ライフサイエンスコーパス: anxiety disorder

1 ion making have been reported in generalized anxiety disorder.

2 with generalized, social, and/or separation anxiety disorder.

3 rments during decision making in generalized anxiety disorder.

4 d reinforcement) is disrupted in generalized anxiety disorder.

5 those with a social component such as social anxiety disorder.

6 s a potent risk factor for development of an anxiety disorder.

7 tests are common preclinical models of human anxiety disorder.

8 disorders, with the exception of separation anxiety disorder.

9 ine a gaze-contingent intervention in social anxiety disorder.

10 isorder protocols (SDPs) in the treatment of anxiety disorders.

11 y among respondents with no history of prior anxiety disorders.

12 e risk of developing depression episodes and anxiety disorders.

13 s to non-threatening stimuli is a feature of anxiety disorders.

14 with other psychiatric illnesses, including anxiety disorders.

15 cated in the pathophysiology of FND and mood/anxiety disorders.

16 ritical role in the neurobiology of mood and anxiety disorders.

17 goals of therapeutic interventions in human anxiety disorders.

18 nding of the molecular mechanisms underlying anxiety disorders.

19 r to a mechanistic understanding of mood and anxiety disorders.

20 contribute to the pathophysiology of chronic anxiety disorders.

21 ole of Asb1-mediated immune dysregulation in anxiety disorders.

22 r trauma- and stressor-related disorders and anxiety disorders.

23 n is widely thought to underpin a variety of anxiety disorders.

24 n may have a profound influence on recurring anxiety disorders.

25 onses may thus form an etiological basis for anxiety disorders.

26 y including ones regarding depressive and/or anxiety disorders.

27 eat responses is a robust clinical marker of anxiety disorders.

28 l appeared to be better than CAU in treating anxiety disorders.

29 rary pathophysiological accounts of clinical anxiety disorders.

30 ment of frontolimbic circuitry implicated in anxiety disorders.

31 processing is a key factor, namely, fear and anxiety disorders.

32 ches for alcohol-withdrawal-induced mood and anxiety disorders.

33 r waiting-list control for depressive and/or anxiety disorders.

34 ger psychiatric illnesses including mood and anxiety disorders.

35 lying vulnerability factor for depressive or anxiety disorders.

36 and outcome of exposure treatment across the anxiety disorders.

37 tion of Mental Disorders) to assess mood and anxiety disorders.

38 R1 as a possible candidate gene for mood and anxiety disorders.

39 ents to SSRI treatment in some patients with anxiety disorders.

40 dysfunctions across the spectrum of mood and anxiety disorders.

41 ry features of these treatments in pediatric anxiety disorders.

42 lleviating maladaptive memories accompanying anxiety disorders.

43 n of the word "bipolar" and search terms for anxiety disorders.

44 c of posttraumatic stress disorder and other anxiety disorders.

45 y the principal treatment for depression and anxiety disorders.

46 therapeutic development in individuals with anxiety disorders.

47 raging further research on generalization in anxiety disorders.

48 npredictable threat in patients with various anxiety disorders.

49 cortical depression, as reported in mood and anxiety disorders.

50 pivotal role in the pathogenesis of mood and anxiety disorders.

51 ss is an established cause of depression and anxiety disorders.

52 2 [0.32%]; 1.54, 1.11-2.13), but not mood or anxiety disorders.

53 has been implicated anxiogenic behaviors and anxiety disorders.

54 al for formulating successful treatments for anxiety disorders.

55 on is likely to contribute to depression and anxiety disorders.

56 ERN is not entirely static in patients with anxiety disorders.

57 ogical organisms, and a laboratory model for anxiety disorders.

58 goals of therapeutic interventions in human anxiety disorders.

59 vel mechanism of action for the treatment of anxiety disorders.

60 c properties and is reduced in patients with anxiety disorders.

61 ed on connections between manic episodes and anxiety disorders.

62 rapy (CBT) and pharmacotherapy for childhood anxiety disorders.

63 for the treatment and prevention of mood and anxiety disorders.

64 mygdala are implicated in the development of anxiety disorders.

65 shed light on the pathogenesis of underlying anxiety disorders.

66 ritical role in the neurobiology of mood and anxiety disorders.

