戻る
「早戻しボタン」を押すと検索画面に戻ります。

今後説明を表示しない

[OK]

コーパス検索結果 (left1)

通し番号をクリックするとPubMedの該当ページを表示します
1                 We demonstrate that alpha 1C antisense ODNs abolish the increase in Gm in response to
2                         In contrast, gamma 2 antisense ODN treatments of cell cultures increased the
3                     Administration of Kir6.2 antisense ODN significantly attenuated apomorphine-induc
4 s, treatment of granule neurons with alpha 6 antisense ODNs caused a decrease in GABA-induced maximal
5    The pharmacodynamic effects of CGP 69846A antisense ODNs are therefore limited by the duration of
6                  iPLA(2)beta or cPLA(2)alpha antisense ODN-treated adoptively transferred mouse monoc
7                             Only the alpha2A antisense ODNs significantly change the hypnotic respons
8                                        At an antisense ODN concentration of 0.2 mumol/L, p42 MAPK pro
9 ense ODNs to neogenin and restin, but not an antisense ODN to rap1GAP, were effective in inhibiting O
10                                 In siRNA and antisense ODN databases, positive correlations are obser
11  the target gene is actively transcribed and antisense ODN is more active than sense ODN.
12  the considerations for RNA interference and antisense ODNs are reported.
13 re measured after infusion of BDNF sense and antisense ODNs with or without BDNF coinfusion, using th
14 gested for the design of efficient siRNA and antisense ODNs and the design of antisense ODNs is more
15 electively decreased only by the appropriate antisense ODNs and not by the "scrambled" ODNs.
16 prepared from cells treated with alpha 1B-AR antisense ODN demonstrated that alpha 1B-AR protein expr
17 i observed in cells treated with alpha 1A-AR antisense ODNs was reduced by 42%.
18 ned treatment with alpha 1A- and alpha 1B-AR antisense ODNs and antagonists additively inhibits PHE-i
19 observed with cells treated with alpha 1D-AR antisense ODNs or the alpha 1D-AR antagonist BMY 7378 co
20                            Furthermore, BDNF antisense ODN infusions into the CeA or MeA, but not int
21                     Here we report that BDNF antisense ODN infusions into the CeA and MeA, but not BL
22  transfect the monocytes and found that both antisense ODNs inhibited expression of their target prot
23 ivity of cortical cells was not disrupted by antisense ODN treatment in mature animals, indicating de
24 phages whose MR expression was suppressed by antisense ODN treatment.
25            Inhibition of Cux-1 expression by antisense ODNs was verified by reverse transcription-PCR
26 ion of DCs transduced with anti-CD80 or CD86 antisense ODN significantly prolonged the survival of he
27 , and transduced with anti-CD80 or anti-CD86 antisense ODNs (base-mismatched ODNs as controls).
28 ve RNase H recognition site) afford chimeric antisense ODNs that retain the ability to inhibit steroi
29                            In contrast, CREB antisense ODN-infused rats exhibited significantly impai
30 .e., short term memory) were similar in CREB antisense ODN and control groups.
31                                  We designed antisense ODNs complementary to the initiation codon reg
32                   Using human c-myc-directed antisense ODNs as a model for the application of this ap
33    Transient MCAO in rats infused with EAAC1 antisense ODNs had no significant effect on any of these
34 e ODN efficacy, but such that more effective antisense ODNs appear to target mRNA regions with greate
35 rovide a useful tool for selecting effective antisense ODNs in antisense research.
36 anslocation of Calpha triggered by exogenous antisense ODN treatment mirrors that observed in cells e
37 fficient siRNA, but the opposite is true for antisense ODNs.
38                                    The c-fos antisense ODN, however, failed to suppress MMP-1 or MMP-
39   When neocortical slices were prepared from antisense ODN-treated rats and incubated for 1 to 2 h in
40 pha 6 receptor subunit protein after a 48-hr antisense ODN treatment was assessed with the use of imm
41                                 Importantly, antisense ODN treatment did not impair visually driven a
42 al horn neurons were dramatically reduced in antisense ODN injected PSNL rats 1 week after injection.
43  difficult to avoid target self-structure in antisense ODN design.
44            Thus, we have shown that the iNOS antisense ODN specifically blocked iNOS expression and a
45                          Efficacy of the KOR antisense ODN treatment was behaviorally evaluated by as
46                   These studies suggest KSR1 antisense ODNs as a treatment for Ras-dependent human ma
47 tes were incubated with linear and stem-loop antisense ODNs targeted to Syk mRNA.
48   These results suggest that the use of mdr1 antisense ODNs in combination with standard antineoplast
49                                    Moreover, antisense ODN did not significantly affect IGFBP-4 prote
50 GF cell line was inhibited by 4 microM c-myc antisense ODN (14% decrease; P < or = 0.006) and 8 micro
51 decrease; P < or = 0.006) and 8 microM c-myc antisense ODN (approximately 80% decrease; P < or = 0.00
52 expressing breast cancer cells with HER2/neu antisense ODNs and conventional chemotherapeutic agents
53 urvival mechanisms and suggest that HER2/neu antisense ODNs may be of use in cancer therapeutics.
54  greatly enhanced the biological activity of antisense ODN.
55                  Moreover, administration of antisense ODN 1 day after training did not affect subseq
56 ocumented that pressure-mediated delivery of antisense ODN can functionally inhibit target gene expre
57                                The effect of antisense ODN on phenotype was examined by flow cytometr
