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1 genes involved in cellular detoxification or antioxidation.
2 lator of mitochondrial energy metabolism and antioxidation.
3 ive system, possibly as a consequence of its antioxidation activity.
4  that include: enhanced cellular DNA repair, antioxidation and anti-inflammation.
5 hway by zinc provides powerful and prolonged antioxidation and detoxification that may explain the be
6 HDL to protect against atherogenesis through antioxidation and reverse cholesterol transport mechanis
7 tion-oxidation balance with yin representing antioxidation and yang as oxidation.
8 il activation, ROS production, intracellular antioxidation, and leukocyte migration plus genes for pr
9  involved in metabolism and nutrient uptake, antioxidation, and regulation of apoptosis.
10 omote direct antiatherogenic effects of HDL (antioxidation, anti-inflammation, antithrombotic effects
11 t ability to inhibit oxidation in a standard antioxidation assay in vitro.
12           It is conceivable that an enhanced antioxidation capability may constitute a common mechani
13 ponsive proteins including those involved in antioxidation, detoxification, and disease resistance.
14 the lipid-lowering, antiatherosclerosis, and antioxidation drug probucol extended life to as long as
15 ression studies show critical alterations in antioxidation, immune response, and cytokine pathways.
16  to clarify whether biomarkers of oxidation, antioxidation, inflammation, and nutritional factors dif
17 nd is probably involved in the regulation of antioxidation metabolism that is shared by both disease
18 t, exhibits effects of anti-inflammation and antioxidation on periodontal inflammation.
19          We propose that yin-yang balance is antioxidation-oxidation balance with yin representing an
20 sponsible for driving the vitamin K-mediated antioxidation pathways.
21               This is likely mediated by its antioxidation properties on LDL and/or macrophages.
22 lycerophospholipid metabolism, oxidation and antioxidation were associated with the process from pre-

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