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4 ession of HB-EGF in rat kidneys treated with anti- glomerular basement membrane (anti-GBM) antibody (
5 partment developed glomerulonephritis in the anti-glomerular basement disease model, whereas nontrans
8 roblast recruitment in renal fibrosis due to anti-glomerular basement membrane (anti-GBM) disease is
12 l chemoattractant, during the progression of anti-glomerular basement membrane (GBM) antibody (Ab) GN
14 f inflammation and in the progression of the anti-glomerular basement membrane (GBM) antibody-induced
20 ere, macrophage-mediated, cytokine-dependent anti-glomerular basement membrane (GBM) glomerulonephrit
21 inflammation, the development of accelerated anti-glomerular basement membrane (GBM) glomerulonephrit
22 autologous phase of an accelerated model of anti-glomerular basement membrane (GBM) glomerulonephrit
23 in vivo, we induced a model of Fc-dependent anti-glomerular basement membrane (GBM) nephritis in wil
25 s to monitor renal disease progression in an anti-glomerular basement membrane (GBM) nephritis mouse
26 e subjected to an augmented passive model of anti-glomerular basement membrane (GBM)-induced experime
27 f glomerulonephritis induced by heterologous anti-glomerular basement membrane Abs in wild-type (IL-4
28 initiated by binding of heterologous (sheep) anti-glomerular basement membrane Abs to the glomeruli o
29 ls of experimental glomerulonephritis, being anti-glomerular basement membrane and lipopolysaccharide
30 distinct, separate disease process, such as anti-glomerular basement membrane antibodies or anti-neu
31 s characterized by circulating and deposited anti-glomerular basement membrane antibodies, focal necr
32 ies has been known to occur with exposure to anti-glomerular basement membrane antibody (AGBM-Ab) in
35 lowing parameters both at baseline and after anti-glomerular basement membrane antibody treatment: bl
36 triggered GN with a subnephritogenic dose of anti-glomerular basement membrane antibody, Aire(-/-) mi
37 issolution of NETs via DNase I did not alter anti-glomerular basement membrane antibody-induced glome
38 ly modulates iNOS expression and activity in anti-glomerular basement membrane antibody-mediated glom
42 s induced with a previously described rabbit anti-glomerular basement membrane antiserum nephritis ap
43 mediated by pathogenic antibodies, including anti-glomerular basement membrane disease and lupus neph
44 d a macrophage- and T-cell-mediated model of anti-glomerular basement membrane disease in STC1 transg
45 nt nephritis (APTN) is an aggressive form of anti-glomerular basement membrane disease that targets t
46 US/TTP), 8; systemic lupus erythematosus, 3; Anti-glomerular basement membrane disease, 2; oxalosis,
47 ytoplasmic antibodies-associated vasculitis, anti-glomerular basement membrane disease, and systemic
48 severe glomerulonephritis after induction of anti-glomerular basement membrane disease, with more inf
50 n of glomerulonephritis after induction with anti-glomerular basement membrane globulin, with enhance
51 ological characteristics during experimental anti-glomerular basement membrane glomerulonephritis (an
52 is, we compared the intensity of accelerated anti-glomerular basement membrane glomerulonephritis bet
53 ed: a diffuse type that is characteristic of anti-glomerular basement membrane glomerulonephritis, an
57 ive glomerulonephritis, type I and II (MPG), anti-glomerular basement membrane nephritis (anti-GBM),
58 and dedifferentiated podocytes of mice with anti-glomerular basement membrane nephritis and in human
61 necessary for local chemokine expression in anti-glomerular basement membrane nephritis, although th
62 /c control mice, were challenged with rabbit anti-glomerular basement membrane nephrotoxic sera (NTS)
63 kidney inflammation in a macrophage-mediated anti-glomerular basement membrane reactive serum-induced
66 erular neutrophil accumulation and damage in anti-glomerular basement membrane-induced (anti-GBM-indu
68 ntaneous chronic glomerulonephritis (GN) and anti-glomerular basement membrane-induced nephritis.
69 In rats, intrarenal arterial injection of anti-glomerular endothelial cell (GEN) antibodies leads
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