67 69, 0.63-0.75 in South London and Maudsley), anxiety disorders (0.81, 0.69-0.96 in Camden and Islingt

68 .05, 95% CI 1.21-3.45), increased odds of an anxiety disorder (2.27, 1.2-4.28), and major depression

69 ire-2 for depression, a two-item Generalized Anxiety Disorder-2 questionnaire for anxiety, and a ques

70 controls (10 women) and 61 individuals with anxiety disorders (36 women).

71 espectively; suicidality, 20.2%; generalized anxiety disorder, 7.9%; posttraumatic stress disorder, 9

72 Patient Health Questionnaire-9, Generalised Anxiety Disorder-7 scale, Alcohol Use Disorder Identific

73 dence, stability, and predictors of mood and anxiety disorders across a 3-year period.

74 Alcohol use disorders (AUDs) and anxiety disorders (ADs) are often seen concurrently, but

75 Anxiety disorders (ADs), namely generalized AD, panic di

76 Depressive disorders (DDs), anxiety disorders (ADs), obsessive-compulsive disorder (

77 external threats is a key factor in mood and anxiety disorder aetiology.

78 Most anxiety disorders affect almost twice as many women as m

79 Anxiety disorders affect approximately 1 in 5 (18%) Amer

80 olar disorder, major depression, generalized anxiety disorder, agoraphobia, social phobia, obsessive-

81 promise for reducing the 1-year incidence of anxiety disorders among offspring of anxious parents.

82 n, which further mediated increased risk for anxiety disorders among these same individuals.

83 six unmedicated individuals with generalized anxiety disorder and 32 healthy comparison subjects grou

84 six unmedicated individuals with generalized anxiety disorder and 32 healthy comparison subjects grou

85 For social anxiety disorder and attention-deficit/hyperactivity dis

86 oderate/high risk), past 6-month generalized anxiety disorder and posttraumatic stress disorder, and

87 itted and current diagnoses of depressive or anxiety disorders and 644 healthy control subjects.

88 e CeA as a potential integrative hub between anxiety disorders and alcohol use disorder, which are co

89 Edition, which addresses OCD separately from anxiety disorders and contains specifiers to delineate t

90 , including schizophrenia, bipolar disorder, anxiety disorders and depression.

91 ive in the treatment of eating disorders and anxiety disorders and has shown promising effects in som

92 call did not differ between individuals with anxiety disorders and healthy controls (etap2 = 0.001, P

93 provide a better cognitive understanding of anxiety disorders and hence inform treatment strategies.

94 s impeded progress in understanding fear and anxiety disorders and hindered attempts to develop more

95 ports, these results suggest that history of anxiety disorders and history of a limited number of ear

96 sive-compulsive disorder, schizophrenia, and anxiety disorders and lower levels associated with subst

97 , it did not demonstrate that depressive and anxiety disorders and LTL change together over time, sug

98 mine the relationship between depressive and anxiety disorders and LTL over a 6-year time period.

99 n strategy for affective disorders including anxiety disorders and major depression.

100 al neurobiology of behavioral inhibition and anxiety disorders and may aid in early risk assessment a

101 ferences in the association between mood and anxiety disorders and myocardial ischemia in patients wi

102 pproaches for psychiatric conditions such as anxiety disorders and posttraumatic stress disorder.

103 therapies for those suffering from mood and anxiety disorders and provide insight into addiction.

104 eCB-based treatment approaches for mood and anxiety disorders and suggest a potentially wider therap

105 f fear conditioning and extinction recall in anxiety disorders and to document how these features dif

106 sed psychological therapy for depression and anxiety disorders) and primary care and community outrea

107 utcomes were PTSD, depression or generalised anxiety disorder, and alcohol misuse at follow-up.

108 ressive episode, phobias, panic, generalized anxiety disorder, and obsessive-compulsive disorder), su

109 s for major depressive disorder, generalized anxiety disorder, and social anxiety disorder, suggest t

110 ting for comorbid major depressive disorder, anxiety disorder, and substance use disorder (suicide at

111 PTSD), major depressive episode, generalized anxiety disorder, and suicidality, as well as presence a

112 China and India, major depressive disorder, anxiety disorders, and alcohol dependence were the most

113 is implicated in schizophrenia, depression, anxiety disorders, and autism.

