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1 ultimately contributed to the reduced use of anorectics.
2 enefit come under greater scrutiny than with anorectics.
4 ese results demonstrate a previously unknown anorectic action of central TTR in the control of energy
5 n of S6K1 (S6K1(-/-)) did not respond to the anorectic action of either leptin or CNTF(Ax15), implyin
6 2.5-250 microg/kg) pretreatment reversed the anorectic action of FEN (1.25 mg/kg) and FLU (5 mg/kg) b
7 lly in the NTS significantly diminished E2's anorectic action, leading to increased food intake and b
11 mice does not prevent their response to the anorectic actions of ciliary neurotrophic factor (CNTF),
12 ding behavior that temporarily abrogates the anorectic actions of leptin despite normalized circulati
14 ficient (Mc4r-/-) mice do not respond to the anorectic actions of MTII, an MSH-like agonist, suggesti
15 that is regulated by nutritional status and anorectic actions, as the endogenous ligand for the guan
17 ood intake in rats but failed to demonstrate anorectic activity in rodent natural feeding models.
26 entermine have been individually approved as anorectic agents by the Food and Drug Administration (FD
27 y, work in other laboratories indicates that anorectic agents consistently increase FLI in BNST and C
28 of these compounds are equipotent to CCK as anorectic agents in rats following intraperitoneal admin
31 ndimetrazine (PDM) is a clinically available anorectic and a candidate pharmacotherapy for cocaine ad
34 inhibition of NTS astrocytes attenuates the anorectic and body weight-suppressive effects of intra-N
37 the role of each component in mediating the anorectic and metabolic effects of leptin, and in regula
38 e that nutrient partitioning, as well as the anorectic and metabolic responses to leptin, are depende
39 opioid control of feeding by comparing rats' anorectic and orexigenic responses to naloxone and butor
41 renal failure patients with malnutrition are anorectic, and dietary counseling has had limited succes
43 enic (appetite-stimulating: MCH and NPY) and anorectic (appetite-suppressing: alpha-MSH, neurotensin,
44 tic agent mazindol and the amphetamine-based anorectic compounds diethylpropion, fenfluramine, and ph
46 ly inhibit the expression of proinflammatory anorectic cytokines such as IL-1beta, IL-6, and TNF-alph
48 to quantitate the brain concentration of the anorectic drug dexfenfluramine (DF) in human subjects re
52 II into brain parenchyma is required for the anorectic effect following intravenous (IV) administrati
54 K2 has a role in appetite regulation and its anorectic effect is mediated partly via the melanocortin
59 ia following administration of DFEN, but the anorectic effect of amylin was completely eliminated.
60 tion of 5-HT1B receptors is required for the anorectic effect of fenfluramine, we assessed food intak
61 rk1/2 signaling in the DVC negated the acute anorectic effect of insulin in healthy rats, while DVC i
62 In addition, the GLP-1r dependence of the anorectic effect of intracerebroventricular Ex4 was asse
65 Decreasing triglycerides may potentiate the anorectic effect of leptin by enhancing leptin transport
68 oth wild-type and 5-HT1B knock-out mice, the anorectic effect of the drug was absent in only the knoc
70 DIO mice, JNK inhibition sensitized leptin's anorectic effect, and enhanced leptin-induced STAT3 acti
74 ary, our results demonstrate that BNP exerts anorectic effects and reduces ghrelin concentrations in
75 isoform-selective inhibitor, exerted similar anorectic effects as SR3306, which suggests JNK2 or JNK3
76 logical blockade of OX-1R by SB334867 caused anorectic effects by reducing food intake and body weigh
79 neurobiological substrates that mediate the anorectic effects of both endogenous glucagon-like pepti
80 (1 nmol) administered ICV did not block the anorectic effects of CART (55-102) (1 h food intake, 0.2
82 microstructure and are sensitive to central anorectic effects of corticotropin-releasing factor type
84 e the dissociability of the conditioning and anorectic effects of estradiol, providing evidence again
87 , but the cellular mechanisms underlying the anorectic effects of GLP-1 require further investigation
91 , preobese Bbs4 mutant mice responded to the anorectic effects of leptin and did not display other ph
92 ning period increases the sensitivity to the anorectic effects of leptin and protects obesity-prone o
93 that neuronal GLP1Rs mediate body weight and anorectic effects of liraglutide, but are not required f
95 t leptin and its receptor play a role in the anorectic effects of nicotine on food intake and body we
96 current study was to investigate whether the anorectic effects of PYY(3-36) and oxyntomodulin can be
98 e revealed that the Y2-receptor mediates the anorectic effects of PYY3-36 whilst mechanistic studies
99 ding virtually obliterated the metabolic and anorectic effects of the central administration of oleic
100 lesions of the medial or lateral PBN on the anorectic effects of two systemically administered drug
102 demonstrate that PACAP in the CeA exerts its anorectic effects via local melanocortin and the TrKB sy
103 ; although mice became resistant to leptin's anorectic effects, the ability to increase renal sympath
108 ause central CRF(2) stimulation retains full anorectic efficacy at higher doses in the DIO model, man
109 tration of a 5-HT1BR agonist potentiates the anorectic efficacy of 5-HT2CR compounds by increasing th
110 idemic must continue, investigation into the anorectic functions of potential molecules we present he
112 e the suppression of appetite induced by the anorectic hormones amylin and cholecystokinin, as well a
120 however, the Fos response to other putative anorectics or weight reducing agents is not affected.
121 PRRSV piglets were febrile (p < 0.0001), anorectic (p < 0.0001), and weighed less at the end of t
123 cuate nucleus, and reduced expression of the anorectic peptide corticotropin-releasing hormone (CRH)
124 ed body weight in rats indicates that NT, an anorectic peptide, is involved in mediating leptin's act
125 Here we investigated whether 5-HT2CR agonist anorectic potency could be significantly enhanced by coa
127 lts lead to the suggestion that fluoxetine's anorectic properties could disrupt the female's normal e
134 et (HFD) has been related to the loss of the anorectic response to insulin injections into the centra
137 in the hypothalamus mediates the febrile and anorectic responses to disease, but the mechanism by whi
138 hort hypothalamic cilia exhibited attenuated anorectic responses to leptin, insulin, and glucose, whi
141 ctive neurons were visualized in Control and anorectic TB rats in the preoptic region, the arcuate nu
142 ostaining in some hypothalamic nuclei of the anorectic TB rats, most prominently in the supraoptic nu
147 that ate the same amount of food as did the anorectic tumor-bearing rats exhibited a 50% decrease in
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