戻る
「早戻しボタン」を押すと検索画面に戻ります。

今後説明を表示しない

[OK]

コーパス検索結果 (1語後でソート)

通し番号をクリックするとPubMedの該当ページを表示します
1 tagonists, kainate lowered the threshold for antidromic action potential generation, suggesting that
2           The dorsal root reflex (DRR) is an antidromic action potential originating in the spinal co
3 ould be recorded at the granule cell soma as antidromic action potentials and from the axons with a n
4                                     However, antidromic action potentials are recruited at lower thre
5 ilaments were determined either by recording antidromic action potentials from the tooth or by using
6 Kainate application also induced spontaneous antidromic action potentials.
7 on was demonstrated by somatic recordings of antidromic action potentials.
8                                     However, antidromic activation (mossy fiber stimulation) evoked a
9    Recorded neurons were classified by their antidromic activation and by their changes in firing rat
10             Neurons were identified by their antidromic activation from contralateral facial and acce
11             Neurons were identified by their antidromic activation from facial nucleus (FN) or red nu
12  latencies to trigeminal ganglion shocks and antidromic activation from thalamus or cerebellum were a
13                     Neurones were tested for antidromic activation from the contra- and ipsilateral v
14                  Neurones were identified by antidromic activation from the contralateral thalamus, a
15   The rmPFC neurons were identified by their antidromic activation from the mediodorsal nucleus and/o
16 ially by driving recurrent inhibition though antidromic activation of corticostriatal axon collateral
17 ations in the orbitofrontal cortex (OFC) via antidromic activation of corticostriatal recurrent inhib
18                                 Furthermore, antidromic activation of electrical activity in the cell
19                                              Antidromic activation of ganglion cell axons also increa
20                   Previous studies show that antidromic activation of sensory fibers is an important
21 s mediated by peripheral release of CGRP via antidromic activation of sensory fibers.
22 lation through sympathetic inhibition and/or antidromic activation of sensory fibers.
23 sodilation at >or=90% of MT may also involve antidromic activation of some unmyelinated C-fibers.
24  cartwheel cells were recorded to ortho- and antidromic activation of the granule cells (i.e., by sti
25 r were further identified as mitral cells by antidromic activation of the lateral olfactory tract and
26 e if SCS produces cutaneous vasodilation via antidromic activation of the unmyelinated C-fibers and/o
27 ere located and identified by the electrical antidromic activation of their constituent motoneurons.
28               Our previous studies show that antidromic activation of transient receptor potential va
29      Retrograde and anterograde labeling and antidromic activation of vBNST neurons by VTA stimulatio
30                                           In antidromic activation studies, mu-opioids inhibited a su
31 ic tract (STT) neurons were identified using antidromic activation techniques and examined for their
32  this projection in rats using the method of antidromic activation to map the axon terminals of neuro
33 entified as NTS or DMN using orthodromic and antidromic activation, respectively, following vagal sti
34 injured neuronal circuits is the same during antidromic activation, stimulation of granule cell axons
35 urons were identified as septohippocampal by antidromic activation.
36  reflex (DRR) and the axonal reflex (AR) are antidromic activities in primary afferents and are invol
37 striatal stimulus current necessary to evoke antidromic activity.
38 linked micropipette-microwire recording, and antidromic and orthodromic activation from the ventral t
39 ey rats demonstrated reciprocal interinsular antidromic and orthodromic activation, elicited with sim
40 ypoxia and 1 hour wash-out of the inhibitors antidromic and orthodromic responses were still blocked
41                                              Antidromic block can cause acceleration due to double-wa
42 riod, and was associated with unidirectional antidromic block of the paced impulse.
43                                              Antidromic CAPs of C-fibers in dorsal roots were evoked
44                               In Protocol 3, antidromic CAPs of the dorsal root were measured in resp
45                                              Antidromic (centrifugal) conduction of these spikes may
46 h participate with substrate glutamine in an antidromic circular arrangement of hydrogen bonds, cause
47                               In Protocol 2, antidromic compound action potentials (CAPs) of the tibi
48 p junctions between AVA and A-MNs only allow antidromic current, but, curiously, disrupting them inhi
49 ger at shorter V1V2 intervals, but a shorter antidromic delay in the area of unidirectional block for
50                        Bursts of spontaneous antidromic dorsal root action potentials, and evoked dor
51  nerve to block both orthodromic signals and antidromic DRRs without affecting ARs.
52                                      We used antidromic electrical stimulation to identify V1 neurons
53                                 Finally, the antidromic ERG recordings obtained in our implanted volu
54 were recorded using 16 scalp electrodes, and antidromic ERGs were obtained using DTL electrodes while
55 ese responses could not be attributed to the antidromic firing of corticothalamic cells, intrathalami
56                      The possible effects of antidromic firing on synaptic strength are unknown.
57         Synaptic stimulation delivered after antidromic firing, which was otherwise too weak to induc
58 glutamate released from its own dendrites by antidromic impulse invasion, or/and lateral excitation b
59  this site was likely to produce block of an antidromic impulse, which may initiate double-wave reent
60       There was no significant difference in antidromic latencies between Type I (m = 1.47 msec) and
61                                     The mean antidromic latency from the L1 level was 42.8 +/- 4.4 ms
