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1 Ketamine is a psychotomimetic and antidepressant drug.
2 ar target for the development of fast-acting antidepressant drugs.
3 attempt after starting treatment with newer antidepressant drugs.
4 ding increased risk of suicide with 10 newer antidepressant drugs.
5 factors mediating the behavioral effects of antidepressant drugs.
6 ay be important for the clinical efficacy of antidepressant drugs.
7 action of norepinephrine reuptake inhibitor antidepressant drugs.
8 effects of norepinephrine reuptake inhibitor antidepressant drugs.
9 release, suggesting a mechanism of action of antidepressant drugs.
10 of inescapable stress, and is used to screen antidepressant drugs.
11 rs, aniline derivatives, and basic tricyclic antidepressant drugs.
12 hanism of action of chronically administered antidepressant drugs.
13 processes after chronic treatment with these antidepressant drugs.
14 vity, which may underlie the actions of many antidepressant drugs.
15 ng-term therapeutic effect of anxiolytic and antidepressant drugs.
16 agents, including amphetamines, cocaine, and antidepressant drugs.
17 systems in the manner seen with established antidepressant drugs.
18 isplay high affinities for antipsychotic and antidepressant drugs.
19 naltered in depression, but is influenced by antidepressant drugs.
20 of the neurotransmitter systems targeted by antidepressant drugs.
21 support and counselling and to therapy with antidepressant drugs.
22 e primary target for the action of selective antidepressant drugs.
23 ffects on animal behavior tests sensitive to antidepressant drugs.
24 y than a wide spectrum of currently marketed antidepressant drugs.
25 argets for the development of rapidly acting antidepressant drugs.
26 ment of depression and on the development of antidepressant drugs.
27 transporter (SERT) is the primary target for antidepressant drugs.
28 elective serotonin reuptake inhibitor (SSRI) antidepressant drugs.
29 improvement of depression severity with two antidepressant drugs.
30 tion, a population likely to be resistant to antidepressant drugs.
31 r antagonists may be potential anxiolytic or antidepressant drugs.
32 for optimal binding of fluoxetine and other antidepressant drugs.
33 r antagonists may be potential anxiolytic or antidepressant drugs.
34 currently being developed as antiobesity and antidepressant drugs.
35 ctivity, and behavioral responses to chronic antidepressant drugs.
36 RF 1 antagonists could be used clinically as antidepressant drugs.
37 ive behaviors, but does hamper the effect of antidepressant drugs.
38 RI prescriptions and prescriptions for other antidepressant drugs.
39 paved the way for the development of modern antidepressant drugs.
40 IMCP increased clinician recommendations for antidepressant drugs, a mental health referral, or both
41 se of depression and that current first-line antidepressant drugs act by restoring excitatory synapti
42 el rodent assay to investigate how different antidepressant drugs act to modify affective biases that
45 urely neurotransmitter-based explanation for antidepressant drug action is challenged by the delayed
49 , we asked whether chronic treatment with an antidepressant drug (AD) that modifies serotonergic func
51 both groups, during the 20-week experiment, antidepressant drug administration was tapered and disco
52 ese data support the hypothesis that chronic antidepressant drug administration, through regulation o
53 ent study demonstrates that the mixed action antidepressant drug amitriptyline enhances norepinephrin
55 nt-reported clinician recommendations for an antidepressant drug among nondepressed patients could no
56 l insight into both the pharmacology of this antidepressant drug and the targeting of the alpha(2A)AR
57 rter (NET) is a site of action for tricyclic antidepressant drugs and for drugs of abuse such as amph
58 site of common transcriptional regulation by antidepressant drugs and in both reversing susceptibilit
59 n region-specific actions of a wide range of antidepressant drugs and indicate that pharmacological i
61 ides strong support for the notion that both antidepressant drugs and psychological therapies modify
63 atments might help make best use of existing antidepressant drugs and reduce the number of treatment-
64 The suicides were divided into those free of antidepressant drugs and those in whom prescription of a
65 on-deficit/hyperactivity disorder drugs, and antidepressant drugs) and for 9 of the 19 AMCs that impl
66 ention-deficit/hyperactivity disorder drugs, antidepressant drugs, and antipsychotic drugs) comparing
67 T is the major binding site in the brain for antidepressant drugs, and it also binds amphetamines and
68 ndings that the analgesic effects of various antidepressant drugs are differentially dependent on the
69 The classical targets for antipsychotic and antidepressant drugs are G protein-coupled receptors and
70 ay contribute to the therapeutic efficacy of antidepressant drugs are likely to occur over distances
72 However, no published evidence exists that antidepressant drugs are safe or efficacious in patients
75 n be reversed by treatment with conventional antidepressants drugs, as well as by subanesthetic doses
76 th week of gestation nor the use of non-SSRI antidepressant drugs at any time during pregnancy was as
77 lecular mechanisms by which various types of antidepressant drugs bind and inhibit SERT and NET are s
78 ment of depression, the molecular details of antidepressant drug binding are still not fully understo
79 athway has been implicated in the actions of antidepressant drugs, but studies to date have not demon
80 (SSRIs) are the most commonly used class of antidepressant drugs, but the cellular and molecular mec
