1  our understanding of the pathophysiology of anti-GBM disease.

2 autoantibodies from patients with autoimmune anti-GBM disease.

3  humoral response, only a select few develop anti-GBM disease.

4  chemoattractant produced by renal cortex in anti-GBM disease.

5 munopathogenic processes in the two forms of anti-GBM disease.

6 microbial Ag may be sufficient to induce the anti-GBM disease.

7 lead to different clinical manifestations of anti-GBM disease.

8 ransplant anti-glomerular basement membrane (anti-GBM) disease.

9 l study involving 30 patients diagnosed with anti-GBM disease and 30 healthy controls matched for the

10  superoxide dismutase as novel biomarkers of anti-GBM disease and lupus nephritis, with stronger corr

11 nusual combination of c-ANCA antibodies with anti-GBM disease, and this association raises complex qu

12 ated in the urine and kidneys of mice during anti-GBM disease, as well as in mice with spontaneously

13 iology of anti-glomerular basement membrane (anti-GBM) disease before clinical presentation is unknow

14                                   We induced anti-GBM disease in DBA/1, C57BL/6, AKR, and NOD mice wi

15 Th2 responses associates with the outcome of anti-GBM disease in mice.

16 is due to anti-glomerular basement membrane (anti-GBM) disease is unknown.

17 ary hemorrhage, another important feature of anti-GBM disease; this was not correlated with the sever

18                                              Anti-GBM disease was induced by injection of guinea pig

19         High antibody titers at diagnosis of anti-GBM disease were associated with ultimate loss of r