motility and anticipate future advances.
89 n the pathophysiology and therapy of certain anorectal motility disorders associated with the IAS dys
90 arations were used to investigate changes in anorectal motility during electrical stimulation of the
91 ation, of which 19% (n = 19)/0% (n = 0) were anorectal only.
92  testing, along with a substantial amount of anorectal-only infections.
93 sorders: fecal incontinence (FI), functional anorectal pain and functional defecation disorders.
94 ctional chest pain, dyspepsia, vomiting, and anorectal pain do not appear to vary by gender.
95                                   Functional anorectal pain syndromes are defined by clinical feature
96                                   Functional anorectal pain syndromes include proctalgia fugax (fleet
97 n levator ani syndrome (LAS) and unspecified anorectal pain the pain lasts more than 30 minutes, but
98 anorectal disorders (ie, fecal incontinence, anorectal pain, and disorders of defecation).
99 ed into levator ani syndrome and unspecified anorectal pain, which are defined by arbitrary clinical
100 gnificantly following ileostomy closure, and anorectal physiological testing was unaltered following
101                                              Anorectal physiology studies revealed significantly lowe
102 clinical histories, anal ultrasound results, anorectal physiology studies, and responses to conservat
103  investigated with endoanal ultrasonography, anorectal physiology, and examination under anaesthetic.
104 oup to divide the cloaca into urogenital and anorectal portions, exhibit complex muscle morphology in
105                                 We evaluated anorectal pressures, measured with high-resolution anore
106 py for refractory fecal incontinence and for anorectal reconstruction to avoid colostomy after abdomi
107 y produce a poor anal "seal" and an impaired anorectal sampling reflex.
108                                              Anorectal screening in women without an indication is fe
109  another testing strategy, routine universal anorectal screening with respect to chlamydia and gonorr
110                 In the recipient, the native anorectal segment was removed and the graft was transpla
111 aborate the predictive value of pretreatment anorectal sensation and the response to sensory retraini
112               The presence of some degree of anorectal sensation is the only preoperative assessment
113 ta of 263 women with at least one genital or anorectal sexually transmitted infection from a cross-se
114                           The urogenital and anorectal sinuses develop from the embryonic cloaca, and
115  Five hundred consecutive cervicovaginal and anorectal specimens submitted for GBS culture were inclu
116   A total of 203 consecutive primary vaginal/anorectal specimens were cultured in selective Todd-Hewi
117 to highlight such advances in colorectal and anorectal surgery for IBD over the last year.
118 aire, and healthcare professionals collected anorectal swabs for cytologic examination and human papi
119 ospectively inoculated with clinical vaginal/anorectal swabs, with 500-mul aliquots collected.
120 can Gastroenterological Association (AGA) on Anorectal Testing Techniques.
121 n women both with and without indication for anorectal testing, along with a substantial amount of an
122 ineum tissue between external urogenital and anorectal tracts; hypospadias - ectopic ventral position
123 s, a novel potential therapeutic strategy is anorectal transplantation (ATx).
124 ere evaluated in the John Radcliffe Hospital anorectal ultrasound unit.
125  maintains the structure and function of the anorectal unit is disrupted.

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