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1 s for safer dosing of this widely prescribed anticancer agent.
2 gene under current investigation as a novel anticancer agent.
3 rest in the potential use of metformin as an anticancer agent.
4 me inhibitor with desirable properties as an anticancer agent.
5 29 that potentially strengthen its use as an anticancer agent.
6 genetic protein 4 (BMP4) has potential as an anticancer agent.
7 n demonstrated to have potent activity as an anticancer agent.
8 PC is a natural pigment with potential as an anticancer agent.
9 days after adjudication) for the index oral anticancer agent.
10 ing its in vivo efficacy as a potential lead anticancer agent.
11 (HCT-116 colon cancer xenograft mouse model) anticancer agent.
12 Doxazolidine (doxaz) is a new anthracycline anticancer agent.
13 It also is in clinical trials as an anticancer agent.
14 explored the potential of stauprimide as an anticancer agent.
15 mpound of profound interest as a prospective anticancer agent.
16 nano-oral delivery system for this promising anticancer agent.
17 dicine has gained approval as a new class of anticancer agent.
18 rol (RES) has been studied extensively as an anticancer agent.
19 a styryl benzylsulfone, is a phase III stage anticancer agent.
20 serve as companion diagnostics for targeted anticancer agents.
21 l formulations of TPT and other weakly-basic anticancer agents.
22 erable interest as potential chemopreventive anticancer agents.
23 efficacious and nontoxic PAI-1 inhibitors as anticancer agents.
24 luable opportunities for rapid assessment of anticancer agents.
25 uinolinones was synthesized and evaluated as anticancer agents.
26 has the potential to enhance the efficacy of anticancer agents.
27 of direct Ras inhibitors as a novel class of anticancer agents.
28 ent a new class of potent microtubule-active anticancer agents.
29 cting compounds may represent novel powerful anticancer agents.
30 on the future development of these effective anticancer agents.
31 TH1 in general as a promising novel class of anticancer agents.
32 f histone deacetylase inhibitors (HDACIs) as anticancer agents.
33 adjudicated prescription for any of 38 oral anticancer agents.
34 exhibit reduced Bim levels and resistance to anticancer agents.
35 a-2) increased sensitivity to platinum-based anticancer agents.
36 inhibitors are gaining interest as potential anticancer agents.
37 oping specific Cdc20 inhibitors as effective anticancer agents.
38 ir immunogenic capacity as novel and dynamic anticancer agents.
39 e ProTide approach to generate promising new anticancer agents.
40 opment of glutamine metabolism inhibitors as anticancer agents.
41 a response mechanism to metabolic stress and anticancer agents.
42 y of various types of therapeutics including anticancer agents.
43 with respect to the development of potential anticancer agents.
44 m of action for PEITC and other ROS-inducing anticancer agents.
45 of alternate oral, injectable, or infusible anticancer agents.
46 a previously unpursued route for developing anticancer agents.
47 stress and contributes to resistance against anticancer agents.
48 nstrated mixed outcomes regarding statins as anticancer agents.
49 gy for development of specific and effective anticancer agents.
50 en prepared as a new chemotype for potential anticancer agents.
51 r sensitivities and resistance mechanisms to anticancer agents.
52 ork synergistically to create more effective anticancer agents.
53 a first step toward a new class of potential anticancer agents.
54 azones (BpT) class, show marked potential as anticancer agents.
55 spliceosome-modulating activity as potential anticancer agents.
56 us has become an attractive target for novel anticancer agents.
57 position 2 of the indole were synthesized as anticancer agents.
58 ce of targeting ERRalpha with antagonists as anticancer agents.
59 size, indicating their in vivo potential as anticancer agents.
60 tothecin and a variety of other DNA-damaging anticancer agents.
61 any type of DSB, including those induced by anticancer agents.
62 analogues have significant potential as new anticancer agents.
63 r the use of cell surface NCL antagonists as anticancer agents.
64 select patients for treatment with specific anticancer agents.
65 isen in developing Na,K-ATPase inhibitors as anticancer agents.
66 ms of RCC provide specific targets for novel anticancer agents.
67 toposide are widely used effective cytotoxic anticancer agents.
68 t of farnesyltransferase inhibitors as novel anticancer agents.
69 unds are members of a promising new class of anticancer agents.
70 t response and inform the development of new anticancer agents.
71 ligands with the aim to obtain multitargeted anticancer agents.
