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1 onger correlates of lung function than other antioxidant vitamins.
2 All of these changes were prevented by antioxidant vitamins.
3 ive role for supplemental vitamin E or other antioxidant vitamins.
4 ions of lipid hydroperoxides and liposoluble antioxidant vitamins.
5 ingredients - polyphenols, carotenoids, and antioxidant vitamins.
6 PANTS: Within an ongoing randomized trial of antioxidant vitamins, 5442 women who were US health prof
9 ients, especially folate, calcium, iron, and antioxidant vitamins, affect the risk of colorectal neop
10 mg pyridoxine, and 0.02 mg cyanocobalamin), antioxidant vitamins alone (150 mg ascorbic acid, 67 mg
12 as to determine the plasma concentrations of antioxidant vitamins and carotenoids in hemodialysis pat
15 dation is maintained for longer than that of antioxidant vitamins and could thus be more protective.
16 cant trends between plasma concentrations of antioxidant vitamins and either measure of carotid ather
17 lind, randomized trial, Supplementation with Antioxidant Vitamins and Minerals (SU.VI.MAX) Study (199
18 f participants from the Supplementation with Antioxidant Vitamins and Minerals (SU.VI.MAX) Study usin
19 patic iron may promote liver injury, whereas antioxidant vitamins and minerals may inhibit it, but fe
20 eficial effects of oral supplementation with antioxidant vitamins and minerals on the development of
22 nt effects on plasma concentrations of other antioxidant vitamins and minerals, glutathione peroxidas
25 tic components, and higher amounts of fibre, antioxidants, vitamins and other beneficial components.
28 tudy suggest a moderate beneficial effect of antioxidant, vitamin, and zinc supplementation in reduci
30 Accumulating evidence suggests that dietary antioxidant vitamins are positively associated with lung
33 of mortality in the general population, and antioxidant vitamins are thought to positively influence
35 re simultaneously assigned to receive either antioxidant vitamins (beta-carotene, ascorbic acid, and
36 .15) mm/y with HRT placebo (P =.17); and for antioxidant vitamins by 0.044 (0.15) mm/y and with vitam
37 mage and mutagenesis, whereas addition of an antioxidant (vitamin C) can counteract the induced effec
38 each eddy, where it mixes and reacts with an antioxidant (vitamin C) present in the bulk solution.
40 as measured before and after infusion of the antioxidant vitamin C (25 mg/min i.a.) to determine the
41 Moreover, depletion of a murine endogenous antioxidant vitamin C (VC), by genetic disruption of VC-
42 lower in vivo HP (13)C DHA reduction to the antioxidant vitamin C (VitC), prior to histological evid
47 etermine whether acute administration of the antioxidant vitamin C modifies sympathetic nerve activit
48 sons who self-select for supplemental use of antioxidant vitamin C or E or multivitamins, large reduc
49 ter pylori have abnormally low levels of the antioxidant vitamin C, which protects against the format
54 (n = 8), or HC diet supplemented daily with antioxidant vitamins C (1 g) and E (100 IU/kg) (HC + vit
55 tenosis, n = 7), RVD daily supplemented with antioxidant vitamins C (1 g), and E (100 IU/kg) (RVD+Vit
56 atorvastatin to reduce LDL to <80 mg/dL plus antioxidant vitamins C (1000 mg/d) and E (800 mg/d) (n=1
57 nd, placebo-controlled trial, the effects of antioxidant vitamins C (500 mg/d) and E (400 IU/d) for 6
58 n pill taking was determined in the Trial of Antioxidant Vitamins C and E (TRACE) Study, a factorial
59 e tested whether long-term administration of antioxidant vitamins C and E improves coronary and brach
63 lated Eye Disease Study (AREDS) formulation (antioxidant vitamins C and E, beta carotene, and zinc) h
64 his decrease is blocked by pretreatment with antioxidant vitamins C and E, suggesting an oxidative me
65 to humans, we tested the hypothesis that the antioxidant, vitamin C, could improve endothelium-depend
66 lementation of subjects with the nutritional antioxidant, vitamin C, influences the ability of lympho
72 rms of AREDS supplement components: placebo, antioxidants (vitamin C, vitamin E, beta-carotene), zinc
73 e for 5 months with a diet supplemented with antioxidants (vitamins C and E, selenium, and N-acetylcy
76 ht to address whether intakes of the dietary antioxidants, vitamin C, alpha-tocopherol, and beta-caro
78 minantly from in vitro studies suggests that antioxidant vitamins can prevent lipid peroxidation and
81 able consumption on plasma concentrations of antioxidant vitamins, daily fruit and vegetable intake,
82 e after one year of therapy with statins and antioxidant vitamins, despite significant lipid-lowering
83 to an LDL level of 80 mg/dL, with or without antioxidant vitamins, does not provide any further benef
84 n of carnosic compared with a reference diet antioxidant (vitamin E), the animals were slaughtered an
85 s investigated the association between serum antioxidant (vitamins E, C, A, carotenoids, selenium) le
86 nt supplementation of the HFHC diet with the antioxidant vitamin E (250, 500, or 1000 IU/kg diet) for
