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1 l-2-targeted antisense G3139 as archetypical antisense therapeutics.
2 action and hampered clinical development of antisense therapeutics.
3 oping potent, cost-effective, self-permeable antisense therapeutics.
4 leotides have been crucial to the success of antisense therapeutics.
5 mRNA molecule constitutes a major problem in antisense therapeutics.
6 roup a leading choice for incorporation into antisense therapeutics.
7 plication of peptide nucleic acids (PNAs) as antisense therapeutics.
8 ides have been crucial to the development of antisense therapeutics.
9 establish FHNA as a useful modification for antisense therapeutics and also confirm the stabilizing
10 ead to identification of clinically relevant antisense therapeutics and can identify which molecular
11 a cause of C9ORF72 ALS and provide candidate antisense therapeutics and candidate human pharmacodynam
12 establish tcDNA as a useful modification for antisense therapeutics and highlight the role of chemica
13 picomoles of remaining protecting groups on antisense therapeutics and oligonucleotide diagnostics.
16 e modifications currently being evaluated as antisense therapeutics are tolerated by the enzyme, amon
17 hosphoramidates are promising candidates for antisense therapeutics, as well as for diagnostic applic
18 reported here are relevant to the design of antisense therapeutics comprised of these modifications.
21 n significant progress in the development of antisense therapeutics for a wide range of medicinal app
22 of the major barriers to the development of antisense therapeutics has been their poor bioavailabili
24 over novel oligonucleotide modifications for antisense therapeutics, we have prepared oligodeoxyribon
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