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1 could predict the reversibility of CP after antiinflammatory therapy.
2 hronic inflammation and future directions in antiinflammatory therapy.
3 transmigration as a potential new target for antiinflammatory therapy.
4 ticosteroids are recognized as the preferred antiinflammatory therapy.
5 eed for trials of more focused and effective antiinflammatory therapy.
6 d the Pyk2 pathway as a potential target for antiinflammatory therapy.
7 re, we have focused on PECAM as a target for antiinflammatory therapy.
8 that responds poorly to currently available antiinflammatory therapy.
9 gression and may benefit from future adjunct antiinflammatory therapies.
10 tworks is providing a new way of identifying antiinflammatory therapies.
11 of RA as well as for preclinical testing of antiinflammatory therapies.
12 in connection with the development of novel antiinflammatory therapies.
13 S-1 pathway represents a possible target for antiinflammatory therapies.
15 further establish NF-kappaB as a target for antiinflammatory therapy and provide support for the use
22 hat tyloxapol is potentially useful as a new antiinflammatory therapy for CF lung disease, and could
24 sis factor-alpha (TNF-alpha) is an effective antiinflammatory therapy for several chronic inflammator
26 tion and suggest IL-1R as a novel target for antiinflammatory therapy in CF and potentially other muc
29 omy group); and lateral right atriotomy with antiinflammatory therapy (methylprednisolone 2 mg/kg per
30 mpt clinicians to consider intervention with antiinflammatory therapy, preferably inhaled corticoster
31 itis, colchicine, when added to conventional antiinflammatory therapy, significantly reduced the rate
32 transcription it may be possible to develop antiinflammatory therapies that spare the constitutive e
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