72 inct from that of these clinically validated anticancer agents.
73 rotects normal tissues during treatment with anticancer agents.
74 Autophagy mediates resistance to various anticancer agents.
75 have received comprehensive investigation as anticancer agents.
76 to quantify tumor changes during exposure to anticancer agents.
77 osomal delivery of poorly soluble, ionizable anticancer agents.
78 functions and can be developed as potential anticancer agents.
79 otubules, is a target for the development of anticancer agents.
80 able preclinical stratification schemata for anticancer agents.
81 s good candidates for further development as anticancer agents.
82 several distinct gold-containing indoles as anticancer agents.
83 el class of compounds that have potential as anticancer agents.
84 tors (FTIs and GGTIs) as chemical probes and anticancer agents.
85 meet safety and efficacy criteria for use as anticancer agents.
86 very and biological evaluations of PACMAs as anticancer agents.
87 compounds represent a new potential class of anticancer agents.
88 soform, and this may limit their efficacy as anticancer agents.
89 eudolaric acid B (PAB) holds promise for new anticancer agents.
90 inhibitors, potential anti-inflammatory and anticancer agents.
91 able core for optimization to identify novel anticancer agents.
92 tunity for endothelial-specific targeting of anticancer agents.
93 he development of these rigidin analogues as anticancer agents.
94 temic multi-delivery of poorly water soluble anticancer agents.
95 lysates for the early stage of apoptosis or anticancer agents.
96 as a potential target for the development of anticancer agents.
97 - and crinine-type alkaloids show promise as anticancer agents.
98 a useful structure for the generation of new anticancer agents.
99 These processes are targets of many anticancer agents.
100 of water-soluble and/or poorly water-soluble anticancer agents.
101 cumulate in DNA following treatment with the anticancer agents 5-fluorouracil and 5-fluorodeoxyuridin
102 and high drug encapsulation is desirable for anticancer agents, a mechanistic understanding is requir
104 analysis of the direct effect of widely used anticancer agents (aclarubicin, ICRF-193, VM26, doxorubi
106 tructure classes were evaluated as potential anticancer agents against human cancer cells from variou
107 they characterize DOT1L inhibitors as novel anticancer agents against MYCN-amplified neuroblastoma.
109 ed the activity of ISC-4 and 19 FDA-approved anticancer agents, alone or in combination, against the
112 Our findings show that hSGZ is an effective anticancer agent and a potential candidate for clinical
113 s study illuminates the function of an early anticancer agent and suggests an intersection with ongoi
114 he chemotherapeutic drug Taxol, an important anticancer agent and the only known stabilizer that redu
115 erlying mechanism of action of aspirin as an anticancer agent and, based on the rapid metabolism of a
116 NAP have been suggested as immunotherapeutic anticancer agents and adjuvants for vaccination but with
117 ecent research on other poorly water-soluble anticancer agents and delivery of drug combinations (i.e
119 es are nanocarriers for poorly water-soluble anticancer agents and have advanced paclitaxel (PTX) to
120 udies that have expanded CTCs for testing of anticancer agents and how these approaches might be used
121 e therapeutic utility of GSTO1 inhibitors as anticancer agents and identify the novel cellular pathwa
122 g the administration of different classes of anticancer agents and in all cases observed that the mod
123 hat SIRT2-selective inhibitors are promising anticancer agents and may represent a general strategy t
124 -directed intraperitoneal targeting of other anticancer agents and nanoparticles using peptides and o
126 ween the mechanisms of action of these novel anticancer agents and the imaging appearance of tumor re
128 a bone targeting ligand, doxorubicin as the anticancer agent, and either an acid-sensitive bond (1)
129 ally important antibiotics, antifungals, and anticancer agents are actually present in the soils of N
134 Moreover, because the latest generations of anticancer agents are founded on biological mechanisms,
135 , providing a rationale for repurposing this anticancer agent as an Alzheimer's disease (AD) therapeu
137 enal transplantation, and of rapamycin as an anticancer agent, as well as from experimental studies.