114 in D deficiency or metabolic bone disorders, anxiety disorders, and fractures; the incidence of pneum

115 nia spectrum disorders, affective disorders, anxiety disorders, and other neurotic and personality di

116 spectrum disorder, substance use disorders, anxiety disorders, and personality disorders.

117 depressive disorder (MDD), bipolar disorder, anxiety disorders, and schizophrenia.

118 hizophrenia, bipolar or unipolar depression, anxiety disorders, and substance use disorders).

119 7; 95% confidence interval: 1.3-2.2) and any anxiety disorder (AOR: 1.8; 1.4-2.2), although not of su

120 ave 2 manic episodes (AOR: 2.2; 1.7-2.9) and anxiety disorders (AOR: 1.7; 1.5-2.0), although not subs

121 Anxiety disorders are associated with disruptions in bot

122 Many anxiety disorders are characterized by generalization of

123 Anxiety disorders are common in adolescents (ages 12 to

124 Anxiety disorders are commonly associated with increased

125 Anxiety disorders are debilitating psychiatric illnesses

126 Anxiety disorders are increasingly recognised as an impo

127 Current treatment strategies for anxiety disorders are predominantly symptom-based.

128 Mood and anxiety disorders are the most prevalent psychiatric con

129 panic disorder, agoraphobia, and generalised anxiety disorder) are common and disabling conditions th

130 Post-traumatic stress disorder (PTSD) is an anxiety disorder arising from exposure to a traumatic ev

131 r, posttraumatic stress disorder, and social anxiety disorder, as well as autism and schizophrenia, a

132 major depression, depressive disorders, and anxiety disorders, as well as those with parental loss a

133 evated adjusted odds of PTSD and generalized anxiety disorder at T2 and T3 and of major depressive ep

134 In the treatment of anxiety disorders, attention bias modification therapy (

135 ty, schizophrenia, mood affective disorders, anxiety disorders, autism spectrum disorders, attention

136 traumatic stress disorder, addiction, social anxiety disorder, bipolar disorder, schizophrenia, and a

137 ditions in psychiatry, including depression, anxiety disorders, bipolar disorder and schizophrenia.

138 with schizophrenia and negative symptoms and anxiety disorder but not with psychotic experiences or d

139 Benzodiazepines are used in the treatment of anxiety disorders but have limited long-term use due to

140 erous domains often dysregulated in mood and anxiety disorders, but that individual signs depend on b

141 hen individuals elude harmless situations in anxiety disorders, but the neural circuits that mediate

142 Changes in the course of depressive or anxiety disorder characteristics over 6 years, however,

143 traumatic stress disorder (PTSD) is a severe anxiety disorder characterized by re-experiencing, avoid

144 an 18% (95% CI, 13%-22%) increased risk for anxiety disorders compared with men with resting heart r

145 y participants and individuals with mood and anxiety disorders completed an approach-avoidance go/no-

146 Anxiety disorders constitute a major disease and social

147 d parasympathetic activity characteristic of anxiety disorders could further augment inflammation and

148 arly-life risk factor for the development of anxiety disorders, depression and co-morbid substance ab

149 After the exclusion of persons with comorbid anxiety disorders, depression, or substance use disorder

150 Comorbid anxiety disorders, depression, or substance use disorder

151 those with antisocial personality disorder, anxiety disorders, depressive disorders, and a history o

152 e-contingent music reward therapy for social anxiety disorder designed to reduce attention dwelling o

153 ng Assessment at 15.5 years of age), and (4) anxiety disorder (Development and Well-Being Assessment

154 nction recall) and the number of co-occuring anxiety disorders diagnosed (etap2 = 0.137, P = .009 for

155 ility significantly decreased adjustment and anxiety disorder diagnoses among their children.

156 t baseline and after 6 years; depressive and anxiety disorder diagnoses and characteristics (course,

157 02) signaling modulate amygdala function and anxiety disorder diagnosis.

158 ce was that major depression and generalized anxiety disorder dimensions had small but significant lo

159 esent potential options for the treatment of anxiety disorders due to their potent anxiolytic profile

160 ss increases vulnerability to alcoholism and anxiety disorders during adulthood.

161 whether they experienced major depression or anxiety disorders during that year as well as before tra

162 Half of all lifetime anxiety disorders emerge before age 12 years; however, a

163 y suggest the relevance of such behavior for anxiety disorders, especially agoraphobia.

