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1 poptosis and p21 repression may also have an anticancer effect.
2 minimal toxicity, and 11a gave a significant anticancer effect.
3 te mechanism by which ibuprofen may exert an anticancer effect.
4 to new clinical strategies to strengthen its anticancer effect.
5  with stage IV NSCLC, suggesting a potential anticancer effect.
6 vity is not always required for the enzyme's anticancer effect.
7 ng antibodies (nAbs) that can diminish their anticancer effect.
8 bilize the G-quadruplexes were shown to have anticancer effects.
9 f the benefits of lifelong CR, including its anticancer effects.
10 ce and telomere-based crisis, exert powerful anticancer effects.
11 ith cerebroprotective, cardioprotective, and anticancer effects.
12 poptosis even in the absence of demonstrable anticancer effects.
13  instant antibacterial effects and sustained anticancer effects.
14 mittent calorie restriction (ICR) have shown anticancer effects.
15 stituent (6a and 6b) exhibited the strongest anticancer effects.
16 r more than 50 years, has been shown to have anticancer effects.
17 ortant roles in metformin's antidiabetic and anticancer effects.
18 n of phenothiazines as a class of drugs with anticancer effects.
19 ol), involve pro-apoptotic ceramide in their anticancer effects.
20 bitors Physcion and its derivative S3, shows anticancer effects.
21 ly bioactive product that exhibits potential anticancer effects.
22 ulatory effects that may contribute to their anticancer effects.
23 e nanoparticle intratumoral distribution and anticancer effects.
24 ulation of regulatory T cells, and preserved anticancer effects.
25 an be targeted by small molecules to achieve anticancer effects.
26  molecular mechanisms by which it exerts its anticancer effects.
27 ng antibodies (nAbs) that can diminish their anticancer effects.
28 of drug cocktails while boosting synergistic anticancer effects.
29 own to bind G-quadruplex structures, showing anticancer effects.
30 suggesting a possible association with their anticancer effect; (2) multiple HMG boxes contributed ad
31  administered in animal models to achieve an anticancer effect and by the even higher doses required
32 ates a new mechanism for the emodin-mediated anticancer effect and justifies further investigation of
33                                 We report an anticancer effect and mechanism of GL against human pros
34 iofrequency (RF) ablation has shown superior anticancer effects and greater survival benefit with res
35 rget for cancer therapy, both through direct anticancer effects and immunomodulatory actions.
36                                 However, its anticancer effects and mechanisms on prostate cancer hav
37 ynchronized delivery system elicits enhanced anticancer effects and merits further development in the
38           A physiologic connection for these anticancer effects and overexpression is difficult to re
39 tea, has been shown, for example, to possess anticancer effects, anti-HIV effects, neuroprotective ef
40                     The in vitro and in vivo anticancer effects are also discussed.
41 ancerous effects are unmasked only after its anticancer effects are blocked.
42  likely to be an important mechanism for its anticancer effects because it protects cutaneous APC fro
43         Our results show that clioquinol has anticancer effects both in vitro and in vivo.
44 cing DNA hypomethylation may have short-term anticancer effects, but might also help speed tumor prog
45 We hypothesize that EGCG exerts anti-AhR and anticancer effects by acting as an hsp90 inhibitor.
46 tat, a histone deacetylase inhibitor, exerts anticancer effects by both histone and nonhistone-mediat
47  metabolism, we hypothesized that BaP exerts anticancer effects by disrupting lipogenesis.
48 drugs (NSAIDs) are believed to mediate their anticancer effects by inducing apoptosis but the molecul
49                 Therefore, sirtuins may have anticancer effects by shifting metabolism to a less prol
50  illustrate how Hsp70 inhibitors mediate the anticancer effects by targeting both tumor cells and tum
51 al alkaloid, was identified to exhibit novel anticancer effects by targeting ROS signaling.
52 this novel flavone has anti-inflammatory and anticancer effects by targeting the IKK complex.
