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1 e production in RAW264.7 cells (a measure of anti-inflammation).
2 possible regulatory pathways of MC1-R beyond anti-inflammation.
3  applied to antimutation, antityrosinase and anti-inflammation.
4 compounds i.e. anti-cancer, anti-allergy and anti-inflammation.
5  for its pharmacological functions including anti-inflammation.
6 anced cellular DNA repair, antioxidation and anti-inflammation.
7 f miR-33 that mediates simvastatin-triggered anti-inflammation.
8 nnexin A1 (AnxA1), an effector of endogenous anti-inflammation.
9 ects in vasodilation, anti-angiogenesis, and anti-inflammation.
10 Progesterone treatment showed no significant anti-inflammation action on cytokine concentrations.
11 ced neurodegeneration involving antioxidant, anti-inflammation and anti-apoptotic mechanisms.
12                                          The anti-inflammation and anti-infection functions for Vitam
13          The involvement of Vitamin D/VDR in anti-inflammation and anti-infection represents a newly
14 ovel cyclooxygenase inhibitors, may serve as anti-inflammation and anticancer agents, and may contrib
15                        Berberine has several anti-inflammation and anticancer biologic effects.
16 nt of green tea extract, exhibits effects of anti-inflammation and antioxidation on periodontal infla
17 sts that WLT can be applied to antimutation, anti-inflammation and antityrosinase.
18 atins have shown pleiotropic effects such as anti-inflammation and bone stimulation.
19 ontributing to the development of a state of anti-inflammation and dichotomy of immunologic polarizat
20 n important role in endogenous mechanisms of anti-inflammation and pro-resolution.
21 sal antimicrobial responses demonstrate that anti-inflammation and proresolution are different respon
22    There is an important distinction between anti-inflammation and resolution; anti-inflammation is p
23 d protective processes: Th2 cytokine-induced anti-inflammation and ROS reduction.
24 ced KLF2 and eNOS expression as well as flow anti-inflammation, and suggest that HDAC5 could be a pot
25  signaling, cell migration, tissue survival, anti-inflammation, and T-cell-mediated cellular immunity
26 tiatherogenic effects of HDL (antioxidation, anti-inflammation, antithrombotic effects, endothelial s
27 acid-stimulated neutrophils, consistent with anti-inflammation but not COX inhibition (IC50s = 1-8 mM
28 pha transcription and suggest a mechanism of anti-inflammation by the antioxidants through control of
29 efficacy in lung function, QOL, and possibly anti-inflammation compared with BUD plus Tulo treatment.
30 ic functions for this effector of endogenous anti-inflammation could be unveiled by studying collagen
31 haracterize the balance of systemic pro- and anti-inflammation early after burn and inhalation injury
32 2-nHP66 scaffold exerts potent antibacterial/anti-inflammation effects in vivo and promotes bone form
33 )-induced metabolic diseases due to enhanced anti-inflammation engineered by lowering receptor intera
34 ranscriptional regulator for antioxidant and anti-inflammation enzymes that binds to its endogenous i
35 ta has been shown to play a critical role in anti-inflammation; however, the signaling mechanisms of
36                                              Anti-inflammation immunotherapy has been successfully ap
37 10 was a critical mediator for PGRN-mediated anti-inflammation in collagen-induced arthritis by using
38 s model to explore the importance of dynamic anti-inflammation in promoting resolution of infection a
39 on between anti-inflammation and resolution; anti-inflammation is pharmacologic intervention in infla
40 o be a key pathway by which immunomodulation/anti-inflammation may operate.
41 tosterol showed a strong Th2-inclination and anti-inflammation potential in vitro.
42 sting that these terpenoid compounds have an anti-inflammation potential through the inhibition of T-
43 in the 1980s demonstrated that pharmacologic anti-inflammation prevented and slowed the progression o
44 3 eicosapentaenoic acid that displays potent anti-inflammation/pro-resolution actions in vivo.
45 ein A-I mimetic peptide with antioxidant and anti-inflammation properties on a high-fat or normal cho
46 -beta1 is a pleiotropic cytokine with potent anti-inflammation property.
47 n (EPRAP), whose function in PGE(2)-mediated anti-inflammation remains undefined.
48 tion and bone loss, suggesting that enhanced anti-inflammation status is an active process.
49 ng in extra-hepatic tissues by statins as an anti-inflammation strategy.
50 ng Gbetagamma-regulation may be an effective anti-inflammation strategy.
51    To investigate the molecular mechanism of anti-inflammation, we analyzed the regulation of tumor n

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