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1 nd vice versa because of a phenomenon termed antibody-dependent enhancement.
2 erity of secondary flavivirus infections via antibody-dependent enhancement.
3 ve broad HA reactivity, yet they do not have antibody-dependent enhancement activity.
4 lusion, here we find that in the presence of Antibody Dependent Enhancement (ADE) heterogeneity can i
5 evere dengue disease focus on the process of antibody-dependent enhancement (ADE) as a primary risk f
6 p-regulated DENV infection by a mechanism of antibody-dependent enhancement (ADE) in a variety of Fc
7 investigates the complex dynamics induced by antibody-dependent enhancement (ADE) in multiserotype di
8                                              Antibody-dependent enhancement (ADE) is implicated in se
9 sed severity seen in secondary infections is antibody-dependent enhancement (ADE) leading to increase
10                                 The proposed antibody-dependent enhancement (ADE) mechanism for sever
11 tate Zika virus (ZIKV) infection through the antibody-dependent enhancement (ADE) mechanism.
12                                              Antibody-dependent enhancement (ADE) of dengue virus (DE
13 protection against DENV disease and prevents antibody-dependent enhancement (ADE) of disease in mice.
14 mmune serum, it has been shown in vitro that antibody-dependent enhancement (ADE) of ZIKV infection c
15 cross-reaction to ZIKV and was able to drive antibody-dependent enhancement (ADE) of ZIKV infection.
16 ble of both cross-neutralization, as well as antibody-dependent enhancement (ADE) of ZIKV infection.
17                                    Moreover, antibody-dependent enhancement (ADE) was not observed ag
18                                              Antibody-dependent enhancement (ADE), a phenomenon in wh
19                                              Antibody-dependent enhancement (ADE), a process best des
20 e virus (DENV) infections is associated with antibody-dependent enhancement (ADE), and it was recentl
21            This phenomenon is referred to as antibody-dependent enhancement (ADE).
22 eutralization potency, enhanced infection by antibody-dependent enhancement (ADE).
23 e-infection, mainly because of the effect of antibody-dependent enhancement (ADE).
24 -glycoprotein (g)D and anti-gK antibodies on antibody-dependent enhancement (ADE).
25 tate infection through a phenomenon known as antibody-dependent enhancement (ADE).
26 ersistent infection, by Fc-receptor-mediated antibody-dependent enhancement (ADE).
27      Progression to DHF/DSS is attributed to antibody-dependent enhancement (ADE); however, because o
28 tions in flavivirus immune vaccinees such as Antibody-Dependent Enhancement (ADE, a phenomenon involv
29 kly neutralizing they also may contribute to antibody-dependent enhancement and flavi virus pathogene
30 unopathology is thought to play a part, with antibody-dependent enhancement and massive immune activa
31 nteractions, arising from cross-immunity and antibody-dependent enhancement, between related pathogen
32 ovide statistical support for the process of antibody-dependent enhancement (but not original antigen
33                         Complement-mediated, antibody-dependent enhancement (C'-ADE) of HIV-1IIIB and
34                                              Antibody-dependent enhancement has been implicated in mo
35  human sera (PHS) and the major mediators of antibody-dependent enhancement in the presence of PHS.
36       Among the 4 serotypes of dengue virus, antibody-dependent enhancement is thought to enhance vir
37                                              Antibody-dependent enhancement may explain the severe di
38 ion showed significant capacity for in vitro antibody dependent enhancement of Dengue-1, 2, 3 and 4 s
39                                 In contrast, antibody-dependent enhancement of bacterial infection is
40 I agonist blocked both primary infection and antibody-dependent enhancement of DENV infection.
41 -gamma receptor (FcgammaR), a process termed antibody-dependent enhancement of DENV infection.
42 on in the Fc region which completely ablates antibody-dependent enhancement of DENV replication in vi
43 ted against DENV-2 by using a mouse model of antibody-dependent enhancement of infection (ADE)-induce
44                 This process has been termed antibody-dependent enhancement of infection and has sign
45 zed all four serotypes of DENV, and mediated antibody-dependent enhancement of infection in Fc recept
46                                              Antibody-dependent enhancement of infectivity was observ
47                   These results suggest that antibody-dependent enhancement of NK activity may yield
48 n in late infancy is a direct consequence of antibody-dependent enhancement of replication.
49                     Although suspected, such antibody-dependent enhancement of severe disease has not
50 raction of FcgammaRI with A1H3 underlays the antibody-dependent enhancement of the cellular effects o
51 rceived wisdom, patterns generated solely by antibody-dependent enhancement or heterogeneity in virus
52                                              Antibody-dependent enhancement was observed in animals i

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