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1 making the tumor cells further resistant to antineoplastic drugs.
2 ity and alter the pharmacokinetic profile of antineoplastic drugs.
3 ffects of reactive oxygen species-generating antineoplastic drugs.
4 lective, small-molecule kinase inhibitors as antineoplastic drugs.
5 ng strategies as a means of developing novel antineoplastic drugs.
6 c target for a number of clinically relevant antineoplastic drugs.
7 clinical studies of anti-HER2 SL loaded with antineoplastic drugs.
8 so induced partial cross-resistance to other antineoplastic drugs.
12 pression also modulates the effects of other antineoplastic drugs and whether it is associated with a
13 ay be most useful when "metronomic" doses of antineoplastic drugs are used, thereby potentially avoid
14 tudy, we monitored the cellular responses to antineoplastic drug at a single cell basis with Raman sp
17 nt studies have shown that some conventional antineoplastic drugs can be exploited for antiangiogenic
18 tential to augment preclinical evaluation of antineoplastic drug candidates for these malignancies.
20 effectively treat gliomas by reaching a high antineoplastic drug concentration at the target site wit
21 ationale can be proposed for intraperitoneal antineoplastic drug delivery in non-ovarian malignant di
22 l modulation of BTB permeability to increase antineoplastic drug delivery selectively to brain tumors
23 ransferases (GSTs) in cellular resistance to antineoplastic drugs, derivatives of MCF7 breast carcino
29 We have investigated the ability of another antineoplastic drug, dolastatin 10, in inducing Bcl2 pho
31 The therapeutic efficacy of the widely used antineoplastic drugs doxorubicin, cisplatin, vincristine
35 en paracrine IL-2 and local i.c. delivery of antineoplastic drugs is novel and may provide a combined
36 side effect associated with cancer-treating antineoplastic drugs is the development of neuropathic p
40 antisense ODNs in combination with standard antineoplastic drugs might be useful in reversing MDR in
41 rom the National Cancer Institute's In Vitro Antineoplastic Drug Screen to assess whether sensitivity
42 tructurally disparate DNA cleavage-enhancing antineoplastic drugs share an overlapping site of action
46 erythroleukemia K562 cells are resistant to antineoplastic drug (taxol)-induced apoptosis through th
47 n over the past decade, yet most preclinical antineoplastic drug testing is still reliant on conventi
48 Microtubule-damaging agents (MDA) are potent antineoplastic drugs that are widely used in clinical tr
50 witch is a rational target for the design of antineoplastic drugs that selectively inhibit PKCepsilon
51 at, when FTI is combined with some cytotoxic antineoplastic drugs, the effects on tumor cells are add
52 and the most commonly fatal complication of antineoplastic drug therapy and may represent a serious
53 he nucleoside analogue floxuridine, a potent antineoplastic drug used in the clinic to treat advanced
54 Arabinofuranosylcytosine (Ara-C) is a potent antineoplastic drug used in the treatment of acute leuke
57 ristol-Myers Squibb, Princeton, NJ) is a new antineoplastic drug with broad-spectrum activity in soli
58 trophy) as well as in chemotheraputic use of antineoplastic drugs with cardiotoxic side effects (i.e.
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