ライフサイエンスコーパス: antisense oligodeoxyribonucleotide

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1 2) or cPLA(2) expression by their respective antisense oligodeoxyribonucleotide.

2 ls and HeLa cells to a calpain small subunit antisense oligodeoxyribonucleotide.

3 PKCdelta protein levels by PKCdelta-specific antisense oligodeoxyribonucleotides also inhibited 15-LO

4 hain product of laminin was reduced by using antisense oligodeoxyribonucleotides and DNA enzymes.

5 ions of JunD were investigated by using JunD antisense oligodeoxyribonucleotides and transient transf

6 agents such as ribozymes, antisense RNA and antisense oligodeoxyribonucleotides, and for optimizing

7 ased cPLA(2) protein expression, mediated by antisense oligodeoxyribonucleotides (AS-ODN) specific fo

8 2'-methoxyethoxy (MOE) nucleotides into the antisense oligodeoxyribonucleotide (ASO) of the heterodu

9 Raf, including small-molecule inhibitors and antisense oligodeoxyribonucleotides (ASON), are undergoi

10 -regulation of PKC epsilon protein levels by antisense oligodeoxyribonucleotides blocks MAPK activati

11 ion of the stability and conformation of the antisense oligodeoxyribonucleotides, d[CGCGTT x TTGCGC]

12 reating donor hearts with a phosphorothioate antisense oligodeoxyribonucleotide directed against Egr-

13 treating donor lungs with a phosphorothioate antisense oligodeoxyribonucleotide directed against the

14 Transfecting cortical neurons with antisense oligodeoxyribonucleotides directed against NP1

15 erapeutic interventional phase III trials of antisense oligodeoxyribonucleotides for a cancer indicat

16 eration that could be reversed by removal of antisense oligodeoxyribonucleotide from the culture medi

17 PKCdelta antisense oligodeoxyribonucleotides inhibited the phosph

18 oup 1 received a daily injection of TNFalpha antisense oligodeoxyribonucleotide (ODN) or control on d

19 Kir6.2 antisense oligodeoxyribonucleotide (ODN) was then admini

20 Although iPLA(2)beta or cPLA(2)alpha antisense oligodeoxyribonucleotide (ODN)-treated monocyt

21 We have designed a new class of modified antisense oligodeoxyribonucleotides (ODN) consisting of

22 bit individual PKC isoforms, we chose to use antisense oligodeoxyribonucleotides (ODN) to specificall

23 esents an attractive target for therapy with antisense oligodeoxyribonucleotides (ODN).

24 y involved in 15-LO expression, we generated antisense oligodeoxyribonucleotides (ODNs) against Tyk2

25 e effects of transduction with high-affinity antisense oligodeoxyribonucleotides (ODNs) designed to t

26 ffects of HER2/neu-specific phosphorothioate antisense oligodeoxyribonucleotides on HER2/neu expressi

27 epeated intracerebroventricular injection of antisense oligodeoxyribonucleotide to Kv1.1 reduces expr

28 nalysis with specific antibodies showed that antisense oligodeoxyribonucleotide to Kv1.4 microinjecte

29 Furthermore, when M21 cells transfected with antisense oligodeoxyribonucleotide to the hnRNP L RNA-bi

30 Using antisense oligodeoxyribonucleotide treatment we found th

31 harmacological inhibitors, PKCdelta-specific antisense oligodeoxyribonucleotides were used.

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