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1 d behaviors, and muscle dysmorphia ("reverse anorexia nervosa").
2 inin (medium effect for ED, large effect for anorexia nervosa).
3 th abnormal leptin levels (e.g., obesity and anorexia nervosa).
4 -8.4) for bulimia nervosa with no history of anorexia nervosa.
5 functioning raise the risk for mortality in anorexia nervosa.
6 developmental delay, autism, psychosis, and anorexia nervosa.
7 d mortality risks, similar to those found in anorexia nervosa.
8 ave been linked to suppression of eating and anorexia nervosa.
9 ent and discharge criteria for patients with anorexia nervosa.
10 limia nervosa were unlikely to cross over to anorexia nervosa.
11 imia nervosa but were likely to relapse into anorexia nervosa.
12 cited risk of premature death in those with anorexia nervosa.
13 th eating disorders, particularly those with anorexia nervosa.
14 is little evidence for any rise in rates of anorexia nervosa.
15 n is frequently prescribed for patients with anorexia nervosa.
16 No specific recommendations were made for anorexia nervosa.
17 jor problem for research in the treatment of anorexia nervosa.
18 w randomized controlled treatment studies of anorexia nervosa.
19 amic amenorrhoea, and to clarify its role in anorexia nervosa.
20 nal nutritional status, eg, in patients with anorexia nervosa.
21 eir first discharge from hospitalization for anorexia nervosa.
22 adolescents, and 75 female adolescents with anorexia nervosa.
23 ization care and relapse prevention of adult anorexia nervosa.
24 inhibitors in the treatment of patients with anorexia nervosa.
25 rheumatic fever but who had possible PANDAS anorexia nervosa.
26 as a marker for susceptibility to a type of anorexia nervosa.
27 rders associated with streptococcus (PANDAS) anorexia nervosa.
28 arding potential treatments for bone loss in anorexia nervosa.
29 adolescent girls who met DSM-IV criteria for anorexia nervosa.
30 examined the efficacy of such therapies for anorexia nervosa.
31 tration on fat distribution in patients with anorexia nervosa.
32 nxiety disorders, personality disorders, and anorexia nervosa.
33 a diagnostic category that is distinct from anorexia nervosa.
34 degenerative diseases, obesity, bulimia, and anorexia nervosa.
35 or the treatment of eating disorders such as anorexia nervosa.
36 dynamics as a risk factor for arrhythmias in anorexia nervosa.
37 ifetime weight and duration of amenorrhea in anorexia nervosa.
38 rtium Stage 1 and the Genetic Consortium for Anorexia Nervosa.
39 iety, depression, and the psychopathology of anorexia nervosa.
40 proach in patients with, or recovering from, anorexia nervosa.
41 it was their first admission to hospital for anorexia nervosa.
42 of obesity, dieting-induced weight gain, and anorexia nervosa.
45 positive individuals among those with PANDAS anorexia nervosa (81%) than among the comparison subject
47 d circuit white matter fiber organization in anorexia nervosa after recovery could indicate a biologi
48 ison subjects, full and partial syndromes of anorexia nervosa aggregated in female relatives of both
51 anges in steroid metabolism in subjects with anorexia nervosa (AN) after weight gain have not been el
57 sensitivity to reward, yet individuals with anorexia nervosa (AN) are not motivated to eat when star
58 ntless pursuit of thinness, individuals with anorexia nervosa (AN) engage in maladaptive behaviors (r
60 al body weight is disrupted in patients with anorexia nervosa (AN) for prolonged periods of time.
