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1 induced via endovascular perforation of the anterior cerebral artery.
2 ebral arteries: middle cerebral artery, 60%; anterior cerebral artery, 20%; posterior cerebral artery
3 nd were middle cerebral artery, 64% and 78%; anterior cerebral artery, 45% and 84%; and internal caro
4 usion, but did not change in the ipsilateral anterior cerebral artery (ACA) territory or the contrala
6 the right middle cerebral artery, the right anterior cerebral artery and the right posterior cerebra
7 terior communicating arteries, (2) bilateral anterior cerebral arteries, and (3) ipsilateral proximal
8 discuss variants of internal carotid artery, anterior cerebral artery, anterior communicating artery,
9 ry apoplexy causing compression of bilateral anterior cerebral artery branches and leading to bilater
10 artery, distal internal carotid artery, and anterior cerebral artery did not vary substantially betw
12 apoplexy can cause compression of bilateral anterior cerebral arteries from the expanding mass and l
13 exceptionally rare form of stroke-bilateral anterior cerebral artery infarction, without rupture or
14 CA] with M1 middle cerebral artery and/or A1 anterior cerebral artery involvement) or tandem (extracr
16 demonstrate that blood flow velocity in the anterior cerebral artery is hyporeactive in Huntington's
17 raphy showed that blood flow velocity in the anterior cerebral artery is significantly less in patien
18 was found to have watershed infarcts in the anterior cerebral artery-middle cerebral artery and midd
20 al carotid artery, anterior communicating or anterior cerebral artery, or middle cerebral artery were
21 arteries, distal internal carotid arteries, anterior cerebral arteries, posterior cerebral arteries,
22 The number of collaterals between middle and anterior cerebral arteries, the anastomoses within middl
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