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1 a necessary step in creating broad-spectrum antivenom.
2 ring medical attention and administration of antivenom.
3 requiring medical intervention and sometimes antivenom.
4 or delayed hypersensitivity reactions to the antivenom.
5 tions during the first application of equine antivenom.
6 epitopes were detected by immunoblotting on antivenoms.
7 e associated with hypersensitivity to equine antivenoms.
8 target of IgE-mediated reactivity to equine antivenom and a possible cause of the high incidence of
11 By detection of binding for three different antivenoms and performing an alanine scan, linear elemen
12 nderstand the molecular interactions between antivenom antibodies and epitopes on snake venom toxins,
16 the efficacy of scorpion-specific F(ab')(2) antivenom, as compared with placebo, was assessed in 15
17 e aimed to develop an equivalent recombinant antivenom composed of a few neutralizing single chain an
18 and ImmunoCAP inhibition indicated that the antivenoms contained lower alpha-gal contents than cetux
21 tests were used to investigate reactivity to antivenoms in samples from 20 patients with specific IgE
22 y rendering the specific antibodies found in antivenoms ineffective against heterologous toxins found
29 stered in the placebo recipients than in the antivenom recipients (mean cumulative dose, 4.61 vs. 0.0
30 oncentrations were undetectable in all eight antivenom recipients but in only one placebo recipient 1
31 , with a resolution of symptoms in all eight antivenom recipients versus one of seven placebo recipie
32 dministration of scorpion-specific F(ab')(2) antivenom resolved the clinical syndrome within 4 hours,
33 esolved more rapidly among recipients of the antivenom than among recipients of placebo, with a resol
34 rong protective power of small quantities of antivenom used in the context of severe envenomation wit
37 othesized that a scorpion-specific F(ab')(2) antivenom would promptly resolve clinical symptoms in ch
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