53  of HDACI and the ganciclovir (GCV)-mediated anticancer effect contributed by HDACI-induced and p21-d
54 tached to carbon-20, was synthesized and the anticancer effects evaluated.
55 nese herbal medicine, is reported to exhibit anticancer effects; however, its mechanism of action is
56 n of cyclooxygenase (COX) with NSAIDs has an anticancer effect in animal models of colon, urinary bla
57 has not consistently demonstrated an in vivo anticancer effect in animal models.
58  the first dual binder 3 possessing a higher anticancer effect in GBM cells than the standards PK1119
59 he evidence suggests that ICR exerts greater anticancer effect in genetically engineered mouse models
60 emonstrate that RARbeta may exert its potent anticancer effect in part through its unique anti-AP-1 a
61 taxel or vinorelbine exhibited a synergistic anticancer effect in these human breast cancer cells in
62 potent SIRT2-specific inhibitor with a broad anticancer effect in various human cancer cells and mous
63                         CB1 exerted a potent anticancer effect in vitro in U-251 MG, U-373 MG, and D-
64  a novel mechanism for isosilybin B-mediated anticancer effects in human PCA cells.
65 n nanoparticle intratumoral distribution and anticancer effects in lung cancer.
66   How DNA methylation inhibitors exert their anticancer effects in patients is not well understood.
67 ase, and zoledronic acid has shown potential anticancer effects in preclinical and clinical studies.
68 e micellar nanocomplexes, which have greater anticancer effects in vitro and in vivo than the free pr
69 nger protein PARP-1 indicate that they exert anticancer effects in vitro based on different mechanism
70  developed nano-assembly showed the enhanced anticancer effects in vivo as well as in vitro.
71                    Furthermore, HMBA exerted anticancer effects in vivo in mouse models of Myc-driven
72 RNAi targeting FAS gene demonstrate systemic anticancer effects in vivo, our results render FAS as a
73 roposed mechanism by which PS-341 exerts its anticancer effect is inactivation of nuclear factor-kapp
74 emopreventive agent but the mechanism of its anticancer effect is not fully understood.
75 wever, the mechanism by which RME exerts its anticancer effect is presently unclear.
76 he molecular mechanism responsible for their anticancer effect is yet to be fully understood.
77 inhibitor, but the biochemical basis for its anticancer effects is not clear.
78  anticancer properties; the mechanism of its anticancer effects is unknown.
79 oral administration, we propose that maximum anticancer effect may be achieved by nanoemulsion mediat
80  inhibition of cyclooxygenase-1, whereas its anticancer effects may be due to inhibition of cyclooxyg
81                              The therapeutic anticancer effect of (177)Lu-cm09 (20 MBq) applied as a
82                                              Anticancer effect of aspirin represents one of the 'Prov
83                        1856) investigate the anticancer effect of bafilomycin, an inhibitor of the va
84     We investigated the in vitro and in vivo anticancer effect of combining lysosomal membrane permea
85                We have reported recently the anticancer effect of flavonoid antioxidant silymarin, th
86 em that has the potential for evaluating the anticancer effect of HDACIs on cancer cells by multiple
87 d with NDI, thus further indicating that the anticancer effect of NDI/2DG combination was indeed due
88 on in various cancer cell lines enhanced the anticancer effect of rapamycin.
89                   Our data demonstrated that anticancer effect of RAR-beta may be related to its abil
90                                          The anticancer effect of RARalpha, together with the newly d
91 f retinoid receptor activity may mediate the anticancer effect of RME.
92 s an excellent reagent for investigating the anticancer effect of selenium in vitro.
93  role of ER stress response in mediating the anticancer effect of selenium.
94 d and translate our observations on in vitro anticancer effect of silibinin/silymarin to an in vivo p
95              These results indicate that the anticancer effect of statins is independent from prenyla
96  to investigate the chemical composition and anticancer effect of the leaf essential oil of Xylopia f
97           Clinical updates indicate that the anticancer effect of these drugs is limited, perhaps due
98                                          The anticancer effect of TM correlates with its ability to d
99       However, the mechanisms underlying the anticancer effect of WA are not fully understood.
100 g an essential role for VDR in mediating the anticancer effects of 1,25-VD.
