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1         Levetiracetam was discovered to have antiseizure activity in animal models and was then found
2 potent adenosine kinase (AK) inhibitors with antiseizure activity in the rat maximum electroshock (ME
3 tones beta-hydroxybutryate or acetone, shows antiseizure activity in two acute ex vivo rat hippocampa
4 iform activity and significantly blocked the antiseizure activity of AP in the kindling model of epil
5                                          The antiseizure activity of benzodiazepines (BDZs) 1-5 in mi
6                     Finasteride reversed the antiseizure activity of DOC (ED50, 7.2 mg/kg); partial a
7 om), compatible with a role for DHDOC in the antiseizure activity of DOC.
8                            Separate from its antiseizure activity, dilantin interferes with microtubu
9                             Lacosamide is an antiseizure agent that targets voltage-dependent sodium
10 inhibitory neurotransmitter and an important antiseizure agent.
11 nosine kinase inhibitors (AKIs) as potential antiseizure agents and demonstrated an adenosine recepto
12 nge of applications, including screening for antiseizure agents and identifying channel blockers of i
13 ctivated states in a manner similar to other antiseizure agents but with slower kinetics of binding a
14 -hormonal drugs, such as antidepressants and antiseizure compounds to alleviate hot flushes.
15            In this study, the effects of the antiseizure drug valproic acid (VPA) on the expression o
16 tives and antipsychotic, antidepressant, and antiseizure drugs as well as drugs of abuse, such as coc
17                 Epilepsy therapy is based on antiseizure drugs that treat the symptom, seizures, rath
18          None of the patients was prescribed antiseizure drugs, either owing to physician recommendat
19 s fail to become seizure-free in response to antiseizure drugs.
20 /ED50) that compared favorably with clinical antiseizure drugs.
21             Low doses of MK801, which had no antiseizure effect, impaired the progression of kindling
22 nse oligonucleotides (antagomirs) had potent antiseizure effects in animal models, whereas genetic de
23       KB alone were sufficient to: (1) exert antiseizure effects in Kcna1-null mice, (2) restore intr
24  of this study was to demonstrate the direct antiseizure effects of KB and to identify an underlying
25 the conversion of DOC to DHDOC, reversed the antiseizure effects of stress.
26 und to mirror and reverse, respectively, the antiseizure effects of the KD in Kcna1-null mice.
27 anner, and this is sufficient to explain its antiseizure effects.
28 C2 inhibitor furosemide is nonselective with antiseizure efficacy in slices and in vivo, leading to a
29 ML297 revealed that in addition to its known antiseizure efficacy, ML297 decreases anxiety-related be
30 uring fasting or treatment with the high-fat antiseizure ketogenic diet (KD).
31 induced gingival overgrowth is caused by the antiseizure medication phenytoin, calcium channel blocke
32 ion with Epstein-Barr virus responded to the antiseizure medication valproate with entry into the lyt
33 dition may remain refractory to conventional antiseizure medications but may respond to immunotherapy
34 seizures despite adequate trials of standard antiseizure medications.
35  is insufficient to determine the effects of antiseizure medications.
36 ffinity at racetam-binding sites and greater antiseizure potency.
37 ice lacking PRs show supersensitivity to the antiseizure responses of progesterone.
38             The need for novel, efficacious, antiseizure therapies is widely acknowledged.
39 dels has led to the development of effective antiseizure therapies that are routinely used in clinica
40 test for seizure threshold, and rapidly tune antiseizure treatments.

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