164 n that dysregulated fear levels characterize anxiety disorders, examining the neural correlates of fe

165 Patients with social anxiety disorder exhibit increased attentional dwelling

166 ponse protects against the development of an anxiety disorder, experiencing more than one trauma (mul

167 dwide, is associated with acute anxiety, and anxiety disorders following regular use.

168 D), major depressive disorder (MDD), and the anxiety disorders-for which environmental stressors and

169 ermine whether panic disorder or generalized anxiety disorder (GAD) in pregnancy, or medications used

170 Generalized anxiety disorder (GAD) is a common chronic condition tha

171 Generalized anxiety disorder (GAD) is characterized by the core symp

172 Generalized anxiety disorder (GAD) is common in older adults; howeve

173 raumatic stress disorder (PTSD), generalized anxiety disorder (GAD), panic disorder (PD), and phobias

174 -72 years) were administered the Generalized Anxiety Disorder (GAD-7) instrument, whole blood DNA met

175 nxiety (measured by the two-item Generalised Anxiety Disorder [GAD]-2 anxiety scale), and post-trauma

176 for full or probable diagnoses of separation anxiety disorder, generalized anxiety disorder, social p

177 individuals with at least 1 of the following anxiety disorders: generalized anxiety disorder, social

178 B=-52.6) and current (B=-60.8) depressive or anxiety disorder had consistently shorter LTL compared w

179 udy of neurobiological mechanisms underlying anxiety disorders has been shaped by learning models tha

180 hment, reoperation within 30 days, dementia, anxiety disorder, hearing difficulty, or history of drug

181 present a novel approach to the treatment of anxiety disorders; however, the functional interactions

182 rlapping behavioral problems (ASD, ADHD, and anxiety disorders), hypotonia, broad-based gait, facial

183 g growth mixture modeling, of past-year mood/anxiety disorder (ie, major depressive episode, phobias,

184 onpsychotic disorders: mood disorder in 49%, anxiety disorder in 35%, and substance use disorder in 2

185 y data show that maternal panic disorder, or anxiety disorders in general, increase the risk for adve

186 aimed to quantify the lifetime prevalence of anxiety disorders in individuals with bipolar disorder a

187 y-based intervention to prevent the onset of anxiety disorders in offspring of anxious parents.

188 herapeutic compounds to treat depression and anxiety disorders in women.

189 ay contribute to increased susceptibility to anxiety disorders in women.

190 relatively high prevalence and emergence of anxiety disorders in youth, very little work has evaluat

191 f psychiatric diseases, such as addiction or anxiety disorders, in which patients generalize inapprop

192 genetic contributions to PTSD, MDD, and the anxiety disorders including genetic epidemiology, the ro

193 etween chronic stress and the development of anxiety disorders, including post-traumatic stress disor

194 is a significant risk factor for developing anxiety disorders, including posttraumatic stress disord

195 the study of GxE interactions in stress and anxiety disorders, including the evolution of genetic as

196 e MDD increased from 46% to 54% and lifetime anxiety disorders increased from 28% to 35%.

197 Like many patients with anxiety disorders, individuals with elevated levels of d

198 r treating posttraumatic stress disorder and anxiety disorders involving abnormal memories are emergi

199 In anxiety disorders, IPT had large effects compared with c

200 At the core of anxiety disorders lies the tendency to generalize fear f

201 For women, anxiety disorders, major depressive disorder, and dysthy

202 renia, and borderline personality and social anxiety disorders, may be reduced by oxytocin administra

203 ttentional processing in social anxiety, and anxiety disorders more broadly, have postulated an initi

204 that reflect other core features of mood and anxiety disorders (motivation, social interaction, and a

205 Compared to patients with generalized anxiety disorder (n = 24) and healthy control subjects (

206 r to treatment, youths (8-17 years old) with anxiety disorders (N=54), as well as healthy comparison

207 M age=25.8+/-8.5; 67% female) with principal anxiety disorders (n=60) or no lifetime history of Axis

208 In addition, anxiety disorder NOS increased from 44.6% in the 1999-20

209 der with or without agoraphobia, generalized anxiety disorder, obsessive-compulsive disorder, or soci

210 : Dates of inpatient/outpatient diagnoses of anxiety disorders, obsessive-compulsive disorder, posttr

211 -environmental reasons why hyperactivity and anxiety disorders occur at higher rates in deaf individu

212 feeding and eating disorders, schizophrenia, anxiety disorder, OCD, and most affective disorders also

213 egulation of anxiety, and may play a role in anxiety disorders of the amygdala.