101                               Therefore, the anticancer effects of 4-MU, an orally bioavailable and r
102 sor genes may explain, at least in part, the anticancer effects of activation of PKG.
103 e used as a novel strategy to potentiate the anticancer effects of adoptively infused NK cells in pat
104 ivity makes PEDF a likely contributor to the anticancer effects of androgen ablation.
105                     These findings show that anticancer effects of ascorbate have been significantly
106 s production may provide a mechanism for the anticancer effects of aspirin.
107                                          The anticancer effects of bisphosphonates, cyclooxygenase-2
108 is, combined with the recent findings of the anticancer effects of bisphosphonates, cyclooxygenase-2
109 onal chemotherapies in order to optimize the anticancer effects of both strategies.
110 ossible mechanisms of the antiangiogenic and anticancer effects of celecoxib.
111 has emerged as a putative contributor to the anticancer effects of chemotherapeutic agents.
112 ivity of EET analogs does not compromise the anticancer effects of cisplatin.
113                                  Synergistic anticancer effects of combined bortezomib and doxorubici
114 ochemical studies indicating that the potent anticancer effects of CR and disrupted insulin/IGFI rece
115 altering this pathway may partly explain the anticancer effects of curcumin.
116                                          The anticancer effects of daidzein and genistein, and their
117                                The selective anticancer effects of DFMO on mouse and human MYCN-ampli
118      This novel effect may contribute to the anticancer effects of DIM because IFNgamma plays an impo
119 t may provide important clues to explain the anticancer effects of DIM because it is well known that
120 uate the molecular mechanisms underlying the anticancer effects of EF24 on CCA tumor growth and metas
121   Here we define the molecular basis for the anticancer effects of EPA.
122 f evidence supports the in vitro and in vivo anticancer effects of genistein, a soybean isoflavone.
123 , some of the reported anti-inflammatory and anticancer effects of green tea.
124         Together, these results suggest that anticancer effects of GSE in PCA be mediated via impairm
125 N-gammaR were particularly important for the anticancer effects of HDACi, and vorinostat and IFN-gamm
126 biology and (4) pharmacological data showing anticancer effects of HIF-1 inhibitors in mouse models o
127                  In addition, given that the anticancer effects of Hsp70 vaccines have been shown to
128 ile the molecular events associated with the anticancer effects of JS-K, HL-60 leukemia cells were tr
129                                          The anticancer effects of lead compound 4-methyl-5-(2-(4-mor
130 TR is an important upstream modulator of the anticancer effects of NSAIDs and that ibuprofen inductio
131 chanisms have been postulated to explain the anticancer effects of NSAIDs, they do not involve merely
132  study, we explored if paricalcitol enhanced anticancer effects of other clinically useful drugs in v
133 r, the molecular mechanisms underlying these anticancer effects of p27 are largely unknown.
134                         It also enhances the anticancer effects of platinum compounds and taxanes in
135 r a preclinical rationale to investigate the anticancer effects of PTP1B inhibitors currently being s
136                    Strategies to enhance the anticancer effects of RA could deepen and broaden its be
137                                          The anticancer effects of red wine have attracted considerab
138                                          The anticancer effects of retinoids are mainly mediated by t
139 its physical interaction with AP-1, promotes anticancer effects of retinoids by potentiating their an
140 beta) plays a critical role in mediating the anticancer effects of retinoids.
141 t RARbeta plays a critical role in mediating anticancer effects of retinoids.
142           In this study, we investigated the anticancer effects of RT53.
143                                 Although the anticancer effects of selenium have been shown in clinic
144 nd is an excellent compound for studying the anticancer effects of selenium in vitro.
145                                These in vivo anticancer effects of silibinin were associated with an
146 contributes to oncogenesis and underlies the anticancer effects of silvestrol and related compounds.