89 treatment for three days with the endogenous antioxidant vitamin E (Vit-E, 100 mg/kg, i.p.) had an ev
90 ion, treatment of cells with a lipid-soluble antioxidant vitamin E blocked TAM-induced caspase-3 and
91 tert-butyl-alpha-phenylnitrone (PBN) and the antioxidant vitamin E established the involvement of fre
92 ent study, we investigated the effect of the antioxidant Vitamin E on the AD-like phenotype when give
95 , as well as the inability of the lipophilic antioxidant vitamin E to abrogate MAPK phosphorylation,
96 mice receiving a diet supplemented with the antioxidant vitamin E, higher levels of RPE bisretinoid
97 ignature of final structure is erased via an antioxidant vitamin E, subsequent to guided self-assembl
100 n = 7) diet, HC diet supplemented daily with antioxidant vitamins E (100 IU/kg) and C (1000 mg; HC+vi
102 viduals who regularly use supplements of the antioxidant vitamins E and C have a lower risk of ALS th
103 x vivo results, we depleted mice of a second antioxidant, vitamin E, which is normally absent under e
104 ination, on the concentrations of lipophilic antioxidants (vitamin E and gamma-oryzanol), soluble (in
106 n of protein kinase C (H-7), the addition of antioxidants (vitamin E or superoxide dismutase), or a p
109 of cats with a high dose combination of the antioxidants vitamin E (200 I.U./day) and selenium (50 m
110 The effects of PM were compared with the antioxidants vitamin E (VE) and R-alpha-lipoic acid (LA)
111 ists CV-6209 and WEB 2086, as well as by the antioxidants vitamin E and 1,1,3,3-tetramethyl-2-thioure
118 revention trial of the benefits and risks of antioxidant vitamins, folic acid, vitamin B6, and vitami
124 n regimen nor the combination of statins and antioxidant vitamins improved flow-mediated vasodilation
126 y intakes of magnesium, vitamin C, and other antioxidant vitamins in a general population cohort in N
128 e results suggest that beneficial effects of antioxidant vitamins in coronary artery disease are rela
129 Few data are available on the importance of antioxidant vitamins in earlier stages of atherogenesis.
133 To assess the role of ascorbate (AA), an antioxidant vitamin, in modulating striatal activity, si
136 oth free radical and oxidant exposure, while antioxidant vitamin intake is positively related to lung
137 e association of self-selected supplementary antioxidant vitamin intake on the rate of progression of
138 y symptoms in adults that are independent of antioxidant vitamin intake, but little is known about fi
142 wever, epidemiological evidence to implicate antioxidant vitamins is weak, and data on the role of fl
143 iatum contains a high level of ascorbate, an antioxidant vitamin known to play a key role in behavior
145 Our findings support the hypothesis that antioxidant vitamins may play a role in respiratory heal
147 tamin C, or supplementation of the diet with antioxidant vitamins, may inhibit oxidation and the proc
148 deficit in the release of ascorbate (AA), an antioxidant vitamin, occurs in the striatum of R6/2 mice
149 termine the long-term effects of statins and antioxidant vitamins on flow-mediated vasodilation of th
157 levels (p < 0.001); however, neither statin, antioxidant vitamin regimen nor the combination of stati
159 tigated the cross-sectional relation between antioxidant vitamin status and carotid atherosclerosis i
162 authors examined whether self-selection for antioxidant vitamin supplement use affects the incidence
163 assessed the effects of B-group vitamins and antioxidant vitamin supplementation on homocysteine conc
165 uggest an increase in overall mortality with antioxidant vitamin supplementation, at least in populat
166 oxidant system, through n-3 fatty acids plus antioxidant vitamin supplementation, could reduce the in
167 ies have demonstrated a beneficial impact of antioxidant vitamin supplements on lipid peroxidation, t
168 women with coronary disease, neither HRT nor antioxidant vitamin supplements provide cardiovascular b
170 his correlates with depletion of liposoluble antioxidant vitamins that are consumed during lipid pero
171 antihypertensives, aspirin, B-vitamins, and antioxidant vitamins) that are of proven or suspected be
173 rpose of this study was to determine whether antioxidant vitamin therapy improves endothelial functio
178 observed associations between mortality and antioxidant vitamin use after adjustment for confounders
179 zed, placebo-controlled studies suggest that antioxidant vitamins (vitamin E, vitamin C, beta-caroten
181 eopenia and fractures; and low levels of the antioxidant vitamins (vitamins A, E, and C) may increase
182 -adjusted intake of fruit and vegetables and antioxidant vitamins was estimated using a semi-quantita
184 sue endogenous oxygen radical scavengers and antioxidant vitamins were depleted and LDL oxidizability
185 ions of lipid hydroperoxides and liposoluble antioxidant vitamins were determined by using the FOX 2
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