81 ST), the most used assay to screen potential antidepressant drugs by decreasing immobility behavior.
82 ained lay health counsellor, supplemented by antidepressant drugs by the primary care physician and s
84 these disorders, but it is not known whether antidepressant drugs can directly modulate the neural pr
87 ificant effect of CBT during continuation of antidepressant drugs compared with antidepressants alone
88 on, but reduced availability in those taking antidepressant drugs, consistent with a possible mode of
90 The effects of chronic administration of the antidepressant drugs desipramine, nortryptiline and paro
97 monstration of genetic variation influencing antidepressant drug effects on emotional processing in h
99 and Western blot analysis revealed that all antidepressant drugs elicited an anatomically specific i
100 after 8 weeks of treatment with one of three antidepressant drugs (escitalopram, sertraline, or venla
101 recent advances in our understanding of how antidepressant drugs exert their antinociceptive effects
103 nd selectivity over NET for the prototypical antidepressant drug fluoxetine (Prozac; Eli Lilly, India
105 e subdivided into those who had been free of antidepressant drugs for at least 3 months and those in
106 e subdivided into those who had been free of antidepressant drugs for at least three months, and thos
107 860 outpatients were screened, yielding 199 antidepressant drug-free patients with unipolar nonpsych
108 ; p<0.002), and the three patients taking an antidepressant drug had globally reduced [(18)F]GE-179 V
110 es of the therapeutic mechanism of action of antidepressant drugs have focused on the role of the mon
114 fluoxetine is the most frequently prescribed antidepressant drug in the United States, its safety in
115 ract risk after exposure to SSRI or to other antidepressant drugs in a large electronic primary care
116 mice mimicked the effects of anxiolytic and antidepressant drugs in a number of behavioral tests, wi
121 nty and controversy remains about the use of antidepressant drugs in the management of depressive epi
122 dysfunction, suggesting a potential for such antidepressant drugs in the treatment of presymptomatic
123 Importantly, the analgesic effect of several antidepressant drugs, including selective serotonin reup
124 c-like actions of several monoamine-directed antidepressant drugs, including tricyclic antidepressant
126 fic comorbid conditions and sustained use of antidepressant drugs may be associated with a medical-of
127 Although an infrequent event, DILI from antidepressant drugs may be irreversible, and clinicians
128 s unclear how the 5-HT signalling effects of antidepressant drugs might alter neuropsychological mech
129 on and subsequently develop more efficacious antidepressant drugs, multiple theories have been propos
130 second medication to an initial, ineffective antidepressant drug, no randomized controlled trial has
132 the previously shown biphasic action by the antidepressant drugs on total BDNF expression is explain
133 ssive phenotypes, which can be alleviated by antidepressant drugs or by overexpression of TrkB in the
134 between behavioral modifications induced by antidepressant drugs or environmental enrichment and cha
135 n or elimination of REM sleep in subjects on antidepressant drugs or with brainstem lesions produces
136 imulation, exercise, dietary restriction and antidepressant drugs preserve brain function during agin
137 uoxetine (FLX), the active ingredient of the antidepressant drug Prozac, inhibits reuptake of the neu
138 ecent surprising findings have revealed that antidepressant drugs reactivate a window of juvenile-lik
139 ssessment of clinician- and patient-reported antidepressant drug recommendation (primary outcomes) wi
140 of the composite measure of patient-reported antidepressant drug recommendation, mental health referr
142 d in studies of antipsychotic drug efficacy, antidepressant drug response, and drug-induced adverse e
144 e reuptake inhibitor, is a widely prescribed antidepressant drug routinely detected in the aquatic en
145 breastfeeding is not necessary, because most antidepressant drugs seem not to affect the infant.
146 istration of paroxetine, a widely prescribed antidepressant drug that acts by inhibiting reuptake of
147 ne, and abolishes the effects of widely used antidepressant drugs that act as catecholamine reuptake
148 nile rats acutely and chronically exposed to antidepressant drugs that act on serotonin and norepinep
149 are implicated in psychiatric disorders, and antidepressant drugs that block the NE transporter (NET)
150 promise as lead compounds in the search for antidepressant drugs that release serotonin rather than
152 ther these behaviours are sensitive to human antidepressant drugs; the striped shore crab, Pachygraps
154 l crying, which has been shown to respond to antidepressant drug therapy, the other post-stroke emoti
156 ide, and because long-term administration of antidepressant drugs to rats down-regulates these protei
161 icidal ideation and behavior associated with antidepressant drug treatment in children and adolescent
163 that combined psychological intervention and antidepressant drug treatment may not be more effective
164 s support careful clinical monitoring during antidepressant drug treatment of severely depressed youn
165 affects transcription and may be involved in antidepressant drug treatment outcome, although response
168 children and adolescents (aged 6-18 years), antidepressant drug treatment was significantly associat
176 e associated with the mechanism of these two antidepressant drug treatments and may contribute to the
177 or dependence comorbidity, suicide attempts, antidepressant drug use, and having a relative with rapi
179 ynchrony system in yeast with phenelzine, an antidepressant drug used in the treatment of affective d
182 ssion who had been successfully treated with antidepressant drugs were randomly assigned to either CB
183 ar follow-up was undertaken, during which no antidepressant drugs were used unless a relapse ensued.
184 r future structure-based drug development of antidepressant drugs with fine-tuned transporter selecti
185 itive transporters, as novel targets for new antidepressant drugs with improved therapeutic potential
186 mal elderly participants who were exposed to antidepressant drugs within the past 5 y to participants
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