1 ds, cholera toxin B subunit and a monoclonal anti-GM1 antibody.

2 oth motor axonal AIP and selective serum IgG anti-GM1 antibodies.

3        Our results show that patient derived anti-GM1 antibodies and cholera toxin beta subunit impai

4                                              Anti-GM1 antibodies are present in some patients with au

5                          It is not clear how anti-GM1 antibodies cause nerve dysfunction and injury;

6   We asked whether passive transfer of human anti-GM1 antibodies from patients with GBS modulate axon

7               In contrast, low levels of IgG anti-GM1 antibodies (> 1:100) were detected in both the

8  the pathogenicity of immunoglobulin M (IgM) anti-GM1 antibodies in serum from patients with multifoc

9 e creation of rabbit models of anti-GD1b and anti-GM1 antibody-mediated sensory and motor neuropathy,

10                                        Human anti-GM1 antibodies were compared with other GM1 ligands

11                                              Anti-GM1 antibodies were found in 54% of C. jejuni-serop

12                                 In contrast, anti-GM1 antibodies were rare in sera that were either s

13 thies associated with elevated titers of IgM anti-GM1 antibodies, with increasing concentrations of i