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1 mechanisms that control endocytosis in this anucleate cell.
2 ization in the regulatory repertoire of this anucleate cell.
3 owth were impaired in viability and produced anucleate cells.
4 tions nor, obviously, transcription by these anucleate cells.
5 he temperature-sensitivity and production of anucleate cells.
6 to a 100-fold increase in the production of anucleate cells.
7 spoIIIE, led to an increase in production of anucleate cells.
8 utants are temperature-sensitive and produce anucleate cells.
9 segregation and divide frequently to produce anucleate cells.
10 tivation leading to increased frequencies of anucleate cells.
14 segregation defects, including formation of anucleate cells, compact nucleoids confined to one half
15 y closer to the cell extremities, whereas in anucleated cells (deletion mutants for mukB), the Tsr cl
16 g aberrant nuclear division, as well as many anucleate cells, demonstrating that the TRF4/5 function
24 ther examination of FtsZ ring positioning in anucleate cells generated by the parC and mukB mutants:
26 in the production of approximately 0.9 to 3% anucleate cells in prfA cultures grown at 30 or 37 degre
27 e ion channels are functional in these tiny, anucleate cells is difficult to assess by direct electro
30 they suppressed temperature sensitivity and anucleate cell production of cells containing null or po
31 ricted to low temperature with production of anucleate cells, reflecting chromosome segregation defec
34 also confirm that this is a feature of other anucleate cells through transcriptome sequencing of matu
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