ライフサイエンスコーパス: antagonistic action

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1 e receptor (TR) that limits studies of their antagonistic actions.

2 Their predominantly antagonistic actions in the mammary gland form the ratio

3 The antagonistic action is interpreted by assuming that lipi

4 ylation of Dzip1, which is controlled by the antagonistic action of CK2 and B56-containing PP2As, has

5 any reduction in cAMP levels, suggesting an antagonistic action of KU14R at a more distal point in t

6 ntrolled by dynamic combinatorial as well as antagonistic action of retinoic acid (RA), Bmp and Fgf s

7 duction of ventral cell types occurs via the antagonistic action of Shh on PKA activity.

8 A similar mechanism could explain the antagonistic action of some peptides on the activation o

9 h modifiers target the same site on PCNA, an antagonistic action of SUMO on ubiquitin-dependent DNA d

10 The results demonstrated an antagonistic action of the antioxidants.

11 nth and reproductive organs are specified by antagonistic action of two floral homeotic genes, APETAL

12 ective point mutants of LT-IIb, mimicked the antagonistic action of wild-type LT-IIb.

13 eractions support that a balance between the antagonistic actions of BR and auxin is required for opt

14 Here, we show that oppositely patterned and antagonistic actions of BR and auxin maintain the stem c

15 Gene expression depends upon the antagonistic actions of chromatin remodeling complexes.

16 These antagonistic actions of endogenous BDNF and NT-3 provide

17 indicated that flowering is regulated by the antagonistic actions of phyB and cry2.

18 administration, but possible synergistic or antagonistic actions of supraspinal alpha2-GABAARs on sp

19 ering appears to be the direct target of the antagonistic actions of the photoreceptors.

20 nt changes via perturbations in the mutually antagonistic actions of the phys and PIFs.

21 bimodality is principally underscored by the antagonistic actions of these ligands at downstream mela

22 o anterior and posterior regions through the antagonistic actions of transcription factors Gli3 and H

23 es into sclerotome and dermomyotome involves antagonistic actions of ventralizing and dorsalizing sig

24 ects of caffeine are mediated largely by its antagonistic action on A2AR in the brain.

25 The low expression of RKIP and its antagonistic action on B-Raf suggests that RKIP may play

26 abiotic stresses by means of synergistic or antagonistic actions referred to as signaling crosstalk.

27 xisome proliferator-activated receptor gamma antagonistic actions requiring the ligand binding domain

28 eine (5.0 mM) and ryanodine (0.1 mM) exerted antagonistic actions upon qgamma charge movements.

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