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1 is) and favors accumulation of surrogates of antimonials.
2 responsible for resistance to arsenicals and antimonials.
3 QP1 that prevents the transport of trivalent antimonials.
4                                              Antimonial agents are a mainstay for the treatment of le
5 roducts of this operon catalyze extrusion of antimonials and arsenicals from cells.
6                              The pentavalent antimonials are considered the first-choice drugs for tr
7                               Arsenicals and antimonials are first line drugs for the treatment of tr
8 h KD Sb(R)LD enhanced the sensitivity toward antimonials compared with infection with Sb(R)LD, and in
9      In Bihar state, India, the cure rate of antimonial compounds (eg, sodium stibogluconate) in the
10                                              Antimonial compounds are used as first-line treatment dr
11 tor R773 confers resistance to arsenical and antimonial compounds in Escherichia coli, where resistan
12 trains with different inherent resistance to antimonial drugs (antimony sensitive strain Sb-S; and an
13       Children have a lower response rate to antimonial drugs and higher elimination rate of antimony
14                           Resistance against antimonial drugs has probably been a contributing factor
15 nonhealing forms of human leishmaniasis with antimonial drugs in combination with gamma interferon (I
16 ptake systems, but the uptake system for the antimonial drugs in Leishmania is unknown.
17 e in Bihar because inadequate treatment with antimonial drugs is not exclusive to India, whereas wide
18 water coexists with widespread resistance to antimonial drugs that are used to treat the parasitic di
19 ttributed to long-term, widespread misuse of antimonial drugs within the Indian private health-care s
20 atients produces a common mode of action for antimonial drugs, which define a novel compound class th
21  choice for the treatment of this disease is antimonial drugs.
22 ain the development of PKDL: (i) the role of antimonial drugs; (ii) UV-induced skin damage; (iii) rei
23       Leishmania resistant to arsenicals and antimonials extrude arsenite.
24  distinct population frequently resistant to antimonials has a two base-pair insertion in the aquagly
25 -dependent extrusion pump for arsenicals and antimonials in Escherichia coli, is allosterically activ
26 responsible for resistance to arsenicals and antimonials in Escherichia coli.
27 on that confers resistance to arsenicals and antimonials in Escherichia coli.
28 ted in deglutathionylation and activation of antimonial prodrugs used to treat leishmaniasis.
29 ficant role in the development of Leishmania antimonial resistance in Bihar because inadequate treatm
30 might have contributed to the development of antimonial resistance in Leishmania parasites in Bihar.
31 coli plasmid R773 that confers arsenical and antimonial resistance is negatively regulated by the Ars
32 s not exclusive to India, whereas widespread antimonial resistance is.
33  cure by chemotherapy (primarily pentavalent antimonials [Sb(V)]).
34   The current standard to assess pentavalent antimonial (SSG) susceptibility of Leishmania is a labor
35 he associations between arsenic exposure and antimonial treatment failure and death in the leishmania
36 ia strains are frequently cross-resistant to antimonials, we considered the possibility that Sb(V) is
37 nfer high level resistance to arsenicals and antimonials, while the chromosomally encoded ars operon

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