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1 lishment of hypoxia, metronidazole showed no antituberculous activity in this in vivo hypoxic granulo
3 trophils contain antimicrobial peptides with antituberculous activity, but their contribution to immu
5 ween two distinct models of human macrophage antituberculous activity: primary infection of naive mac
6 bility of M. tuberculosis to four first-line antituberculous agents demonstrated equivalent interpret
7 hepatotoxicity was observed with first line antituberculous agents using four drug standard inductio
11 hese agents may be considered as adjuncts to antituberculous chemotherapy, and may be particularly us
15 traditional view that acquired resistance to antituberculous drugs is driven by poor compliance and p
16 n cases) had isolates resistant to 1 or more antituberculous drugs, and 3 (19% of all Tibetan cases)
17 e-bound structure, we suggest a new class of antituberculous drugs, made by connecting two trehalose
18 Given the low potency of many second-line antituberculous drugs, we hypothesized that an aggressiv
23 atient was originally started on a four-drug antituberculous regimen of isoniazid, rifampin, ethambut
25 rculous symptomatology and lesions following antituberculous therapy (paradoxical response) has previ
26 tients with AIDS and TB who are treated with antituberculous therapy and subsequently with combinatio
27 ransferases are highly promising targets for antituberculous therapy by using antisense or other anti
28 ogical immunomodulatory compounds to enhance antituberculous therapy has been hampered by the limited
29 he myocardium, and we believe that effective antituberculous therapy has led to resolution of the GCM
34 spite a generally good response to effective antituberculous therapy, the prognosis remains poor.
37 positive early in the course of efficacious antituberculous treatment, we predict that both breath a
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