214 Patients with anxiety disorders often experience a relapse in symptoms

215 Incidence of new cases of anxiety disorders or reduction of anxiety symptoms as me

216 , 1.21; 95% CI, 1.08-1.36; R(2) = 0.007) and anxiety disorder (OR per SD increase in PRS, 1.17; 95% C

217 mood disorder (OR, 1.1; 95% CI, 0.8-1.4) or anxiety disorder (OR, 0.9; 95% CI, 0.7-1.1).

218 ime DUD was also associated with generalized anxiety disorder (OR, 1.3; 95% CI, 1.06-1.49), panic dis

219 OR, 6.26 [95% CI, 3.07-12.76]), generalized anxiety disorder (OR, 5.19 [95% CI, 2.01-13.38]), conduc

220 iety disorder, specific phobias, generalized anxiety disorder, or separation anxiety and who received

221 the 16 patients had comorbid mood disorders, anxiety disorders, or both.

222 matic stress disorder, eating disorders, and anxiety disorders other than specific phobia, and was po

223 lence of major depressive disorder (MDD) and anxiety disorders over time, with adverse outcomes predi

224 diagnosed as having specific phobia, social anxiety disorder, panic disorder with or without agoraph

225 , selective mutism, specific phobias, social anxiety disorder, panic disorder, agoraphobia, and gener

226 dogenous brain OXT system in the etiology of anxiety disorders, particularly those with a social comp

227 campus and amygdala differ between pediatric anxiety disorder patients and healthy volunteers (HVs).

228 Anxiety disorders peak in incidence during adolescence,

229 The onset of depression and anxiety disorders peaks during adolescence and there are

230 en largely unexplored (for example, mood and anxiety disorders, persistent pain syndromes or even Par

231 of GPR88 therefore extends to cognitive and anxiety disorders, possibly in interaction with other re

232 and social dysfunctions, such as generalized anxiety disorder, posttraumatic stress disorder, and soc

233 same instrument in a study of patients with anxiety disorders recruited at the Max Planck Institute

234 In addition, individuals with generalized anxiety disorder relative to healthy participants showed

235 uring feedback, individuals with generalized anxiety disorder relative to healthy subjects showed a r

236 Although anxiety disorders represent a major societal problem dem

237 ertoire of effective treatments for mood and anxiety disorders represents a critical unmet need.

238 ive fear-related behaviors characteristic of anxiety disorders result.

239 rnalizing conditions of major depression and anxiety disorders, risk was associated with low SA but w

240 ressive disorder (RR 1.61; 95%CI 1.55-1.68), anxiety disorder (RR 1.52; 95%CI 1.48-1.56), and ADHD (R

241 ual disability (RR, 11.5; 95% CI, 8.5-15.6), anxiety disorders (RR, 2.9; 95% CI, 1.3-6.5), autism spe

242 Many patients with social anxiety disorder (SAD) experience inadequate symptom rel

243 Social anxiety disorder (SAD) is characterized by fears of soci

244 Cognitive models posit that social anxiety disorder (SAD) is maintained by biased informati

245 ity were measured in 38 patients with social anxiety disorder (SAD) to predict subsequent treatment r

246 ut whether anxiety syndromes, such as social anxiety disorder (SAD), are characterized by an overacti

247 haracteristic negative memory bias in social anxiety disorder (SAD).

248 eases the risk for later diagnosis of social anxiety disorder (SAD).

249 g to the emergence and maintenance of social anxiety disorder (SAD).

