ライフサイエンスコーパス: anterior horn

1 y alpha-motor neuron loss in the spinal cord anterior horn.

2 bodies and processes of interneurons in the anterior horn.

3 microdissection and one from the surrounding anterior horns.

4 few GFAP-cells in Layer II in the SVZ of the anterior horn and the body of the lateral ventricle appe

5 onal loss in the lower cranial nerve nuclei, anterior horns and corresponding nerves, atrophy of the

6 A) is a motor neuron disorder resulting from anterior horn cell death.

7 non-immune forms of PNH that include toxins, anterior horn cell degeneration in motor neurone disease

8 tract but persists in the face of selective anterior horn cell destruction.

9 re syndrome 1 and lethal arthrogryposis with anterior horn cell disease are autosomal recessive fetal

10 enerative conditions such as Alzheimer's and anterior horn cell disease.

11 Spinal cord had axonal spheroids, anterior horn cell loss and axonal degeneration in anter

12 oma-spinal cord-34 motor neurons and primary anterior horn cell neurons showed that IL-6 exerted a ne

13 is virus (TMEV) cleared virus infection from anterior horn cell neurons.

14 ot accompanied by significant axonal loss or anterior horn cell pathology.

15 eased myofiber size and number and increased anterior horn cell size.

16 The motor unit includes the anterior horn cell, the motor axon and the muscle fibers

17 he distal motor nerve up to the level of the anterior horn cell.

18 of the central grey matter, with predominant anterior horn-cell involvement, and nine (75%) children

19 A) is a motor-neuron disorder resulting from anterior-horn-cell death.

20 ed atrophic spinal cords with marked loss of anterior horn cells and degeneration of corticospinal tr

21 d disorders characterized by degeneration of anterior horn cells and progressive muscle weakness.

22 ized by specific degeneration of spinal cord anterior horn cells and subsequent muscle atrophy.

23 atrophy, there is selective degeneration of anterior horn cells but a normal corticospinal tract.

24 Virus antigen was localized predominantly to anterior horn cells in infected IFN-gamma(-/-) H-2(q) mi

25 Virus antigen was localized predominantly to anterior horn cells in infected IL-6-/- H-2q mice.

26 There was a loss of anterior horn cells in the spinal cord.

27 ly less affected neuronal subpopulation, the anterior horn cells of the spinal cord.

28 and paralysis as a result of degeneration of anterior horn cells of the spinal cord.

29 aracterized by selective degeneration of the anterior horn cells with subsequent weakness and atrophy

30 ron" pattern: a symmetric involvement of the anterior horn cells.

31 iquitin-positive intraneuronal aggregates in anterior horn cells.

32 7% in Purkinje cells to 80.6 +/- 2.8% in the anterior horn cells.

33 corticospinal tract, without involvement of anterior horn cells.

34 erebral and brainstem neurons to spinal cord anterior horn cells; thus, severe poliomyelitis, but not

35 In the anterior horn enriched mRNA pool, we could not clearly i

36 eus, and paraventricular nucleus, and in the anterior horn, interomediolateral cell column, and Clark

37 both transgenic mouse and human spinal cord anterior horn motor neurons, indicating that members of

38 oplasmic Lewy body-like inclusions in spinal anterior horn motor neurons.

39 erior columns of the spinal cord and loss of anterior horn neurons but without other involvement of t

40 oreactive inclusions were observed in spinal anterior horn neurons in all SALS and FALS cases, except

41 ic LDVs possess the unique ability to infect anterior horn neurons of ADPM-susceptible mice, they exh

42 gic analysis demonstrated dramatic injury to anterior horn neurons of IL-6-/- H-2q mice at 12 d after

43 c isolates in their unique ability to infect anterior horn neurons of immunosuppressed C58 and AKR mi

44 hat mHTT was preferably expressed within the anterior horn of the gray matter, in both cervical and l

45 nts), (g) cartilage thinning adjacent to the anterior horn of the lateral meniscus (in 19, 19, and 21

46 Arthroscopic examination of the anterior horn of the lateral meniscus in all 22 patients

47 d speckled increased signal intensity at the anterior horn of the lateral meniscus near its central a

48 Increased signal intensity at the anterior horn of the lateral meniscus near its central a

49 Increased signal intensity at the anterior horn of the lateral meniscus was seen on the im

50 Some motor neurons in the anterior horn of the spinal cord also were immunopositiv

51 and, to a lesser extent, glial cells in the anterior horn of the spinal cord exhibit robust Cox-2 im

52 gments are found in apoptotic neurons in the anterior horn of the spinal cord of affected transgenic

53 ting/degenerated motor neurons in the lumbar anterior horn of the spinal cord, suggesting a direct ca

54 In the anterior horn of the spinal cord, virtually all motor ne

55 pathy with prominent vacuolar changes in the anterior horn of the spinal cord.

56 generation of the alpha-motor neurons in the anterior horn of the spinal cord.

57 proteins selectively in motor neurons of the anterior horn of the spinal cord.

58 Neurons in the anterior horn of the spinal cords of paralyzed mice exhi

59 l, facial, and acoustic cranial ganglia; the anterior horns of the spinal cord in the region of the d

60 eatures of inflammation, particularly in the anterior horns of the spinal cord, the dorsal pons, and

61 of abnormal signal intensity confined to the anterior horns on a lumbar spine magnetic resonance imag

62 r horn was greater than that adjacent to the anterior horn (P <.05), and enhancement of the lateral m

63 sy; (2) laterally directed biopsies from the anterior horn should be included in extended biopsy prot

64 nce of abundant migratory neuroblasts in the anterior horn SVZ forming structures here denominated ce

65 ere performed of the ventricular index (VI), anterior horn width (AHW), and thalamo-occipital distanc