ライフサイエンスコーパス: antibody reactivity

1 de novo synthesis of TIN-ag protein, and its antibody reactivity.

2 osus (SLE) patients (n = 11) with raised aCL antibody reactivity.

3 essing of this allergen greatly affected its antibody reactivity.

4 nd meaningful readouts of influenza-specific antibody reactivity.

5 and half of them developed CD4 binding site antibody reactivity.

6 crambling their grafted sequences eliminates antibody reactivity.

7 ial fluid (P<0.0001) and more often had ECGF antibody reactivity.

8 ion of both gp120 and CD4 residues to enable antibody reactivity.

9 ain, resulting in substantial variability in antibody reactivity.

10 also selectively enhanced human anti-Neu5Gc antibody reactivity.

11 ation responsible for the lack of monoclonal antibody reactivity.

12 tion with H185 antibody resulted in no OC125 antibody reactivity.

13 te a specificity matrix useful in predicting antibody reactivity.

14 HCV infection was assessed by antibody reactivity.

15 en hybrids of the p53 core domain and tested antibody reactivity.

16 r binding site as assessed by 17b monoclonal antibody reactivity.

17 rise to frequent modulation in the levels of antibody reactivity.

18 th different sizes and conformation-specific antibody reactivities.

19 that the arrays allow detection of specific antibody reactivity across a broad dynamic range using c

20 Most individuals showed unexpected antibody reactivities against peptides unique to autosom

21 aScreen-based immunoreactivity measurements, antibody reactivity against 3 proteins was positively as

22 We validated increased antibody reactivity against AKT3, FCGR3A and ARL8B in pa

23 une individuals from Kenya was skewed toward antibody reactivity against asexual blood stage antigens

24 his study is to evaluate the human preformed antibody reactivity against DKO renal microvascular endo

25 ining measurements of expression profiles of antibody reactivity against each protein (295 antigens a

26 We identified strong antibody reactivity against PA-X in most H7N7-exposed in

27 Importantly, we identified strong antibody reactivity against PA-X, a putative virulence f

28 their sera were examined by immunoassay for antibody reactivity against synthetic peptides represent

29 Moreover, antibody reactivity against these antigens was temporall

30 Approximately half of the patients showed antibody reactivity against up to 7 out of the 33 tumor

31 Northern analysis, N-terminal sequence, and antibody reactivity analyses indicated the absence of mR

32 sequence, which may relate to their specific antibody reactivities and host cell interactions.

33 ls, protect against reactive oxygen species, antibody reactivity and half-life in serum were influenc

34 nt dilution analysis, we analysed the BK.T-1 antibody reactivity and identified the bound cellular pr

35 y (but not sufficient) to induce a change of antibody reactivity and in some cases is not even necess

36 he E. coli host causes loss of O:23 and O:36 antibody reactivity and restores reactivity with WGA.

37 To determine the role of antibody reactivity and the impact of somatic hypermutat

38 Anti-alphav and -laminin antibody reactivity and their respective incorporated sp

39 On the basis of subunit molecular weights, antibody reactivity, and DNA binding activities, DRP-3 w

40 her by mass, N-terminal amino acid sequence, antibody reactivity, and enzymatic activity.

41 rse-phase chromatography, spectrophotometry, antibody reactivity, and kinetics of deglycosylation, de

42 te treatment of recombinant gp36 reduced the antibody reactivity, and nonglycosylated synthetic pepti

43 mothers exhibited infrequent, low-level SV40 antibody reactivity, and only six case mothers seroconve

44 odocytes), anti-heparan-sulfate-proteoglycan antibody reactivity, and wheat germ agglutinin lectin st

45 henotypic characteristics (coaggregation and antibody reactivity) as well as in their genotypic chara

46 gens identified were further scrutinized for antibody reactivity at primary, secondary, and latent di

47 Patients' sera were also tested for antibody reactivities by immunoblotting with M. paratube

48 ected mutagenesis of this sequence decreased antibody reactivity by 30%.

49 12 of the 14 patients; most rapidly lost all antibody reactivity by NIH technique in an average time

50 o correlation was noted between neutralizing antibody, reactivity by Western blot, and subsequent pro

51 icated that the pathogenicity and monoclonal antibody reactivity differences between two molecularly

52 tional context by which to quantify specific antibody reactivities even in complex sera.

53 By plotting antibody reactivity (fluorescence intensity) for known p

54 or recognition, transduction, and anticapsid antibody reactivity for AAV2.

55 The HIV-1 and HIAP antibody reactivity found in patients with primary bilia

56 racellular antigens that elicited high-titer antibody reactivity greater in post-DLI than in pre-DLI

57 ZFYVE19, DAPK3, and OGFOD1 elicited minimal antibody reactivity in 12 normal subjects and 12 chemoth

58 Additionally, patterns of antibody reactivity in both GCF and serum in the subject

59 luenza virus and observed low but detectable antibody reactivity in elderly subjects, suggesting that

60 roarray analysis to characterize patterns of antibody reactivity in MS serum against a panel of CNS p

61 l killer cell infiltration and a presence of antibody reactivity in the absence of complement deposit

62 polymerase chain reaction and assessed HHV-6 antibody reactivity in the cerebrospinal fluid of enceph

63 Concomitantly, fibrillin-2 mRNA, antibody reactivity in the explants, and fibrillin-2-spe

64 Furthermore, high anti-HIP1 antibody reactivity in the sera of a cohort of MCC patie

65 However, the antibody reactivity increased substantially when a mixtu

66 ent of ectodomain conformation by monoclonal antibody reactivity indicate that this suppression of fu

67 ion of allergic disease independent of their antibody reactivity, is still lacking.