250 Anxiety disorders (separation anxiety disorder, selective mutism, specific phobias, so

251 Anxiety disorders (separation anxiety disorder, selectiv

252 duct disorder, major depression, generalized anxiety disorder, separation anxiety disorder, social ph

253 nduct disorder,major depression, generalized anxiety disorder, separation anxiety disorder, social ph

254 94 participants; 3.60, 2.45-5.30, p<0.0001), anxiety disorders (seven studies, 5091 participants; 1.9

255 Behaviorally, individuals with generalized anxiety disorder showed impaired reinforcement-based dec

256 ightened autonomic drive, as seen in chronic anxiety disorders.SIGNIFICANCE STATEMENT The dialogue be

257 e show in the current report that only prior anxiety disorders significantly predicted PTSD in a mult

258 llowing outcomes: major depressive disorder, anxiety disorder, smoking and alcohol consumption, illic

259 the following anxiety disorders: generalized anxiety disorder, social anxiety disorder, specific phob

260 of separation anxiety disorder, generalized anxiety disorder, social phobia, major depression, dysth

261 on, generalized anxiety disorder, separation anxiety disorder, social phobia, specific phobia, agorap

262 on, generalized anxiety disorder, separation anxiety disorder, social phobia, specific phobia, agorap

263 orders: generalized anxiety disorder, social anxiety disorder, specific phobia, and panic disorder.

264 onfirmed diagnoses of panic disorder, social anxiety disorder, specific phobias, generalized anxiety

265 hletes reported no premorbid mood disorders, anxiety disorders, substance abuse, or alcohol abuse.

266 which is involved in stress, addiction, and anxiety disorders such as depression, acts through G-pro

267 nce responses can also be maladaptive, as in anxiety disorders such as phobias (e.g., avoiding air tr

268 and could serve as a therapeutic target for anxiety disorders, such as PTSD.

269 er, generalized anxiety disorder, and social anxiety disorder, suggest that CRF1 receptor antagonists

270 -onset obsessive-compulsive disorder and the anxiety disorders, suggest a broad and important role fo

271 long been established that individuals with anxiety disorders tend to overgeneralize attributes of f

272 If untreated, anxiety disorders tend to recur chronically.

273 ded greater reductions of symptoms of social anxiety disorder than the control condition on both clin

274 ications for the management of addiction and anxiety disorders that require treatments based on the o

275 depressant effects in patients with mood and anxiety disorders that were previously resistant to trea

276 ms between posttraumatic stress disorder and anxiety disorders, the latter has received less attentio

277 ic acid (GABA)) neurotransmitter circuits in anxiety disorders, the stress system has been directly i

278 Absent in generalized anxiety disorder, these aberrations highlight a unique s

279 DCS augmentation of exposure therapy for the anxiety disorders to DCS enhancement of learning-based i

280 ealth and Care Excellence for depression and anxiety disorders to more than 537 000 patients in the U

281 ectional studies have related depressive and anxiety disorders to shorter leukocyte telomere length (

282 ERN can inform the choice between first-line anxiety disorder treatments and whether the ERN changes

283 ctural neuroimaging studies of patients with anxiety disorders utilize adult samples, and the few stu

284 step towards improving our understanding of anxiety disorder vulnerability.

285 3.5% (2.4-4.6) in Indian men; prevalence of anxiety disorders was 2.0% (1.1-3.2) and 1.9% (1.2-2.3),

286 % UI 3.3-6.2) in Indian women; prevalence of anxiety disorders was 3.3% (1.6-5.3) and 4.1% (3.3-5.0),

287 symptoms, and the treatment response rate of anxiety disorders was significantly higher in stepped ca

288 her parental education and having a comorbid anxiety disorder were protective factors.

289 er, obsessive-compulsive disorder, or social anxiety disorder were randomly assigned by principal dia

290 METHOD: Forty patients with social anxiety disorder were randomly assigned to eight session

291 Anxiety disorders were assessed using the Mini-Internati

292 Neither depressive nor anxiety disorders were directly associated with telomere

293 Comorbid anxiety disorders were more likely in psychosis spectrum

294 r these delays may be influenced by mood and anxiety disorders, which are more prevalent in women and

295 aumatic stress disorder (PTSD) is a frequent anxiety disorder with higher prevalence rates in female

296 gulation contributes to the heterogeneity of anxiety disorders with implications for cognitive-behavi

297 evidence-based psychological treatments for anxiety disorders with less attrition.

298 firmed robust associations of depressive and anxiety disorders with shorter telomeres, but interestin

299 ct likelihood of treatment response in adult anxiety disorders, with potential implications for clini

300 stigation of vmPFC safety signaling in other anxiety disorders, with potential implications for the d