62 on site and distally through orthodromic and antidromic mechanisms for several stimulation frequencie
63 ng neuroendocrine GABA neurons identified by antidromic median eminence stimulation.
64                                              Antidromic (n=35), monosynaptic (n=2), di-or tri-synapti
65     Both during normal bursting activity and antidromic nerve stimulation, the conduction delay over
66                               A mechanism of antidromic passage of depolarizing current from a neuron
67                                              Antidromic population events in subiculum were single sp
68 for 35 min after trauma injury, improved CA1 antidromic population spike (PS) recovery to 91 +/- 2%,
69                                              Antidromic population spikes confirmed projections from
70                                              Antidromic potentials are larger if the afferent is clos
71 pagation of V2 around the line of block, and antidromic propagation through the original location of
72 ion of the lateral olfactory tract evoked an antidromic pulse followed by a short EPSP, which could a
73                                   The use of antidromic-rectifying gap junctions to amplify chemical
74 so released from sensory-motor nerves during antidromic reflex activity, to produce relaxation of som
75 flammation is believed to originate with the antidromic release of substance P, and of other neurokin
76                            The proportion of antidromic responses consisting of full spikes from anti
77 hose neurons projecting to MT, identified by antidromic responses to electrical stimulation of MT.
78                                   Reciprocal antidromic responses were absent.
79 h could also be elicited independently of an antidromic spike in the recorded cell.
80                                 Although the antidromic spike latency of the single-spiking and burst
81 m depression between synaptically evoked and antidromic spike trains emphasize that the properties of
82 s was compared quantitatively to that during antidromic spike trains evoked by electrical stimulation
83 naptic spike width that did not occur during antidromic spike trains under physiological calcium conc
84 kes in a burst could be made to collide with antidromic spike.
85 generated rhythmic barrages (up to 25 Hz) of antidromic spikes during BMPs.
86                During BMPs the soma received antidromic spikes generated in processes in the buccal g
87 , we used optogenetic stimulation to trigger antidromic spikes in a local region of primary visual co
88 lation of the medial forebrain bundle evoked antidromic spikes in both burst-firing neurones and in s
89 firing neurones and classical 5-HT neurones, antidromic spikes made collisions with spontaneously occ
90                 Plateau potentials after the antidromic spikes or local cerebral inputs will locally
91                        During the barrage of antidromic spikes, high-frequency firing will produce st
92 curred spontaneously (53%) or in response to antidromic stimulation (81%).
93 ses in Ca(2+) transients evoked by light and antidromic stimulation are blocked by the purinergic ant
94 r pharmacological gap junction blockade, but antidromic stimulation could not drive activity in contr
95  in a small number of burst-firing neurones, antidromic stimulation evoked spike doublets, similar to
96 ioid, GABA(A), and NK1 receptor antagonists, antidromic stimulation of a population of striatal proje
97                                              Antidromic stimulation of cardiac sensory C fibers relea
98 rneurons in the medial prefrontal cortex via antidromic stimulation of cortico-accumbal afferents.
99         Skin blood flow was monitored during antidromic stimulation of identified cutaneous C fibres
100 dromic stimulation of the olfactory nerve or antidromic stimulation of mitral and tufted (M/T) cells.
101                           Here, we show that antidromic stimulation of motor axons evokes depolarizin
102 ntial [total motor activity (TMA)] evoked by antidromic stimulation of the distal ventral root.
103  In bicuculline (10 microM) and 6 mM [K +]o, antidromic stimulation of the granule cells evoked burst
104 es on granule cells that can be activated by antidromic stimulation of the lateral olfactory tract (L
105 ith extracellular field potentials following antidromic stimulation of the lateral olfactory tract (L
106 ing bursts of action potentials generated by antidromic stimulation of the mossy fibers.
107                     They can be activated by antidromic stimulation of the trigeminal nerve, as well
108 furcating axon collaterals in the chicken by antidromic stimulation of two sites along each branch an
109 ctivation of sensory nerve fibers, either by antidromic stimulation or capsaicin, depolarized these n
110 ive green fluorescent protein expression and antidromic stimulation or retrograde Evans blue dye trac
111 ptic to local glutamatergic neurons, we used antidromic stimulation to reveal that many of these cell
112                            Furthermore, such antidromic stimulation was adequate to rescue "tagged" s
113 s activation of a pool of identified SPNs by antidromic stimulation.
114 eutic NAc-DBS might exert its effect through antidromic stimulation.
115 he contralateral thalamus were determined by antidromic stimulation.
116 +) activity were recorded in area CA3 during antidromic stimulation.
117 ic transmission are much more effective than antidromic stimuli that do not.
118                                           In antidromic studies, NA dose-dependently increased firing
119 e refractoriness followed by resetting of an antidromic tachycardia (AT) in patients with decremental
120 rats was significantly greater, and the mean antidromic threshold was significantly lower than in con
121 tion requires a sufficient excitable gap and antidromic unidirectional block of the paced impulse in
122 res were tested for their ability to produce antidromic vasodilatation.
123   These effects were potent: the area of the antidromic volley evoked in the sural nerve by intraspin

WebLSDに未収録の専門用語(用法)は "新規対訳" から投稿できます。
 
Page Top