1 rn for each sst mRNA was identical with both
antisense probes.
2 We performed in situ hybridization using an
antisense probe against the major epitope of PDC-E2.
3 During isoflurane gas anesthesia,
antisense probes against the c-fos mRNA sequence were in
4 Using
antisense probes and polyclonal antibodies, we show that
5 The remaining three
antisense probes based upon exons 1 and 2 are inactive.
6 so acts through delta2 receptors and all the
antisense probes block spinal DPDPE analgesia.
7 ha 1, Gi alpha 3, G(o) alpha, and Gx/z alpha
antisense probes block spinal M6G analgesia.
8 analgesia, whereas Gi alpha 2 and Gx/z alpha
antisense probes block spinal mu analgesia.
9 Gi alpha 1 and Gx/z alpha
antisense probes block supraspinal M6G analgesia, wherea
10 ail vein administration of the (99m)Tc-CCND1
antisense probe but not the control probe.
11 Administration of miR-21
antisense probes diminished the severity of experimental
12 The screening of the cDNA libraries with
antisense probes from the three conditions enhanced the
13 probe at a much lower concentration than the
antisense probe generates a larger product that can be r
14 ay, 75% of the sense probes and 11.9% of the
antisense probes have signal above background and can be
15 In situ hybridization with sense and
antisense probes localized Rgs13 expression to the germi
16 Conversely, an
antisense probe selectively targeting nNOS-2 blocks morp
17 s examined by in situ hybridization using an
antisense probe specific of HHV-8.
18 However, supraspinally only the two
antisense probes targeting exon 3 block DPDPE analgesia.
19 ine injections for 5 days, mice treated with
antisense probes targeting nNOS-1 show no decrease in th
20 The
antisense probe that is being used to detect endogenous
21 n situ hybridization histochemistry using an
antisense probe to this novel receptor mRNA shows a dist
22 Spinal delta-opioid analgesia is blocked by
antisense probes to all of the G protein alpha subunits
23 dy the possible role of IK in hematopoiesis,
antisense probes were used.