58 3 was observed 10 d after the termination of antisense ODN treatment.
59                           Because the use of antisense ODN in human disease is already established in
60 se of electroporation to enhance delivery of antisense ODNs is a promising new approach towards ex vi
61 t siRNA and antisense ODNs and the design of antisense ODNs is more challenging.
62 promising lead analog for the development of antisense ODNs with increased potency.
63 e self-structure correlations to efficacy of antisense ODNs, conversely, are insignificant.
64                                 Inclusion of antisense ODNs, derived from the phosphotyrosine kinase
65                        Five-day infusions of antisense ODNs (5 and 10 nmol/day) in rats decreased imm
66 y improves the cellular uptake properties of antisense ODNs, as well as plasmid DNA.
67                                CD spectra of antisense ODNs exhibited specific responses to divalent
68 ng novel polyamines to facilitate the use of antisense ODNs for controlling HER-2 gene expression.
69            This agent expands the utility of antisense ODNs for their use in understanding gene funct
70                           A phosphorothioate antisense ODN against Bcl-2 reduced the AMPA-stimulated
71                             Phosphorothioate antisense ODNs targeted against accessible sites reduced
72                             Conventional PKC-antisense ODN treatment completely and significantly inh
73                                      PKCbeta-antisense ODN caused 89.2% inhibition of chemotaxis at i
74               A 15-mer phosphorothioate (PS) antisense ODN (erbB1AS15) induced a concentration-depend
75  of A10 cells to ISIS 11061, an active C-Raf antisense ODN, resulted in a potent, dose-dependent inhi
76 ent exposure of TGF-alpha or IGF-I and c-ret antisense ODN explants caused partial recovery from the
77 ramatic effects were observed with the c-ret antisense ODN.
78                         Both c-ros and c-ret antisense ODNs reduced the gene expression and biosynthe
79 unocytochemistry that treatment with RIalpha antisense ODN induces translocation of the Calpha subuni
80 ional PKC family, and next by using specific antisense ODN for PKCalpha and PKCbeta.
81                Transfection of MNCs with Src antisense ODNs blocked H-2g-induced MNC recruitment into
82 n, during cell-attached patch-clamp studies, antisense ODNs to alpha1c completely blocked the swellin
83                                         Such antisense ODNs suppress HER2/neu mRNA and protein levels
84                            The stem-loop Syk antisense ODN at a concentration of 0.2 microM inhibited
85  data indicate the efficacy of stem-loop Syk antisense ODN for targeting and degrading Syk mRNA and p
86                   In addition, stem-loop Syk antisense ODN inhibited Fc gamma RI and Fc gamma RIIIA-m
87 lexed with cationic liposomes, stem-loop Syk antisense ODN with phosphorothioate modification exhibit
88                                      The Syk antisense ODNs did not change beta-actin mRNA levels and
89                                          The antisense ODN treatment significantly reduced the clinic
90                                          The antisense ODN was administered intraventricularly to mic
91 n for efficient gene repair exhibited by the antisense ODN is its increased accessibility to the non-
92 phate were also significantly reduced by the antisense ODN treatment.
93 itric oxide synthase was not affected by the antisense ODN.
94 nimals killed to determine the effect of the antisense ODN on Kir6.2 mRNA.
95                       In the presence of the antisense ODN, but not sense or scrambled ODNs, the toxi
96 at we have targeted for disruption using the antisense ODN strategy one that has been of particular i
97 was unaffected in the same rats in which the antisense ODN effectively knocked-down the KOR as assess
98                             Furthermore, the antisense ODNs to Jak2 and Tyk2 both inhibited the induc
99 as not affected by treatment with any of the antisense ODNs.
100                                        These antisense ODNs were then administered three times, on al
101  altered mice and the rats treated with TLR4 antisense ODN displayed significantly attenuated behavio
102                             Injection of TNF antisense ODN on days 1 through 7 increased the area of
103    Group 2 received a daily injection of TNF antisense ODN or control on days 5 through 15 post-lesio
104  the day following the last injection of TNF antisense ODN.
105  control (reverse sense) ODN treatment or to antisense ODN injections targeted anterior or posterior
106 ry rate of Raf-1 mRNA after cell exposure to antisense ODNs as the half-life (t1/2 approximately 50 h
107           Moreover, inhibition of MDM2 using antisense ODNs also triggered MM cell apoptosis as evide
108 ture in the target appears to correlate with antisense ODN efficacy, but such that more effective ant
109  increase was abolished by pretreatment with antisense ODN.
110                            Transduction with antisense ODN targeting CD80 or CD86mRNA is a feasible a
111 completely abolished by prior treatment with antisense ODN, which had no effect on its own.
112                                Compared with antisense ODNs targeting of individual oncogenes, downre
113 NA that are accessible to hybridization with antisense ODNs.
114  to acetylcholine, whereas pretreatment with antisense ODNs blocked this effect.
115                         Embryos treated with antisense ODNs cleave normally and initiate gastrulation
116 or effects following systemic treatment with antisense ODNs plus doxorubicin in nude mice bearing hum

WebLSDに未収録の専門用語(用法)は "新規対訳" から投稿できます。
 
Page Top