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1 ing to the proposal that it is an endogenous antiepileptic agent.
2 confirm that norepinephrine (NE) is a potent antiepileptic agent.
3 for neuroprotection after stroke and use as antiepileptic agents.
4 m channel dependent and a synaptic dependent antiepileptic agents.
5 ntidepressants, atypical antipsychotics, and antiepileptic agents.
6 h is responsible for clearing the endogenous antiepileptic agent adenosine (Ado) from the extracellul
9 women with epilepsy who were taking a single antiepileptic agent (carbamazepine, lamotrigine, phenyto
10 g of live exposed cortex showed that several antiepileptic agents, including valproate, gabapentin an
12 ether the prophylactic administration of the antiepileptic agent phenytoin would reduce its occurrenc
13 the activities observed for the traditional antiepileptic agents phenytoin, phenobarbital, and valpr
14 (SAR) for the N-benzyl group in the clinical antiepileptic agent (R)-lacosamide [(R)-N-benzyl 2-aceta
15 ta are emerging regarding the use of several antiepileptic agents such as topiramate, disodium valpro
16 a small number of "high-risk" drugs such as antiepileptic agents, sulfonamides, and antiretroviral d
17 tem that is suspected of being an endogenous antiepileptic agent that can control propagation of limb
21 convulsant, unrelated to currently available antiepileptic agents, with activity in a broad range of
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