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1 poptosis and p21 repression may also have an anticancer effect.
2 minimal toxicity, and 11a gave a significant anticancer effect.
3 te mechanism by which ibuprofen may exert an anticancer effect.
4 to new clinical strategies to strengthen its anticancer effect.
5 with stage IV NSCLC, suggesting a potential anticancer effect.
6 vity is not always required for the enzyme's anticancer effect.
7 ng antibodies (nAbs) that can diminish their anticancer effect.
8 bilize the G-quadruplexes were shown to have anticancer effects.
9 f the benefits of lifelong CR, including its anticancer effects.
10 ce and telomere-based crisis, exert powerful anticancer effects.
11 ith cerebroprotective, cardioprotective, and anticancer effects.
12 poptosis even in the absence of demonstrable anticancer effects.
13 instant antibacterial effects and sustained anticancer effects.
14 mittent calorie restriction (ICR) have shown anticancer effects.
15 stituent (6a and 6b) exhibited the strongest anticancer effects.
16 r more than 50 years, has been shown to have anticancer effects.
17 ortant roles in metformin's antidiabetic and anticancer effects.
18 n of phenothiazines as a class of drugs with anticancer effects.
19 ol), involve pro-apoptotic ceramide in their anticancer effects.
20 bitors Physcion and its derivative S3, shows anticancer effects.
21 ly bioactive product that exhibits potential anticancer effects.
22 ulatory effects that may contribute to their anticancer effects.
23 e nanoparticle intratumoral distribution and anticancer effects.
24 ulation of regulatory T cells, and preserved anticancer effects.
25 an be targeted by small molecules to achieve anticancer effects.
26 molecular mechanisms by which it exerts its anticancer effects.
27 ng antibodies (nAbs) that can diminish their anticancer effects.
28 of drug cocktails while boosting synergistic anticancer effects.
29 own to bind G-quadruplex structures, showing anticancer effects.
30 suggesting a possible association with their anticancer effect; (2) multiple HMG boxes contributed ad
31 administered in animal models to achieve an anticancer effect and by the even higher doses required
32 ates a new mechanism for the emodin-mediated anticancer effect and justifies further investigation of
34 iofrequency (RF) ablation has shown superior anticancer effects and greater survival benefit with res
37 ynchronized delivery system elicits enhanced anticancer effects and merits further development in the
39 tea, has been shown, for example, to possess anticancer effects, anti-HIV effects, neuroprotective ef
42 likely to be an important mechanism for its anticancer effects because it protects cutaneous APC fro
44 cing DNA hypomethylation may have short-term anticancer effects, but might also help speed tumor prog
46 tat, a histone deacetylase inhibitor, exerts anticancer effects by both histone and nonhistone-mediat
48 drugs (NSAIDs) are believed to mediate their anticancer effects by inducing apoptosis but the molecul
50 illustrate how Hsp70 inhibitors mediate the anticancer effects by targeting both tumor cells and tum
53 of HDACI and the ganciclovir (GCV)-mediated anticancer effect contributed by HDACI-induced and p21-d
55 nese herbal medicine, is reported to exhibit anticancer effects; however, its mechanism of action is
56 n of cyclooxygenase (COX) with NSAIDs has an anticancer effect in animal models of colon, urinary bla
58 the first dual binder 3 possessing a higher anticancer effect in GBM cells than the standards PK1119
59 he evidence suggests that ICR exerts greater anticancer effect in genetically engineered mouse models
60 emonstrate that RARbeta may exert its potent anticancer effect in part through its unique anti-AP-1 a
61 taxel or vinorelbine exhibited a synergistic anticancer effect in these human breast cancer cells in
62 potent SIRT2-specific inhibitor with a broad anticancer effect in various human cancer cells and mous
66 How DNA methylation inhibitors exert their anticancer effects in patients is not well understood.
67 ase, and zoledronic acid has shown potential anticancer effects in preclinical and clinical studies.