138 d with paclitaxel, a microtubule-stabilizing anticancer agent, as well as with 6-coumarin, a fluoresc
139 implications); and there are highly specific anticancer agents available that effectively modulate th
143 ized DNA-targeted platinum-containing hybrid anticancer agents based on metal-mediated amine-to-nitri
144 new 2-phenylindole derivatives as potential anticancer agents bearing the 3,4,5-trimethoxyphenyl moi
145 as promising probes of DNA structure and as anticancer agents because of their unique photophysical
146 ell death makes CDD a potentially attractive anticancer agent, because tumor resistance to the main m
147 vance to a broad spectrum of next-generation anticancer agents being developed by the medicinal inorg
151 e cancer cells are resistant to conventional anticancer agents but are sensitive to glycolytic inhibi
152 have demonstrated or predicted potential as anticancer agents, but a detailed structural basis for t
153 proliferation and is inhibited by promising anticancer agents, but its mode of action and the conseq
155 m, where the biologic effects of traditional anticancer agents can be delineated, prognostic and pred
156 n drug-mediated anticancer activity and that anticancer agents can be identified through their abilit
158 Despite the discovery of a large number of anticancer agents, cancer still remains among the leadin
160 sive efforts to target mutated RAS proteins, anticancer agents capable of selectively killing tumour
161 e) complexes have been shown to be promising anticancer agents, capable of overcoming major drawbacks
162 cules and their gene clusters, including the anticancer agent cinerubin B from Streptomyces sp. SPB74
163 sociated with the pattern of response to the anticancer agent cisplatin in human melanoma cells in vi
168 tecan (LMP400) and indimitecan (LMP776), two anticancer agents currently under investigation in a Pha
169 and cancer and also suggest the potential of anticancer agents designed to specifically neutralize ex
171 years, with the development of numerous oral anticancer agents, dosing options are arbitrarily and in
172 -dimethoxyflavone) has great potential as an anticancer agent, due to its specific chemopreventive ac
173 clinical translation of poorly water-soluble anticancer agents, especially for promising, rapidly eme
174 enium complexes are promising candidates for anticancer agents, especially NKP-1339 (sodium trans-[te
178 t are currently under development for use as anticancer agents following the FDA approval of two HDAC
179 of EGFR protein abundance and as a potential anticancer agent for aneuploidy-driven colorectal cancer
180 ptide has potential to be developed as a new anticancer agent for intratumoral administration and is
181 cancers and that MLN4924 may be an effective anticancer agent for targeted ovarian cancer therapy.
183 ant approach for the development of targeted anticancer agents for ovarian cancer therapy, and they c
185 in situ generation of a triazole-containing anticancer agent from two benign components, opening up
186 reductase (RNR) is an attractive target for anticancer agents given its central function in DNA synt
188 mmetric bis-benzimidazoles (BBZ) designed as anticancer agents have previously been shown to possess
189 -arylpyrrole (ARAP) derivatives as potential anticancer agents having different substituents at the p
190 Recent studies indicate that a class of anticancer agents, histone deacetylase (HDAC) inhibitors
191 drug conjugates hold considerable promise as anticancer agents, however, producing them remains a cha
192 These efforts include screening for novel anticancer agents, identifying novel therapeutic targets
193 ormin is currently considered as a promising anticancer agent in addition to its anti-diabetic effect
194 tion in vitro and in vivo and an efficacious anticancer agent in animal models of human cancers.
199 otropic autophagy inhibitors may have use as anticancer agents in combination with conventional thera
203 iety of combinations of different classes of anticancer agents including small molecule drugs and bio
204 ollowing exposure to several stress-inducing anticancer agents, including ionizing radiation (IR), et
205 hat copes with DNA damage induced by several anticancer agents, including ionizing radiation and temo
206 tion of Rac hyperactivation to resistance to anticancer agents, including targeted therapies, as well
207 bocyclic analogues of an important series of anticancer agents inhibiting MDM2-p53 interactions.
