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5 s and heterologous virus neutralization, and antibody-dependent cell cytotoxicity (ADCC) responses we
8 activity, anti-CMV IgG, and NK cell-mediated antibody-dependent cell cytotoxicity were present in ale
9 as prominent effector cells and induction of antibody-dependent cell phagocytosis as one of the prima
10 the immunologic synapse and potently induce antibody-dependent cell-mediated antiviral responses: (i
11 ngagement and provide them with an intrinsic antibody-dependent cell-mediated cytotoxic (ADCC) potent
12 d with their parental forms, potent in vitro antibody-dependent cell-mediated cytotoxicity (0.1-0.3 m
13 FcgammaRIIIa binding and function, including antibody-dependent cell-mediated cytotoxicity (ADCC) act
14 histochemistry, for affinity by BIACORE, for antibody-dependent cell-mediated cytotoxicity (ADCC) and
15 ental insights into the relationship between antibody-dependent cell-mediated cytotoxicity (ADCC) and
16 uring the killing of virus-infected cells by antibody-dependent cell-mediated cytotoxicity (ADCC) are
17 to ipilimumab by multicolor flow cytometry, antibody-dependent cell-mediated cytotoxicity (ADCC) ass
19 Results from serum transfer experiments and antibody-dependent cell-mediated cytotoxicity (ADCC) ass
21 evels of indirect tumor cell killing such as antibody-dependent cell-mediated cytotoxicity (ADCC) by
23 es that enhance tumor cell susceptibility to antibody-dependent cell-mediated cytotoxicity (ADCC) by
24 but their potential for cancer treatment via antibody-dependent cell-mediated cytotoxicity (ADCC) has
25 ils cultured from mouse bone marrow produced antibody-dependent cell-mediated cytotoxicity (ADCC) in
26 novicida DeltafopC immune serum, suggesting antibody-dependent cell-mediated cytotoxicity (ADCC) in
27 odies and explains how they achieve a higher antibody-dependent cell-mediated cytotoxicity (ADCC) pot
28 rent study, we evaluated the kinetics of the antibody-dependent cell-mediated cytotoxicity (ADCC) res
29 ion, downregulate HER3, and mediate enhanced antibody-dependent cell-mediated cytotoxicity (ADCC) via
30 oma activity by spontaneous cytotoxicity and antibody-dependent cell-mediated cytotoxicity (ADCC) whe
31 the susceptibility of HIV-infected cells to antibody-dependent cell-mediated cytotoxicity (ADCC), an
32 effector functions of antibodies, including antibody-dependent cell-mediated cytotoxicity (ADCC), in
33 asure the killing of virus-infected cells by antibody-dependent cell-mediated cytotoxicity (ADCC), we
35 effectively targeted CML LSPCs by selective antibody-dependent cell-mediated cytotoxicity (ADCC)-fac
36 ured by glycans that either promote or block antibody-dependent cell-mediated cytotoxicity (ADCC).
37 ability to eliminate virus-infected cells by antibody-dependent cell-mediated cytotoxicity (ADCC).
38 unctions of the innate immune system such as antibody-dependent cell-mediated cytotoxicity (ADCC).
39 their surface are preferentially targeted by antibody-dependent cell-mediated cytotoxicity (ADCC).
40 complement-dependent cytotoxicity (CDC) and antibody-dependent cell-mediated cytotoxicity (ADCC).
41 rum including pseudovirus neutralization and antibody-dependent cell-mediated cytotoxicity (ADCC).
42 urface can be bound by antibodies to mediate antibody-dependent cell-mediated cytotoxicity (ADCC).
43 enhanced virus replication and resistance to antibody-dependent cell-mediated cytotoxicity (ADCC).
44 ic antibodies, arising in part from enhanced antibody-dependent cell-mediated cytotoxicity (ADCC).
45 cy by palivizumab in the cotton rat and that antibody-dependent cell-mediated cytotoxicity activity c
46 as glycoengineered, which resulted in strong antibody-dependent cell-mediated cytotoxicity activity.
47 es, and generation of antibodies with potent antibody-dependent cell-mediated cytotoxicity activity.
