ライフサイエンスコーパス: antitumor action

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1 ic side effects of DOX without affecting its antitumor action.

2 cells and certain heat shock proteins having antitumor action.

3 structure and function and its unique potent antitumor action.

4 also weakened its interaction with STAT3 and antitumor action.

5 nt in vitro activities and promising in vivo antitumor actions.

6 The antitumor action and quantitative tumor perfusion studie

7 visualization of new targets exploitable for antitumor action; and (c) multifactorial analysis of the

8 wn, the structural elements required for its antitumor actions are not understood.

9 r demonstrated, in vivo, the efficacy of VSV antitumor action by showing that tumors that are defecti

10 determine the mechanism behind the observed antitumor action, cell cycle analysis was performed, and

11 nitrite than Saq-NO in cell cultures and its antitumor action depended on the intracellular NO releas

12 Lack of antitumor action despite positive immune stimulation was

13 The molecular basis for their antitumor action, however, is largely unknown.

14 showed that RNPS1 and ADD1 exerted multiple antitumor actions in OVCA cells, while PLXNA3 only inhib

15 The proposed mechanisms of antitumor action include direct or indirect inhibition o

16 ns, using mass spectrometry, that permit its antitumor actions led to the inhibitor of cyclin-depende

17 To investigate the antitumor action of arsenic trioxide in solid tumors, we

18 regulation of WT p53 might contribute to the antitumor action of BN/GRP antagonists.

19 Moreover, the mechanism of antitumor action of compound 30 was validated by its eff

20 The molecular mechanism of the antitumor action of DBPT was further analyzed in three h

21 by the IGFR/EGFR heterodimer counteracts the antitumor action of erlotinib, indicating the needs of i

22 partially maintaining P-Akt and limiting the antitumor action of lapatinib.

23 ith anti-CD40 mAb treatment, assessed by the antitumor action of macrophages in vitro.

24 ations that could be targeted to enhance the antitumor action of these drugs.

25 eeded to determine if this has a role in the antitumor actions of EGCG.

26 c amino acids within this motif cripples the antitumor actions of GRIM-19.

27 tumor viruses are etiologic agents and that antitumor actions of IRF-1/ICSBP can be exploited therap

28 We tested the hypothesis that these antitumor actions of n-3 PUFA may involve the n-3 olefin

29 ctions of IFNs, are also responsible for the antitumor actions of the IFN-RA combination.

30 , it is unknown whether IFN-alpha exerts its antitumor actions primarily through a direct effect on m

31 of diminished general toxicity and effective antitumor action; this group included VSV-M51, VSV-CT9-M

32 ased barely detectable amounts of NO and its antitumor action was NO-independent.

33 importance of Akt signaling in trastuzumab's antitumor action, we hypothesized that concurrent inhibi

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