208 he maximum tolerated dose of investigational anticancer agents is based on the cytotoxic chemotherapy
213 the development of BXI(s) as a new class of anticancer agents is warranted and represents a novel st
214 Although TRAIL is considered a potential anticancer agent, it enhances tumor progression by activ
215 ones with camptothecin (CPT), a largely used anticancer agent, KuQs have been tested against Cisplati
216 ring resistance to many structurally diverse anticancer agents, leading to the phenomenon called mult
218 he real-world clinical effectiveness of oral anticancer agents may be diminished and may not emulate
219 ests that sorafenib, in addition to being an anticancer agent, may have potential to be developed as
220 a (PDA) cells by treatment with DNA-damaging anticancer agents (mitomycin C, oxaliplatin, cisplatin,
221 g lead for further development to obtain new anticancer agents of potential value for the treatment o
222 ly apparent that treatment with a variety of anticancer agents often is associated with adverse neuro
223 al proof of concept for a survivin-targeting anticancer agent, our work offers novel in silico screen
226 omprehensive conformational analysis for the anticancer agent pironetin (1) was achieved by molecular
228 endazole, which may represent a new class of anticancer agents showing selective toxicity against can
229 received much attention as immunotherapeutic anticancer agents since the discovery that they harbor f
230 omatitis virus (VSV) are being considered as anticancer agents since they readily infect transformed
231 ivery of siRNA/miRNA combinations with other anticancer agents (siRNA/miRNA/pDNA/chemodrugs) to treat
232 nes such as TP53, BRAF, ERBBs, and ATAD5 and anticancer agents such as nutlin, vemurafenib, erlotinib
234 is in human cancer and are targets for novel anticancer agents such as the Bcl-2 antagonists, ABT-263
235 iaryl (biaryl-benzenesulfonamides) as potent anticancer agents targeting both STAT3 and tubulin.
238 his study, we present a small-molecule-based anticancer agent that acts by inhibiting dimerization of
239 ddition of iniparib, a novel investigational anticancer agent that does not possess characteristics t
241 ed apoptosis-inducing ligand) is a potential anticancer agent that induces apoptosis in a wide variet
243 tosis-inducing ligand (TRAIL) is a promising anticancer agent that kills various tumor cells without
246 pe compounds form a small distinct family of anticancer agents that are involved in both photo-activa
247 ted their potential as promising theranostic anticancer agents that can circumvent cisplatin resistan
248 ting PIP3 binding by PH domains as potential anticancer agents that can simultaneously interfere with
249 hes an integrated approach for testing novel anticancer agents that captures the inherent variability
250 compound would constitute a unique class of anticancer agents that could potentially have significan
251 a possible new route for the development of anticancer agents that could suppress KRAS expression.
252 r-chain n-3 MUFAs may be prototypes of novel anticancer agents that decrease the formation of PGE(2)
254 cs have facilitated the development of novel anticancer agents that have decreased side effects and i
255 Recombinant immunotoxins (RITs) are potent anticancer agents that have produced many complete remis
259 r therapies involve the systemic delivery of anticancer agents that neither discriminate between canc
260 nia is a major side effect of a new class of anticancer agents that target histone deacetylase (HDAC)
262 atment, and there is a clinical need for new anticancer agents that would selectively target MDR mali
263 affinity of ellipticine, a DNA-intercalating anticancer agent, the relative energetics between the A-
266 nts a potent strategy for using conventional anticancer agents to alter the tumor microenvironment an
269 barrier (BBB) prevents the transport of most anticancer agents to the central nervous system and rest
271 to monitor liposomal release kinetics of the anticancer agent topotecan (TPT) in physiological fluids
275 of specialized metabolites that includes the anticancer agent vincristine, antimalarial quinine and n
277 rspective of using HTyr-derived compounds as anticancer agents, we have taken into account only studi
278 ng pursuit of CXCR4 antagonists as potential anticancer agents, we recently developed a potent, selec
279 ese cells, for the first time, two different anticancer agents were synthesized simultaneously intrac
280 cisplatin as the drug, since it is a potent anticancer agent which exhibits serious side effects due
282 g conjugates (ADCs) are a promising class of anticancer agents which have undergone substantial devel
283 that serves both as an anti-inflammatory and anticancer agent, which differentially affects MDSC acti
284 es to use the toxin as a potent pH-selective anticancer agent, which predominantly targets highly pro
285 and complications associated with the novel anticancer agents will be summarized, since these are fr
286 oof-of-concept for ABDNAZ as a promising new anticancer agent with a favorable toxicity profile, eith
287 further investigation as a novel preclinical anticancer agent with application in the treatment of nu
289 Acylfulvenes are a class of experimental anticancer agents with a unique repair profile suggestin
291 of 7-azaindenoisoquinolines resulted in new anticancer agents with improved Top1 inhibitory potencie
292 II) complexes are a novel class of promising anticancer agents with potential clinical significance.
293 model coupled tumor responses to individual anticancer agents with their mechanisms of action in viv
294 ase I (Top1) inhibitors are a novel class of anticancer agents with two compounds in clinical trials.
295 is focused on the benzoquinone ansamycins as anticancer agents, with several derivatives of the natur
298 eered MSCs loaded with high concentration of anticancer agents without affecting their tumor-targetin
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