68 e micellar nanocomplexes, which have greater anticancer effects in vitro and in vivo than the free pr
69 nger protein PARP-1 indicate that they exert anticancer effects in vitro based on different mechanism
72 RNAi targeting FAS gene demonstrate systemic anticancer effects in vivo, our results render FAS as a
73 roposed mechanism by which PS-341 exerts its anticancer effect is inactivation of nuclear factor-kapp
79 oral administration, we propose that maximum anticancer effect may be achieved by nanoemulsion mediat
80 inhibition of cyclooxygenase-1, whereas its anticancer effects may be due to inhibition of cyclooxyg
84 We investigated the in vitro and in vivo anticancer effect of combining lysosomal membrane permea
86 em that has the potential for evaluating the anticancer effect of HDACIs on cancer cells by multiple
87 d with NDI, thus further indicating that the anticancer effect of NDI/2DG combination was indeed due
94 d and translate our observations on in vitro anticancer effect of silibinin/silymarin to an in vivo p
96 to investigate the chemical composition and anticancer effect of the leaf essential oil of Xylopia f
103 e used as a novel strategy to potentiate the anticancer effects of adoptively infused NK cells in pat
108 is, combined with the recent findings of the anticancer effects of bisphosphonates, cyclooxygenase-2
114 ochemical studies indicating that the potent anticancer effects of CR and disrupted insulin/IGFI rece
118 This novel effect may contribute to the anticancer effects of DIM because IFNgamma plays an impo
119 t may provide important clues to explain the anticancer effects of DIM because it is well known that
120 uate the molecular mechanisms underlying the anticancer effects of EF24 on CCA tumor growth and metas
122 f evidence supports the in vitro and in vivo anticancer effects of genistein, a soybean isoflavone.
125 N-gammaR were particularly important for the anticancer effects of HDACi, and vorinostat and IFN-gamm
126 biology and (4) pharmacological data showing anticancer effects of HIF-1 inhibitors in mouse models o
128 ile the molecular events associated with the anticancer effects of JS-K, HL-60 leukemia cells were tr
130 TR is an important upstream modulator of the anticancer effects of NSAIDs and that ibuprofen inductio
131 chanisms have been postulated to explain the anticancer effects of NSAIDs, they do not involve merely
132 study, we explored if paricalcitol enhanced anticancer effects of other clinically useful drugs in v
135 r a preclinical rationale to investigate the anticancer effects of PTP1B inhibitors currently being s
139 its physical interaction with AP-1, promotes anticancer effects of retinoids by potentiating their an
146 contributes to oncogenesis and underlies the anticancer effects of silvestrol and related compounds.
149 ct immune system was required for the robust anticancer effects of the HDACi vorinostat and panobinos
152 well as a direct comparison of the in vitro anticancer effects of the two clinically available COX-2
156 aditional Chinese medicine, can modulate the anticancer effects of TRAIL, the cytokine that is curren
160 erpene from tropical ginger, can enhance the anticancer effects of tumor necrosis factor-related apop
161 ination resulted in a remarkable synergistic anticancer effect on intracranial human and murine gliob
163 inhibit ATP1A1 function, exhibited selective anticancer effects on STK11 mutant lung cancer cell line
164 (IPA) suggested that curcumin may exert its anticancer effects over multiple critical biological pat
165 ity, effective tumour targeting and superior anticancer effects owing to favourable doxorubicin-bindi
168 xpression in HPV-positive HNSCC has a global anticancer effect resulting in a decrease in cell prolif
169 c-Myc/miR-29c/REV3L signalling may have dual anticancer effects, sensitizing the resistant tumours to
170 ta suggest that TLS inhibition may have dual anticancer effects, sensitizing tumors to therapy as wel
171 erior in their selective apoptosis-mediating anticancer effect than free form of these proteins and 5
172 ogenic effect, and a significant "bystander" anticancer effect that leads to enhanced production of t
174 inhibiting telomerase may not result in the anticancer effects that were originally hypothesized.
176 d transferrin receptor and exhibit potential anticancer effects through a signaling mechanism that is
177 e, bortezomib) are unlikely to mediate their anticancer effects through suppression of NF-kappaB.
178 ic acid (trans-RA) and other retinoids exert anticancer effects through two types of retinoid recepto
179 ng the first molecular target with potential anticancer effect, translating into the development of t
181 d long-term use of NSAIDs is required for an anticancer effect--up to 15 or 20 years before a reduced
182 To correlate the AT1 receptor blockage to anticancer effects, VEGF levels and microvessel densitie
183 AR gamma agonists coordinately mediate their anticancer effect via both COX-dependent (inhibition of
184 xerts its highly potent in vitro and in vivo anticancer effects via tubulin-based antimitotic mechani
185 atural product previously unreported to have anticancer effects, was found to have potent antimyeloma
187 ent chloroquine also displayed a synergistic anticancer effect with 2DG, whereas glucose deprivation
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