68 with early or later Lyme borreliosis to the antibody reactivities of sera from controls.

69 oximately 80% of the genome, we compared the antibody reactivities of sera from patients with early o

70 .4.2009 New Orleans strains, we compared the antibody reactivity of a panel of mouse monoclonal antib

71 The molecular size and antibody reactivity of gp45 expressed by the JG-29 clone

72 human immune response to syphilis, the serum antibody reactivity of syphilitic patients was examined

73 The sizes and antibody reactivity of the intermediates suggest that HM

74 ivalent influenza vaccine and determined the antibody reactivity of these sera to influenza array ant

75 zes of the proteins estimated by the loss of antibody reactivity on Western blots were essentially id

76 All four cats developed similar antibody reactivity patterns to B. koehlerae OMP antigen

77 ope immunogenicity and the interpretation of antibody reactivity patterns with HLA panels.

78 nd specificities of the Pmp subtype-specific antibody reactivity relating to gender and clinical outc

79 Antibody reactivity significantly decreased during UDCA

80 e criteria of the variant CstF-64, including antibody reactivity, size, germ cell expression, and a c

81 quences with the IUPred-L scale, followed by antibody reactivity testing of 16-30-aa peptides from pe

82 In one large clonal lineage of gp41-reactive antibodies, reactivity to HIV-1 Env was acquired only af

83 and 75 normal blood donors were screened for antibody reactivity to 77 serologically defined tumor an

84 iruses and the development of heterosubtypic antibody reactivity to animal influenza viruses.

85 ice was used to establish an animal model of antibody reactivity to citrullinated proteins.

86 rredoxin:NADP+ oxidoreductase (FNR), as were antibody reactivity to FNR and diaphorase activity.

87 Despite a decrease in antibody reactivity to HIV Gag and Pol proteins, patient

88 Cellular and antibody reactivity to hsp65 was assessed.

89 tations over 1000 resamplings, we identified antibody reactivity to influenza whole-protein and pepti

90 All IVIg preparations varied in level of antibody reactivity to intact HLA antigens.

91 While heating caused a reduction in antibody reactivity to multimeric forms of parvalbumins

92 dividuals suspected of having HGE reaffirmed antibody reactivity to multiple antigens of B. burgdorfe

93 remic chimpanzees had a higher prevalence of antibody reactivity to NS3, NS4, and NS5.

94 There was frequent antibody reactivity to protein 41-G (p41-G), outer surfa

95 IgG4 antibody reactivity to purified Ll-SXP-1 was assessed by

96 ties of antiviral IgG changed over time, and antibody reactivity to some viral proteins was detected

97 Antibody reactivity to the HCV envelope proteins E1 or E

98 Antibody reactivity to the orf 65 protein (ELISA) and to

99 phaScreen procedure was developed to measure antibody reactivity to the recombinant products.

100 rtunity to evaluate the relationship between antibody reactivity to these antigens and infection outc

101 ccessive samples was just a part of changing antibody reactivity to these peptides that again became

102 Serum antibody reactivity to this peptide epitope increased in

103 djustment for individuals' ages in years and antibody reactivity to whole-schizont extract (Chonyi, r

104 Antibody reactivity was detectable before microfilaremia

105 Antibody reactivity was detected in 79% of the patients'

106 79% of the patients' sera, and the level of antibody reactivity was directly correlated with disease

107 Antibody reactivity was first established upon expressio

108 Antibody reactivity was most prominent against oligomeri

109 CF-7 tumor cells in 16 patients, whereas IgG antibody reactivity was observed in a few patients.

110 Finally, anti-cytomegaloviral IgG antibody reactivity was significantly inhibited in the D

111 The pattern of antibody reactivity we observed may, in part, result fro

112 the gene(s) potentially responsible for the antibody reactivities were carried out, and an animal de

113 The Ehx activities and antibody reactivities were compared with those of Hly.

114 c2A, two levels (strong and undetectable) of antibody reactivity were detected, suggesting that weak

115 Two bands of the same mobility and antibody reactivity were found in Western blots of plasm

116 nitored by immunoblot, increases in specific antibody reactivity were more prevalent among volunteers

117 Patterns of antibody reactivity were stable in most of the patients

118 teady-state levels of subunits by monoclonal antibody reactivity, when used in combination with a dis

119 exposed on the capsid surface for polyclonal antibody reactivity, while the small HA epitope was inac

120 HHV-8 infection was assessed by measuring antibody reactivity with a K8.1 (lytic-phase antigen) im

121 lly significant differences were observed in antibody reactivity with a panel of six partial P1 polyp

122 roliferative cellular reactivity and 95% had antibody reactivity with at least one of the spirochetal

123 ssis-infected human donors were screened for antibody reactivity with Bordetella iron-repressible cel

124 with their control counterparts for overall antibody reactivity with organisms of different chlamydi

125 C and FlaB, only a few patients had marginal antibody reactivity with OspA.

126 Serum samples which differed in antibody reactivity with P1 polypeptides generated by pa

127 sorbent assay, demonstrated immunoglobulin G antibody reactivity with peptides 6 and 11 and a T-cell