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1 n, cyclophosphamide, fludarabine, and rabbit anti-thymocyte globulin.
2 ed, consisting of fludarabine, busulfan, and anti-thymocyte globulin.
3 4 +/- 0.9 versus 0.2 +/- 0.4, p = 0.011) and anti-thymocyte globulin 0.8 +/- 0.4 versus 0, p = 0.018)
4 dose per day on days -8 to -3), serotherapy (anti-thymocyte globulin [10 mg/kg, one dose per day on d
5 ulatory drug regimen includes induction with anti-thymocyte globulin and alphaCD20 antibody, followed
6 of a combination of immunomodulatory agents, anti-thymocyte globulin and pegylated granulocyte CSF, n
7 t posttransplant total lymphoid irradiation, anti-thymocyte globulin, and an intravenous donor blood
9 ated whether the combination of fludarabine, anti-thymocyte globulin, and total body irradiation (TBI
10 nsiveness of some patients with trisomy 8 to anti-thymocyte globulin (ATG) and cyclosporine (CsA) wou
11 hypothesized that a combination of low-dose anti-thymocyte globulin (ATG) and pegylated granulocyte
14 ransplantation); (ii) induction therapy with anti-thymocyte globulin (ATG) instead of anti-interleuki
16 disease (GVHD), source of stem cells, use of anti-thymocyte globulin (ATG) or cyclophosphamide in the
22 usly (IV) on days -5 and -4; 15 mg/kg equine anti-thymocyte globulin (ATGAM) IV on days -1, +1, and +
23 e in vitro generation of regulatory cells by anti-thymocyte globulins could provide ad-ditional thera
27 sphamide was added to immunosuppression with anti-thymocyte globulin induction, cyclosporine, mycophe
29 emia received intraportal islet grafts under anti-thymocyte globulin-mycophenolate mofetil-tacrolimus
30 an in 1 patient), fludarabine, thiotepa, and anti-thymocyte globulin or alemtuzumab conditioning were
37 nd 100 mg/kg with TBI 2 Gy, fludarabine, and anti-thymocyte globulin results in effective conditionin
38 Standard immunosuppression included rabbit anti-thymocyte globulin-rituximab induction with tacroli
40 n immunotherapy regimen consisting of rabbit anti-thymocyte globulin (Thymoglobulin) and the monoclon
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