76 nts were included in the dataset as follows: Anorexia Nervosa (AN) n = 171; Bulimia Nervosa (BN) n =
81 ed a genome-wide association study (GWAS) of anorexia nervosa (AN) using a stringently defined phenot
82 dolescence is a common time for the onset of anorexia nervosa (AN), a condition associated with long-
83 isms was suggested in the pathophysiology of anorexia nervosa (AN), but the role of the endogenous me
85 ve implications for eating disorders such as anorexia nervosa (AN), in which there is a high prevalen
86 distribution has previously been studied in anorexia nervosa (AN), its influence in women with AN on
88 onsidered one of the core characteristics of anorexia nervosa (AN), the exact nature of this complex
89 Previous studies have shown that women with anorexia nervosa (AN), when ill and after recovery, have
96 ratio was 4.37 (95% CI=2.4-7.3) for lifetime anorexia nervosa and 2.33 (95% CI=0.3-8.4) for bulimia n
97 Sixty-one adolescent female patients with anorexia nervosa and 45 age- and sex-matched healthy vol
100 e genetic correlations were observed between anorexia nervosa and body mass index, insulin, glucose,
101 wins were interviewed to assess diagnoses of anorexia nervosa and bulimia nervosa (per DSM-IV and bro
102 t as chronic as the well-validated disorders anorexia nervosa and bulimia nervosa and likely represen
103 unity level, 5-year recovery rates for DSM-5 anorexia nervosa and bulimia nervosa are 69 and 55%, res
108 ings support the longitudinal distinction of anorexia nervosa and bulimia nervosa but do not support
109 ell-characterized sample of individuals with anorexia nervosa and bulimia nervosa from the Price Foun
110 riatum volumes were reduced in the recovered anorexia nervosa and bulimia nervosa groups and predicte
112 in particular was much higher in people with anorexia nervosa and bulimia nervosa than in a nonclinic
113 amine diagnostic crossover longitudinally in anorexia nervosa and bulimia nervosa to inform the valid
115 agnosis EDNOS, by lowering the threshold for anorexia nervosa and bulimia nervosa, and adding BED as
116 ts suggest that eating disorders, especially anorexia nervosa and bulimia nervosa, are more common am
117 men participating in a longitudinal study of anorexia nervosa and bulimia nervosa, now in its 12th ye
118 w is reflected by the diagnostic criteria of anorexia nervosa and bulimia nervosa, which emphasize in
122 ldhood'; clarifications and modifications to anorexia nervosa and bulimia nervosa; and the inclusion
123 vosa, 282 with bulimia nervosa, and 293 with anorexia nervosa and bulimia were given the Structured C
124 conducted a genome-wide association study of anorexia nervosa and calculated genetic correlations wit
125 omen who had recovered from restricting-type anorexia nervosa and had 1 year of normal weight and reg
126 covered group (lower in women recovered from anorexia nervosa and higher in women recovered from buli
127 literature on the development and course of anorexia nervosa and interpreted critical features in li
128 le of perfectionism as a phenotypic trait in anorexia nervosa and its relevance across clinical subty
131 he most established treatment for youth with anorexia nervosa and may be efficacious for youth with b
132 nalysis of the lumbar spine in patients with anorexia nervosa and normal-weight control subjects and
134 tly are no significantly associated SNPs for anorexia nervosa and only three for educational attainme
135 uitry may contribute to restricted eating in anorexia nervosa and overeating in bulimia nervosa.