49 (hYP7 and hYP9.1b) in the IgG format induced antibody-dependent cell-mediated cytotoxicity and comple
51 anti-neuraminidase antibodies weakly induced antibody-dependent cell-mediated cytotoxicity and enhanc
52 binding, neuraminidase inhibition, in vitro antibody-dependent cell-mediated cytotoxicity and in viv
54 feron gamma production, resulting in greater antibody-dependent cell-mediated cytotoxicity compared w
55 In addition, SEFL variants demonstrated no antibody-dependent cell-mediated cytotoxicity in vitro a
56 odies displayed cellular phagocytosis and/or antibody-dependent cell-mediated cytotoxicity in vitro O
57 outbreak variants of Ebola virus and mediate antibody-dependent cell-mediated cytotoxicity in vitro.
58 ch combines potent signaling inhibition with antibody-dependent cell-mediated cytotoxicity induction
59 ying specificities regulate the magnitude of antibody-dependent cell-mediated cytotoxicity induction.
60 ging Microscopy in Nanowell Grids to analyze antibody-dependent cell-mediated cytotoxicity kinetics o
62 reactive antibodies were capable of inducing antibody-dependent cell-mediated cytotoxicity to autolog
63 hu14.18 has been shown to elicit a level of antibody-dependent cell-mediated cytotoxicity toward hum
64 chanisms (complement-dependent cytotoxicity, antibody-dependent cell-mediated cytotoxicity, and antib
65 TF:FVIIa-dependent intracellular signaling, antibody-dependent cell-mediated cytotoxicity, and rapid
66 d optimized structure for the enhancement of antibody-dependent cell-mediated cytotoxicity, complemen
67 D16-dependent degranulation pathway, but not antibody-dependent cell-mediated cytotoxicity, contribut
68 receptor alpha that depletes eosinophils by antibody-dependent cell-mediated cytotoxicity, for patie
69 ween milk or plasma neutralization activity, antibody-dependent cell-mediated cytotoxicity, or HIV-1
70 ticular the IgG1 and IgG3 subclass mediating antibody-dependent cell-mediated cytotoxicity, seem to p
71 role in patients undergoing mAb therapy via antibody-dependent cell-mediated cytotoxicity, thus expl
72 and engagement of myeloid effector cells for antibody-dependent cell-mediated cytotoxicity, were simi
73 of NMO patient serum reduced by >95% CDC and antibody-dependent cell-mediated cytotoxicity, without i
74 ased in vivo tumor cell clearance, NKG2D- or antibody-dependent cell-mediated cytotoxicity-induced tu
85 ent cellular cytotoxicity (ADCC), and modest antibody-dependent cell-mediated virus inhibition (ADCVI
86 for Fcgamma receptor and variant function in antibody-dependent cell-mediated virus inhibition (ADCVI
88 l blood mononuclear cell assay, and moderate antibody-dependent, cell-mediated cytotoxicity activity
89 d followed by isotype switching for improved antibody-dependent, cell-mediated cytotoxicity activity.
90 and fixation as well as Fc-gamma-dependent, antibody-dependent, cell-mediated cytotoxity in both mur
91 de FcgammaR signals to stimulate or suppress antibody-dependent, cell-mediated depletion of antigen-b
94 ecificity) and effector function activities (antibody dependent cellular phagocytosis, cellular cytot
95 lobulin G1 antibody variant with compromised antibody-dependent cellular cytotoxicity (ADCC) activity
96 ust cross-clade binding and neutralizing and antibody-dependent cellular cytotoxicity (ADCC) activity
97 ngly, the presence of antibodies with potent antibody-dependent cellular cytotoxicity (ADCC) activity
98 antibodies (Abs), those capable of mediating antibody-dependent cellular cytotoxicity (ADCC) activity
99 (Abs) to the V1V2 region of gp120 with high antibody-dependent cellular cytotoxicity (ADCC) activity
100 and that this protection may correlate with antibody-dependent cellular cytotoxicity (ADCC) activity
104 ility to opsonize viral particles, to direct antibody-dependent cellular cytotoxicity (ADCC) against
105 b) directs natural killer (NK) cell-mediated antibody-dependent cellular cytotoxicity (ADCC) against
106 iminating latent HIV infection, specifically antibody-dependent cellular cytotoxicity (ADCC) and anti
107 These Treg suppressed cetuximab-mediated antibody-dependent cellular cytotoxicity (ADCC) and thei
109 phagocytosis, NK cell activation assays, and antibody-dependent cellular cytotoxicity (ADCC) assays.
