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2 n vivo TNF inhibition assays, was tested for antiarthritic activity in the AA rat model of arthritis.
3 of aminopterin analogues were evaluated for antiarthritic activity in the mouse type II collagen mod
4 llagen antibody production suggests that the antiarthritic activity of the effective compounds result
6 ther exploited for the selective delivery of antiarthritic agents into the inflamed joints to enhance
7 d arthritis and has the capacity to generate antiarthritic CD8+ Treg cells and expand the relative nu
9 nthesized, characterized, and along with the antiarthritic drug, auranofin, tested as inhibitors of t
10 ble model for identifying remission-inducing antiarthritic drugs and has predictive value with respec
17 efore, we undertook studies to determine the antiarthritic efficacy and mechanism of action of a well
22 angiogenesis and suggest that an additional antiarthritic mechanism afforded by IL-1 inhibitors, but
26 eptides activate specific receptors to exert antiarthritic properties, avoiding excessing inflammatio
27 logy to deliver genes whose products possess antiarthritic properties; the current emphasis is on tra
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