136 disorders: stabilization of the incidence of anorexia nervosa and possibly lower incidence rates of b
137 current first-line treatment for adolescent anorexia nervosa and promising for adolescent bulimia ne
138 of research on molecular genetic studies of anorexia nervosa and provide guidance for future researc
139 umes were increased on the right side in the anorexia nervosa and recovered anorexia nervosa groups a
140 results include genetic correlations between anorexia nervosa and schizophrenia, anorexia and obesity
141 e genetic correlations were observed between anorexia nervosa and schizophrenia, neuroticism, educati
142 factors significantly influence the risk for anorexia nervosa and substantially contribute to the obs
143 processes are engaged in the development of anorexia nervosa and that stimulus-response learning (th
144 on exists against schizophrenia, autism, and anorexia nervosa and that these variants may be maintain
146 number of empirical articles published about anorexia nervosa and/or bulimia nervosa and the number o
147 disorders (10 with bulimia nervosa, 16 with anorexia nervosa) and 19 healthy female comparison subje
148 80 females (0.70%) and 453 males (0.04%) had anorexia nervosa, and 3349 females (0.30%), and 61 males
149 aptured by 3 variables (any eating disorder, anorexia nervosa, and bulimia nervosa) identified by any
151 xercise-induced hypothalamic amenorrhoea and anorexia nervosa are also associated with low concentrat
155 on of the dieting behavior characteristic of anorexia nervosa as a well-entrenched habit provides a b
156 hyperactivity disorder, alcohol dependence, anorexia nervosa, autism spectrum disorder, bipolar diso
157 t restricted eating and weight loss occur in anorexia nervosa because of a failure to accurately reco
158 purging behaviors (i.e., bulimia nervosa or anorexia nervosa, binge eating/purging type);14 with ano
161 adly resembled the DSM-IV classifications of anorexia nervosa, bulimia nervosa, and binge-eating diso
162 goal of this study was to determine whether anorexia nervosa, bulimia nervosa, and eating disorder n
163 uals born in 1975-1998 and followed them for anorexia nervosa, bulimia nervosa, and eating disorder n
164 re divided into three diagnostic categories: anorexia nervosa, bulimia nervosa, and the atypical eati
165 w, and the probable four new categories are: anorexia nervosa, bulimia nervosa, binge eating disorder
166 sted of 201 participants with a diagnosis of anorexia nervosa, bulimia nervosa, binge eating disorder
167 hors found little evidence that the risk for anorexia nervosa, bulimia nervosa, or disordered eating
168 6.9; 95% confidence interval, 15.3-145.7) in anorexia nervosa but not for death (1.3; 95% confidence
169 moderately-to-severely ill adolescents with anorexia nervosa, but it is costly, and the risks of rel
171 rmulation in which the marked persistence of anorexia nervosa can be usefully understood as a well-in
172 in 12 case-control cohorts comprising 3,495 anorexia nervosa cases and 10,982 controls, the authors
174 ding nasogastric refeeding for patients with anorexia nervosa, current methods of inpatient care ofte
175 quired deficiencies, including patients with anorexia nervosa, cystic fibrosis, patients receiving lo
176 n patients with chronic treatment-refractory anorexia nervosa, DBS is well tolerated and is associate
178 Over 7 years, the majority of women with anorexia nervosa experienced diagnostic crossover: more
181 (95% CI=0.9-8.3) for patients with lifetime anorexia nervosa for 0 to 15 years (4/119 died), and 6.6
183 ignment in 32 weight-recovered subjects with anorexia nervosa from the New York site of the Fluoxetin
185 t side in the anorexia nervosa and recovered anorexia nervosa groups and on the left side in the buli
186 nce of breast cancer; nulliparous women with anorexia nervosa had a 23% (95% CI, 79% higher to 75% lo
187 ower) lower incidence, and parous women with anorexia nervosa had a 76% (95% CI, 13%-97%) lower incid
195 he authors hypothesize that individuals with anorexia nervosa have an imbalance in information proces
196 y, several large population-based studies of anorexia nervosa have been conducted in twins; it is pos
197 of the prevalence, incidence, and outcome of anorexia nervosa have been limited to cases detected thr
204 ler discusses two trials of individuals with anorexia nervosa in which deep brain stimulation of diff
219 ological impact of exercise in patients with anorexia nervosa is complex, and exercise may have a neg