110 Natural killer (NK) immune cells mediate antibody-dependent cellular cytotoxicity (ADCC) by aggre
111 ycan fucosylation have been shown to improve antibody-dependent cellular cytotoxicity (ADCC) by allow
112 g antibodies with effector functions such as antibody-dependent cellular cytotoxicity (ADCC) contribu
113 oss-reactive antibodies capable of mediating antibody-dependent cellular cytotoxicity (ADCC) effector
114 ible (CDi) neutralizing epitopes targeted by antibody-dependent cellular cytotoxicity (ADCC) effector
115 onses at an early time point correlated with antibody-dependent cellular cytotoxicity (ADCC) function
116 virus type 1 (HIV-1) Env and able to mediate antibody-dependent cellular cytotoxicity (ADCC) have bee
117 ng evidence supports a role for HIV-specific antibody-dependent cellular cytotoxicity (ADCC) in contr
118 -HIV-1 Env IgA antibodies and high levels of antibody-dependent cellular cytotoxicity (ADCC) inversel
122 Nonneutralizing antibodies (Abs) involved in antibody-dependent cellular cytotoxicity (ADCC) may prov
124 levels of HIV-1-neutralizing antibodies and antibody-dependent cellular cytotoxicity (ADCC) response
125 itopes) as targets of potentially protective antibody-dependent cellular cytotoxicity (ADCC) response
126 ding IgG and IgA as well as neutralizing and antibody-dependent cellular cytotoxicity (ADCC) response
128 complement-dependent cytotoxicity (CDC) and antibody-dependent cellular cytotoxicity (ADCC) through
129 ith prechallenge serum antienvelope avidity, antibody-dependent cellular cytotoxicity (ADCC) titers,
131 e HIV-1 reservoir.IMPORTANCE Mobilization of antibody-dependent cellular cytotoxicity (ADCC) to elimi
132 protein expressed by infected cells mobilize antibody-dependent cellular cytotoxicity (ADCC) to elimi
134 tes biological effector functions, including antibody-dependent cellular cytotoxicity (ADCC) which is
135 ating the factors that modulate HIV-specific antibody-dependent cellular cytotoxicity (ADCC) will hel
137 e breadth of HIV-1 gp120 and V1V2 responses, antibody-dependent cellular cytotoxicity (ADCC), and low
139 Very high IgG binding titers, substantial antibody-dependent cellular cytotoxicity (ADCC), and mod
140 b triggers natural killer (NK)-cell-mediated antibody-dependent cellular cytotoxicity (ADCC), but lit
141 functions, including tier 1 neutralization, antibody-dependent cellular cytotoxicity (ADCC), infecte
142 cts on virus infectivity, antibodies mediate antibody-dependent cellular cytotoxicity (ADCC), the kil
143 ese epitopes and sensitize infected cells to antibody-dependent cellular cytotoxicity (ADCC), we trea
144 e, core fucosylation significantly decreases antibody-dependent cellular cytotoxicity (ADCC), whereas
146 Fc N-glycans leads to drastic enhancement of antibody-dependent cellular cytotoxicity (ADCC), while t
147 Cross-reactive influenza virus-specific antibody-dependent cellular cytotoxicity (ADCC)-activati
148 ed that human natural killer (NK) cells, via antibody-dependent cellular cytotoxicity (ADCC)-like mec
149 tion with either lineage induces HA-specific antibody-dependent cellular cytotoxicity (ADCC)-mediatin
163 s, of which the major mechanism of action is antibody-dependent cellular cytotoxicity (eg, trastuzuma
164 infected cells by natural killer (NK) cells (antibody-dependent cellular cytotoxicity [ADCC]) or comp
165 te fucose-deficient antibodies with enhanced antibody-dependent cellular cytotoxicity activities.
166 g to FcgammaRIIIA and thereby decreasing the antibody-dependent cellular cytotoxicity activities.
