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1 se these drugs in patients already receiving antihypertensive therapy.
2 atient was discharged with anticoagulant and antihypertensive therapy.
3 equired further intensification of discharge antihypertensive therapy.
4 RDN emerged as an innovative interventional antihypertensive therapy.
5 or antagonist, or AngII infusion with triple-antihypertensive therapy.
6 mm Hg diastolic and treated with intravenous antihypertensive therapy.
7 bo required the initiation of or a change in antihypertensive therapy.
8 t is resistant to most forms of conventional antihypertensive therapy.
9 t the study, most were on antithrombotic and antihypertensive therapy.
10 the score components of diabetes and use of antihypertensive therapy.
11 ssociated with nonadherence to statin and/or antihypertensive therapy.
12 ossibility of targeting the EP1 receptor for antihypertensive therapy.
13 e provider in improving patient adherence to antihypertensive therapy.
14 ians than in African Americans regardless of antihypertensive therapy.
15 he physiologic evidence of renoprotection by antihypertensive therapy.
16 cal cardiovascular disease and not receiving antihypertensive therapy.
17 pine, or placebo in addition to conventional antihypertensive therapy.
18 sease and should be preferred for first-line antihypertensive therapy.
19 kers, should no longer be used as first-line antihypertensive therapy.
20 on who were under evaluation for a change in antihypertensive therapy.
21 disease and those receiving anticoagulant or antihypertensive therapy.
22 those adherent to statin, but nonadherent to antihypertensive, therapy.
23 rovide a potential target for individualized antihypertensive therapies.
24 wer risk of combined MI or stroke than other antihypertensive therapies.
25 59) for those nonadherent both to statin and antihypertensive therapy, 1.82 (95% CI: 1.43 to 2.33) fo
27 tions between 1953 and 1957 and were free of antihypertensive therapy and cardiovascular disease.
29 ians at all exercise workloads regardless of antihypertensive therapy and had over a 90% higher likel
30 and of all-cause mortality in the setting of antihypertensive therapy and regression of ECG left vent
31 ith statins and other lipid-lowering agents, antihypertensive therapies, and antihyperglycemic treatm
32 those non-adherent to statin but adherent to antihypertensive therapy, and 1.30 (95% CI: 0.53 to 3.20
33 ead time, reduced effectiveness of intensive antihypertensive therapy, and increased relative risk re
35 ramework for addressing patient adherence to antihypertensive therapy, and to propose future directio
36 ust, 2010, for randomised clinical trials of antihypertensive therapy (ARBs, angiotensin-converting-e
38 d ACEI/ARB and 24,001 (61.1%) received other antihypertensive therapy at year 1 after transplantation
39 d ACEI/ARB and 24,001 (61.1%) received other antihypertensive therapy at year 1 after transplantation
40 multivariable models adjusting for age, sex, antihypertensive therapy, body mass index, heart rate, t
41 nzyme (ACE) inhibitors are used primarily in antihypertensive therapy but also are known to improve w
43 terol, estimated glomerular filtration rate, antihypertensive therapy, diabetes mellitus, and smoking
44 sence of renal failure, and documentation of antihypertensive therapy, diabetic status, proteinuria s
45 further reducing the diastolic pressure with antihypertensive therapy, especially in patients with co
47 lyzed according to whether ACEI/ARB or other antihypertensive therapy (excluding diuretics) was admin
48 lyzed according to whether ACEI/ARB or other antihypertensive therapy (excluding diuretics) was admin
50 Large-scale trials to assess the value of antihypertensive therapy for older patients with SBP of
51 te, free of coronary heart disease (CHD) and antihypertensive therapy, from the Chicago Heart Associa
53 hough black patients received more intensive antihypertensive therapy, Hispanics were undertreated.
54 the pathophysiology, risks, and benefits of antihypertensive therapies in the patient with intracran
56 aptured year-by-year adherence to statin and antihypertensive therapy in both study groups and estima
57 e endothelial, cardiac, and renal effects of antihypertensive therapy in hypertension and may explain
58 hypotension; the effectiveness of nocturnal antihypertensive therapy in patients with coexistent neu
59 cal trials from 1965 through October 2010 of antihypertensive therapy in patients with type 2 diabete
63 aracteristics associated with an increase in antihypertensive therapy included increased levels of bo
64 nell product electrocardiographic LVH during antihypertensive therapy is associated with a lower like
65 nell product electrocardiographic LVH during antihypertensive therapy is associated with fewer hospit
66 uct and Sokolow-Lyon voltage criteria during antihypertensive therapy is associated with lower likeli
68 ough literature on reduction of LV mass with antihypertensive therapy is extensive, little informatio
70 re have been advances in management, such as antihypertensive therapy, magnesium sulphate, and fluid
72 blind candesartan or placebo with open-label antihypertensive therapy (mostly thiazide diuretics) add
75 f incident dementia; however, the effects of antihypertensive therapy on cognitive function in contro
77 combined outcome of MI and stroke than other antihypertensive therapies (OR, 0.49; 95% CI, 0.32-0.77)
78 diabetes mellitus, systolic blood pressure, antihypertensive therapy, prior coronary disease, and le
79 hest approved dosage of losartan and optimal antihypertensive therapy reduces albuminuria over 6 mo a
81 with hypertension who were not receiving any antihypertensive therapy (relative hazard, 0.91; 95 perc
83 baseline severity of ECG LVH, losartan-based antihypertensive therapy resulted in greater regression
85 sis was managed by withholding pre-apheresis antihypertensive therapy, saline prehydration, and reduc
90 ave less regression of CP LVH in response to antihypertensive therapy than patients without diabetes,
93 relative to those who adhered to statins and antihypertensive therapy, the odds ratio at the year of
94 or age, sex, systolic blood pressure, use of antihypertensive therapy, total and high-density lipopro
95 tics will rightfully remain a cornerstone in antihypertensive therapy, we should remember (as we were
99 derate evidence to support initial or add-on antihypertensive therapy with an angiotensin-converting
100 nsion (ACCOMPLISH) trial showed that initial antihypertensive therapy with benazepril plus amlodipine
101 HEP) trial, conducted between 1985 and 1990, antihypertensive therapy with chlorthalidone-based stepp
103 partitioning to assess the probability that antihypertensive therapy would be increased at a given c
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