147 al and epidemiological evidence supports the anticancer effects of statins.
148                             Importantly, the anticancer effects of the drugs are ablated if CLU expre
149 ct immune system was required for the robust anticancer effects of the HDACi vorinostat and panobinos
150                         The inflammatory and anticancer effects of the NBPs might be associated with
151 se two mechanisms has been implicated in the anticancer effects of the respective drug.
152  well as a direct comparison of the in vitro anticancer effects of the two clinically available COX-2
153 dle formation, likely explaining some of the anticancer effects of these drugs.
154 Ks, and they may play a critical role in the anticancer effects of this and related compounds.
155 may represent a significant mechanism in the anticancer effects of this drug.
156 aditional Chinese medicine, can modulate the anticancer effects of TRAIL, the cytokine that is curren
157 nduction and resulting in enhancement of the anticancer effects of TRAIL.
158                                  Comparative anticancer effects of TRES and RES on pancreatic cancer
159                                 However, the anticancer effects of triacetylresveratrol (TRES, an ace
160 erpene from tropical ginger, can enhance the anticancer effects of tumor necrosis factor-related apop
161 ination resulted in a remarkable synergistic anticancer effect on intracranial human and murine gliob
162                    However, whether EF24 has anticancer effects on cholangiocarcinoma (CCA) cells and
163 inhibit ATP1A1 function, exhibited selective anticancer effects on STK11 mutant lung cancer cell line
164  (IPA) suggested that curcumin may exert its anticancer effects over multiple critical biological pat
165 ity, effective tumour targeting and superior anticancer effects owing to favourable doxorubicin-bindi
166            In cell culture, MTAs exert their anticancer effects primarily by causing mitotic arrest a
167       However, how RARbeta exerts its potent anticancer effects remains largely unknown.
168 xpression in HPV-positive HNSCC has a global anticancer effect resulting in a decrease in cell prolif
169 c-Myc/miR-29c/REV3L signalling may have dual anticancer effects, sensitizing the resistant tumours to
170 ta suggest that TLS inhibition may have dual anticancer effects, sensitizing tumors to therapy as wel
171 erior in their selective apoptosis-mediating anticancer effect than free form of these proteins and 5
172 ogenic effect, and a significant "bystander" anticancer effect that leads to enhanced production of t
173 Pterostilbene (PT) possesses antioxidant and anticancer effects that are mediated by ER-beta.
174  inhibiting telomerase may not result in the anticancer effects that were originally hypothesized.
175                  In spite of these promising anticancer effects, the molecular mechanism(s) of FF-ind
176 d transferrin receptor and exhibit potential anticancer effects through a signaling mechanism that is
177 e, bortezomib) are unlikely to mediate their anticancer effects through suppression of NF-kappaB.
178 ic acid (trans-RA) and other retinoids exert anticancer effects through two types of retinoid recepto
179 ng the first molecular target with potential anticancer effect, translating into the development of t
180 ic ultrasound pulses as well as the improved anticancer effects under hypoxic conditions.
181 d long-term use of NSAIDs is required for an anticancer effect--up to 15 or 20 years before a reduced
182    To correlate the AT1 receptor blockage to anticancer effects, VEGF levels and microvessel densitie
183 AR gamma agonists coordinately mediate their anticancer effect via both COX-dependent (inhibition of
184 xerts its highly potent in vitro and in vivo anticancer effects via tubulin-based antimitotic mechani
185 atural product previously unreported to have anticancer effects, was found to have potent antimyeloma
186             To improve TS inhibition and the anticancer effect, we have developed 35 peptides by modi
187 ent chloroquine also displayed a synergistic anticancer effect with 2DG, whereas glucose deprivation
188 ch yields a remarkable H2 O2 -NO cooperative anticancer effect with minimal adverse effect.
189 lioma therapy and honokiol shows synergistic anticancer effects with doxorubicin.
190              This will allow maximization of anticancer effects without induction of unacceptable tox

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