226 and no black women met lifetime criteria for anorexia nervosa; more white women (N=23, 2.3%) than bla
227 (N = 246) diagnosed as having either DSM-IV anorexia nervosa (n = 136) or bulimia nervosa (n = 110)
228 omparison women (N=14), women recovered from anorexia nervosa (N=14) had significantly diminished and
229 over 8 to 25 years in 1,885 individuals with anorexia nervosa (N=177), bulimia nervosa (N=906), or ea
231 lumes in women with current restricting-type anorexia nervosa (N=19), women recovered from restrictin
233 N=19), women recovered from restricting-type anorexia nervosa (N=24), women with bulimia nervosa (N=1
234 ixteen deaths (6.5%) were recorded (lifetime anorexia nervosa, N=14; bulimia nervosa with no history
237 enetic correlation between the Eyes Test and anorexia nervosa, openness (NEO-Five Factor Inventory),
239 aging studies of adolescents and adults with anorexia nervosa or bulimia nervosa that, together, impl
240 A total of 216 women with a diagnosis of anorexia nervosa or bulimia nervosa were followed for 7
241 , 246 treatment-seeking female patients with anorexia nervosa or bulimia nervosa were interviewed eve
246 premature death among patients with lifetime anorexia nervosa peaked within the first 10 years of fol
247 trait marker, identifies a possible type of anorexia nervosa: pediatric autoimmune neuropsychiatric
248 t, particularly for adolescent patients with anorexia nervosa, point to the benefits of specialised f
249 nts were 7303 Swedish women hospitalized for anorexia nervosa prior to age 40 years between 1965 and
250 h general population, women hospitalized for anorexia nervosa prior to age 40 years had a 53% (95% co
252 anuary 2000 until May 2005, 93 patients with anorexia nervosa received intensive inpatient or day-pro
254 hanges in cerebral glucose metabolism in key anorexia nervosa-related structures at both 6 months and
255 The role of exercise in the management of anorexia nervosa remains controversial and begs future i
258 were aged 20-60 years and had a diagnosis of anorexia nervosa (restricting or binge-purging subtype)
259 nervosa, binge eating/purging type);14 with anorexia nervosa, restricting type; and 13 healthy compa
263 care in adolescent patients with non-chronic anorexia nervosa seems no less effective than IP for wei
265 I), mood, anxiety, affective regulation, and anorexia nervosa-specific behaviours at 12 months after
266 ggesting a combination of re-nourishment and anorexia nervosa-specific psychotherapy is most effectiv
268 and lower BMI at admission, and restrictive anorexia nervosa subtype predicted fatal outcome for ano
269 effect of percent body fat, body mass index, anorexia nervosa subtype, waist-to-hip ratio, and serum
270 gh rates of diagnostic "crossover" among the anorexia nervosa subtypes and bulimia nervosa may reflec
271 een the restricting and binge eating/purging anorexia nervosa subtypes over time; one-third crossed o
275 higher lifetime prevalence and incidence of anorexia nervosa than reported in previous studies, most
276 ions may represent a phenotype of adolescent anorexia nervosa that does not respond well to treatment
277 contribute information about bone health in anorexia nervosa that is independent of that provided wi
278 ings of altered striatal dopamine binding in anorexia nervosa, the authors sought to assess the respo
279 aking to eat is crucial for survival, but in anorexia nervosa, the brain persistently supports reduce
281 ltiple medical complications associated with anorexia nervosa, the primary care physician plays a cen
283 This model helps explain the resistance of anorexia nervosa to interventions that have established
291 win pairs, the prevalence of DSM-IV or broad anorexia nervosa was not significantly different than th
294 ncidence rates, and 5-year recovery rates of anorexia nervosa were calculated on the basis of data fr
296 n amenorrheic women aged 26.6 +/- 1.2 y with anorexia nervosa were identified through an outpatient s
297 ter hospitalization, 33 patients with DSM-IV anorexia nervosa were randomly assigned to 1 year of out
298 tem responsiveness is elevated in adolescent anorexia nervosa when underweight and after weight resto
299 closer to understanding the neurobiology of anorexia nervosa, which still remains a mystery and pose
300 The authors found that individuals with anorexia nervosa, who make maladaptive food choices to t
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