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1                   However, mechanisms of its antiarthritic activities are still poorly understood.
2 n vivo TNF inhibition assays, was tested for antiarthritic activity in the AA rat model of arthritis.
3  of aminopterin analogues were evaluated for antiarthritic activity in the mouse type II collagen mod
4 llagen antibody production suggests that the antiarthritic activity of the effective compounds result
5                        IL-10 is an effective antiarthritic agent and has a major effect on the presen
6 ther exploited for the selective delivery of antiarthritic agents into the inflamed joints to enhance
7 d arthritis and has the capacity to generate antiarthritic CD8+ Treg cells and expand the relative nu
8 e SSRIs may provide a template for potential antiarthritic drug development.
9 nthesized, characterized, and along with the antiarthritic drug, auranofin, tested as inhibitors of t
10 ble model for identifying remission-inducing antiarthritic drugs and has predictive value with respec
11                     Many of the conventional antiarthritic drugs are effective in suppressing inflamm
12  scaffold for development of next-generation antiarthritic drugs.
13                            Interestingly, an antiarthritic effect was also observed in opposing contr
14 rticular transgene expression with promising antiarthritic effects in animal models.
15 so synergized with the steroid to elicit its antiarthritic effects.
16 xidant, antimutagenic, anti-inflammatory and antiarthritic effects.
17 efore, we undertook studies to determine the antiarthritic efficacy and mechanism of action of a well
18       Scientific evidence is lacking for the antiarthritic efficacy of turmeric dietary supplements t
19                          Here, we assess the antiarthritic efficiency of glutaminyl cyclase (QC) inhi
20             In this study we queried whether antiarthritic functions for this effector of endogenous
21 e represent a novel class of small molecular antiarthritic inhibitors.
22  angiogenesis and suggest that an additional antiarthritic mechanism afforded by IL-1 inhibitors, but
23 d individually and in combination, for their antiarthritic potency in the mouse CIA model.
24      The aim of this study was to assess the antiarthritic potential of 2 SSRIs, fluoxetine and cital
25                     These data highlight the antiarthritic potential of the SSRI drug family and prov
26 eptides activate specific receptors to exert antiarthritic properties, avoiding excessing inflammatio
27 logy to deliver genes whose products possess antiarthritic properties; the current emphasis is on tra

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