167 No alteration in gamma-receptor binding and antibody-dependent cellular cytotoxicity activity was ob
168 by generation of reactive oxygen species or antibody-dependent cellular cytotoxicity activity, but l
169 cking core N-glycan residues mediated higher antibody-dependent cellular cytotoxicity against human t
170 An afucosylated Fc form (Pr20M) directed antibody-dependent cellular cytotoxicity against PRAME+H
171 fic CD16(pos) gammadelta T cells can perform antibody-dependent cellular cytotoxicity against stromal
172 ti-IL1RAP antibody capable of both achieving antibody-dependent cellular cytotoxicity and blocking of
173 pecific monoclonal antibodies display robust antibody-dependent cellular cytotoxicity and CD4-depende
175 tes killing of infected cells by Fc-mediated antibody-dependent cellular cytotoxicity and complement-
176 f a CD20-targeting antibody had no impact on antibody-dependent cellular cytotoxicity and did not cha
177 emistry, and a target for cetuximab-mediated antibody-dependent cellular cytotoxicity and in vivo eli
178 redicted susceptibility to cetuximab-induced antibody-dependent cellular cytotoxicity and occurred in
179 tes immunological effector functions such as antibody-dependent cellular cytotoxicity and phagocytosi
180 odification in the Fc domain that eliminates antibody-dependent cellular cytotoxicity at clinically r
181 ivation of the classical complement pathway, antibody-dependent cellular cytotoxicity by innate immun
182 he asymmetrically engineered Fc variants for antibody-dependent cellular cytotoxicity enhancement cou
184 Our study highlights the potential role that antibody-dependent cellular cytotoxicity might play in a
185 dy inhibits cell growth and induces in vitro antibody-dependent cellular cytotoxicity of human neurob
186 ne neutralization of RANKL with induction of antibody-dependent cellular cytotoxicity of natural kill
187 mulation, by the use of antibodies to induce antibody-dependent cellular cytotoxicity or to block iKI
188 NSCC sensitivity in a manner associated with antibody-dependent cellular cytotoxicity rather than EGF
189 levels of IgA antibodies, and high levels of antibody-dependent cellular cytotoxicity responses and H
190 In each study, protection correlated with antibody-dependent cellular cytotoxicity specific for CD
191 ed in N. benthamiana are capable of inducing antibody-dependent cellular cytotoxicity, an activity no
192 both macrophage-dependent FcgammaR-mediated antibody-dependent cellular cytotoxicity, and by direct
193 le mechanisms including apoptosis induction, antibody-dependent cellular cytotoxicity, antibody-depen
194 including complement-dependent cytotoxicity, antibody-dependent cellular cytotoxicity, antibody-depen
195 2M activates natural killer cells to enhance antibody-dependent cellular cytotoxicity, mediates compl
196 d functional activity (virus neutralization, antibody-dependent cellular cytotoxicity, phagocytosis,
197 f natural killer cells, the key mediators of antibody-dependent cellular cytotoxicity, to human AMR i
198 biting complement-dependent cytotoxicity and antibody-dependent cellular cytotoxicity, which suggests
199 ) arm that was derived from broadly binding, antibody-dependent cellular cytotoxicity-mediating antib
200 highest titers of binding, neutralizing, and antibody-dependent cellular cytotoxicity-mediating antib
201 ecific MAbs that showed Galcer blocking, the antibody-dependent cellular cytotoxicity-mediating CH38
213 bs, readily induced by vaccines, can trigger antibody-dependent cellular effector functions, through
215 ed functional activities, including ADCC and antibody-dependent cellular phagocytosis (ADCP) activiti
216 ve antitumor antibody responses by enhancing antibody-dependent cellular phagocytosis (ADCP) in xenog
217 xenografts by enhancing macrophage-mediated antibody-dependent cellular phagocytosis (ADCP), but syn
219 6M0-mcMMAF recruits macrophages and mediates antibody-dependent cellular phagocytosis of MM cells.
220 dy-dependent cell-mediated cytotoxicity, and antibody-dependent cellular phagocytosis) and direct apo
221 n, antibody-dependent cellular cytotoxicity, antibody-dependent cellular phagocytosis, and complement
222 y, antibody-dependent cellular cytotoxicity, antibody-dependent cellular phagocytosis, programmed cel
223 to avidly capture HIV virions and to mediate antibody-dependent cellular phagocytosis, suggesting a r
225 their surface are preferentially targeted by antibody-dependent cellular-mediated cytotoxicity (ADCC)
226 I antibodies of various isotypes in ADCC and antibody-dependent cellular-phagocytosis (ADCP) assays.
227 ell surface, thereby reducing recognition by antibody-dependent clearance by natural killer cells.
230 ng capacity) and has increased resistance to antibody-dependent complement-mediated killing compared
231 ansferase that is required for resistance to antibody-dependent complement-mediated killing in a muri
232 ndings indicate that increased resistance to antibody-dependent complement-mediated killing secondary
233 ors of complement activation (RCA) to resist antibody-dependent complement-mediated lysis (ADCML).
234 eir viral envelopes and, as a result, escape antibody-dependent complement-mediated lysis (ADCML).
235 quality and the quantity of NK cell-mediated antibody-dependent cytotoxicity by endowing more NK cell
236 ation activity limited to tier 1 viruses and antibody-dependent cytotoxicity responses (ADCC) after D
239 v antigen engagement which lead to effective antibody-dependent effector function directed to the non
240 s provide additional evidence that effective antibody-dependent effector function in the cluster A re
241 ad compound against all 4 serotypes of DENV, antibody-dependent enhanced (ADE) infection, and ex vivo
243 lusion, here we find that in the presence of Antibody Dependent Enhancement (ADE) heterogeneity can i
244 ion showed significant capacity for in vitro antibody dependent enhancement of Dengue-1, 2, 3 and 4 s
245 evere dengue disease focus on the process of antibody-dependent enhancement (ADE) as a primary risk f
249 protection against DENV disease and prevents antibody-dependent enhancement (ADE) of disease in mice.
250 mmune serum, it has been shown in vitro that antibody-dependent enhancement (ADE) of ZIKV infection c
251 ble of both cross-neutralization, as well as antibody-dependent enhancement (ADE) of ZIKV infection.
252 cross-reaction to ZIKV and was able to drive antibody-dependent enhancement (ADE) of ZIKV infection.
254 e virus (DENV) infections is associated with antibody-dependent enhancement (ADE), and it was recentl
258 Progression to DHF/DSS is attributed to antibody-dependent enhancement (ADE); however, because o
259 tions in flavivirus immune vaccinees such as Antibody-Dependent Enhancement (ADE, a phenomenon involv
260 ovide statistical support for the process of antibody-dependent enhancement (but not original antigen
268 ted against DENV-2 by using a mouse model of antibody-dependent enhancement of infection (ADE)-induce
270 zed all four serotypes of DENV, and mediated antibody-dependent enhancement of infection in Fc recept
277 st closely paralleled clinical outcomes, IgE antibody-dependent functional assays remained inhibited
279 osolic immunoglobulin receptor that mediates antibody-dependent intracellular neutralization (ADIN).
280 ly induced lymphoma Gal-1 expression ablated antibody-dependent lymphoma phagocytosis in vitro and ly
281 ibits subsequent S. aureus acquisition in an antibody-dependent manner and elicits antibody that cros
286 ovide epidemiological evidence that multiple antibody-dependent mechanisms contribute to protective i
287 Pathological and clinical studies implicate antibody-dependent mechanisms in the immunopathogenesis
288 primary lymphoma cells through monocyte- and antibody-dependent mechanisms, we found that lymphoma de
290 of FcgammaR function dramatically inhibited antibody-dependent murine ITP and successfully circumven
293 to the production of class-switched anti-MOG antibodies, dependent on the presence of hemagglutinin-s
294 ed binding antibody against scaffolded V1V2, antibody-dependent phagocytic activity against VLP-coate
295 y-dependent cellular cytotoxicity (ADCC) and antibody-dependent phagocytosis (ADP), are unclear.
296 Using intravital microscopy, we found that antibody-dependent phagocytosis (ADPh) by macrophages is
300 urvival, suggesting antibody-independent and antibody-dependent roles for B cells in the outcome to s
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